Host defense response to cytomegalovirus in the central nervous system. Predominance of the monocyte.
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CD4+ T-cell reconstitution reduces cytomegalovirus in the immunocompromised brainSystemic immune deficiency necessary for cytomegalovirus invasion of the mature brain.BH3 Profiling Reveals Selectivity by Herpesviruses for Specific Bcl-2 Proteins To Mediate Survival of Latently Infected Cells.Roles of phosphatidylinositol 3-kinase and NF-kappaB in human cytomegalovirus-mediated monocyte diapedesis and adhesion: strategy for viral persistence.Human cytomegalovirus induction of a unique signalsome during viral entry into monocytes mediates distinct functional changes: a strategy for viral disseminationHuman cytomegalovirus stimulates monocyte-to-macrophage differentiation via the temporal regulation of caspase 3.Guinea Pig Cytomegalovirus (GPCMV): A Model for the Study of the Prevention and Treatment of Maternal-Fetal Transmission.Human Cytomegalovirus Promotes Survival of Infected Monocytes via a Distinct Temporal Regulation of Cellular Bcl-2 Family Proteins.Guinea pig cytomegalovirus immediate-early transcription.Transcriptome analysis reveals human cytomegalovirus reprograms monocyte differentiation toward an M1 macrophage.PI3K-dependent upregulation of Mcl-1 by human cytomegalovirus is mediated by epidermal growth factor receptor and inhibits apoptosis in short-lived monocytes.Human cytomegalovirus induces monocyte differentiation and migration as a strategy for dissemination and persistence.HCMV reprogramming of infected monocyte survival and differentiation: a Goldilocks phenomenon.Cytomegalovirus encephalitis: neuropathological comparison of the guinea pig model with the opportunistic infection in AIDS.Enhanced cytomegalovirus infection of developing brain independent of the adaptive immune systemThe T-cell-independent role of gamma interferon and tumor necrosis factor alpha in macrophage activation during murine cytomegalovirus and herpes simplex virus infections.Human cytomegalovirus subverts the functions of monocytes, impairing chemokine-mediated migration and leukocyte recruitment.HCMV utilizes a non-traditional STAT1 activation cascade via signaling through EGFR and integrins to efficiently promote the motility, differentiation, and polarization of infected monocytes.Brain trauma enhances transient cytomegalovirus invasion of the brain only in mice that are immunodeficientProtein pUL128 of human cytomegalovirus is necessary for monocyte infection and blocking of migration.Cytomegalovirus induces interferon-stimulated gene expression and is attenuated by interferon in the developing brain.HCMV Infection and Apoptosis: How Do Monocytes Survive HCMV Infection?
P2860
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P2860
Host defense response to cytomegalovirus in the central nervous system. Predominance of the monocyte.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
Host defense response to cytom ...... Predominance of the monocyte.
@ast
Host defense response to cytom ...... Predominance of the monocyte.
@en
type
label
Host defense response to cytom ...... Predominance of the monocyte.
@ast
Host defense response to cytom ...... Predominance of the monocyte.
@en
prefLabel
Host defense response to cytom ...... Predominance of the monocyte.
@ast
Host defense response to cytom ...... Predominance of the monocyte.
@en
P2093
P2860
P1476
Host defense response to cytom ...... Predominance of the monocyte.
@en
P2093
P2860
P407
P577
1989-01-01T00:00:00Z