How does type 1 diabetes develop?: the notion of homicide or β-cell suicide revisited. - Wikidata - awgldk - TriplyDB

How does type 1 diabetes develop?: the notion of homicide or β-cell suicide revisited.

How does type 1 diabetes develop?: the notion of homicide or β-cell suicide revisited.

http://www.wikidata.org/entity/Q35796906

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Reversal of type 1 diabetes via islet β cell regeneration following immune modulation by cord blood-derived multipotent stem cells β-cell replacement sources for type 1 diabetes: a focus on pancreatic ductal cells Stem cell therapy to cure type 1 diabetes: from hype to hope.Dendritic cell subsets in type 1 diabetes: friend or foe?Type 1 diabetes Concise review: pancreas regeneration: recent advances and perspectives Development, growth and maintenance of β-cell mass: models are also part of the story Tcf19 is a novel islet factor necessary for proliferation and survival in the INS-1 β-cell line Deoxyhypusine synthase promotes differentiation and proliferation of T helper type 1 (Th1) cells in autoimmune diabetes Islet β-cell endoplasmic reticulum stress precedes the onset of type 1 diabetes in the nonobese diabetic mouse model.Autoimmune responses in T1DM: quantitative methods to understand onset, progression, and prevention of disease Macrophages and β-cells are responsible for CXCR2-mediated neutrophil infiltration of the pancreas during autoimmune diabetes Characteristics and determinants of partial remission in children with type 1 diabetes using the insulin-dose-adjusted A1C definition Mapping I-A(g7) restricted epitopes in murine G6PC2.Loss of Fbw7 reprograms adult pancreatic ductal cells into α, δ, and β cells.Minireview: 12-Lipoxygenase and Islet β-Cell Dysfunction in Diabetes.Unraveling the contribution of pancreatic beta-cell suicide in autoimmune type 1 diabetes.The changing landscape of type 1 diabetes: recent developments and future frontiers.Regionally distinct alterations in the composition of the gut microbiota in rats with streptozotocin-induced diabetes.Proinsulin misfolding and endoplasmic reticulum stress during the development and progression of diabetes.Gender-dimorphic regulation of DJ1 and its interactions with metabolic proteins in streptozotocin-induced diabetic rats.STAT1-mediated down-regulation of Bcl-2 expression is involved in IFN-γ/TNF-α-induced apoptosis in NIT-1 cells.Juvenile Diabetes Research Foundation: mission, strategy, and priorities Th1-Like ICOS+ Foxp3+ Treg Cells Preferentially Express CXCR3 and Home to β-Islets during Pre-Diabetes in BDC2.5 NOD Mice Induction of protective genes leads to islet survival and function.Evidence for Residual and Partly Reparable Insulin Secretory Function and Maintained β-Cell Gene Expression in Islets From Patients With Type 1 Diabetes ER stress as a trigger for β-cell dysfunction and autoimmunity in type 1 diabetes.Moderate Intensity Training Impact on the Inflammatory Status and Glycemic Profiles in NOD Mice.Calcium-independent phospholipases A2 and their roles in biological processes and diseases.Continuum model of T-cell avidity: Understanding autoreactive and regulatory T-cell responses in type 1 diabetes.The potential of Fas ligand (apoptosis-inducing molecule) as an unconventional therapeutic target in type 1 diabetes.Islet autoantigens: structure, function, localization, and regulation.Deficiency in the nuclear factor E2-related factor 2 renders pancreatic β-cells vulnerable to arsenic-induced cell damage.The pathogenesis and natural history of type 1 diabetes.Evidence of β-Cell Dedifferentiation in Human Type 2 Diabetes Trimeprazine increases IRS2 in human islets and promotes pancreatic β cell growth and function in mice Divergent effects of sulforaphane on basal and glucose-stimulated insulin secretion in β-cells: role of reactive oxygen species and induction of endogenous antioxidants Protective effects of polyamine depletion in mouse models of type 1 diabetes: implications for therapy.Mild hyperglycemia triggered islet function recovery in streptozotocin-induced insulin-deficient diabetic rats.Osteocalcin promotes β-cell proliferation during development and adulthood through Gprc6a.

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How does type 1 diabetes develop?: the notion of homicide or β-cell suicide revisited.

http://www.wikidata.org/entity/Q35796906

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

How does type 1 diabetes develop?: the notion of homicide or β-cell suicide revisited.

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How does type 1 diabetes develop?: the notion of homicide or β-cell suicide revisited.

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How does type 1 diabetes develop?: the notion of homicide or β-cell suicide revisited.

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How does type 1 diabetes develop?: the notion of homicide or β-cell suicide revisited.

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Christopher J Rhodes

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Dorene S Markel

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George S Eisenbarth

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Jeffrey A Bluestone

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Kevan C Herold

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Mark A Atkinson

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Massimo Pietropaolo

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Matthias Hebrok

http://www.w3.org/2001/XMLSchema#string

Matthias Von Herrath

http://www.w3.org/2001/XMLSchema#string

Peter R Arvan

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P2860

Chromogranin A is an autoantigen in type 1 diabetes

C-peptide levels and insulin independence following autologous nonmyeloablative hematopoietic stem cell transplantation in newly diagnosed type 1 diabetes mellitus

The honeymoon phase: intersection of metabolism and immunology

Genetics, pathogenesis and clinical interventions in type 1 diabetes

Interleukin-1-receptor antagonist in type 2 diabetes mellitus

The endoplasmic reticulum in pancreatic beta cells of type 2 diabetes patients.

ROS signaling, oxidative stress and Nrf2 in pancreatic beta-cell function.

Mononuclear cell infiltration and its relation to the expression of major histocompatibility complex antigens and adhesion molecules in pancreas biopsy specimens from newly diagnosed insulin-dependent diabetes mellitus patients.

Regulation of insulin secretion: role of mitochondrial signalling.

Diabetogenic T cells recognize insulin bound to IAg7 in an unexpected, weakly binding register.

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type-1 diabetes

apoptotic process

veteran suicide

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3292309

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