Plasticity of the beta cell insulin secretory competence: preparing the pancreatic beta cell for the next meal.
about
The required beta cell research for improving treatment of type 2 diabetesControl of mRNA translation preserves endoplasmic reticulum function in beta cells and maintains glucose homeostasisAnchored phosphatases modulate glucose homeostasisUbiquitin fold modifier 1 (UFM1) and its target UFBP1 protect pancreatic beta cells from ER stress-induced apoptosisGlucose-induced O₂ consumption activates hypoxia inducible factors 1 and 2 in rat insulin-secreting pancreatic beta-cellsTranscriptional response of pancreatic beta cells to metabolic stimulation: large scale identification of immediate-early and secondary response genes.Extent of beta cell destruction is important but insufficient to predict the onset of type 1 diabetes mellitus.Zinc transporters and their role in the pancreatic β-cell.Glucose-dependent insulinotropic polypeptide signaling in pancreatic β-cells and adipocytesLoss of high-frequency glucose-induced Ca2+ oscillations in pancreatic islets correlates with impaired glucose tolerance in Trpm5-/- miceClusters of conserved beta cell marker genes for assessment of beta cell phenotype.Polymorphism rs11085226 in the gene encoding polypyrimidine tract-binding protein 1 negatively affects glucose-stimulated insulin secretion.Type 2 diabetes mellitus and the cardiometabolic syndrome: impact of incretin-based therapies.Interleukin-6 enhances insulin secretion by increasing glucagon-like peptide-1 secretion from L cells and alpha cells.Glucose controls CREB activity in islet cells via regulated phosphorylation of TORC2Rapid changes of mRNA-binding protein levels following glucose and 3-isobutyl-1-methylxanthine stimulation of insulinoma INS-1 cells.Growth factor control of pancreatic islet regeneration and functionMethyl succinate antagonises biguanide-induced AMPK-activation and death of pancreatic beta-cells through restoration of mitochondrial electron transfer.Epac2A makes a new impact in β-cell biology.Mitochondrial oxidative stress contributes differently to rat pancreatic islet cell apoptosis and insulin secretory defects after prolonged culture in a low non-stimulating glucose concentration.Glucokinase activation is beneficial or toxic to cultured rat pancreatic islets depending on the prevailing glucose concentration.Re-analysis of public genetic data reveals a rare X-chromosomal variant associated with type 2 diabetes.Testing the plasticity of insulin secretion and β-cell functionin vivo: responses to chronic hyperglycaemia in the sheep
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P2860
Plasticity of the beta cell insulin secretory competence: preparing the pancreatic beta cell for the next meal.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
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2004年论文
@zh-cn
name
Plasticity of the beta cell in ...... c beta cell for the next meal.
@ast
Plasticity of the beta cell in ...... c beta cell for the next meal.
@en
type
label
Plasticity of the beta cell in ...... c beta cell for the next meal.
@ast
Plasticity of the beta cell in ...... c beta cell for the next meal.
@en
prefLabel
Plasticity of the beta cell in ...... c beta cell for the next meal.
@ast
Plasticity of the beta cell in ...... c beta cell for the next meal.
@en
P2093
P2860
P1476
Plasticity of the beta cell in ...... c beta cell for the next meal.
@en
P2093
Frans C Schuit
Karine Hellemans
Simon A Hinke
P2860
P304
P356
10.1113/JPHYSIOL.2004.064881
P407
P577
2004-06-04T00:00:00Z