Synaptic pathology and glial responses to neuronal injury precede the formation of senile plaques and amyloid deposits in the aging cerebral cortex.
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A preclinical cognitive test battery to parallel the National Institute of Health Toolbox in humans: bridging the translational gapAgrin in Alzheimer's disease: altered solubility and abnormal distribution within microvasculature and brain parenchymaNonhuman primate models of Alzheimer-like cerebral proteopathyMitochondrial and Cell Death Mechanisms in Neurodegenerative Diseases.CD45 deficiency drives amyloid-β peptide oligomers and neuronal loss in Alzheimer's disease miceThe mitochondrial permeability transition pore regulates nitric oxide-mediated apoptosis of neurons induced by target deprivationAmyloid-β as a modulator of synaptic plasticityMicroglial response to amyloid plaques in APPsw transgenic mice.Astrocytes containing amyloid beta-protein (Abeta)-positive granules are associated with Abeta40-positive diffuse plaques in the aged human brainExpression and distribution of amyloid precursor protein-like protein-2 in Alzheimer's disease and in normal brainAmyloid-β oligomers stimulate microglia through a tyrosine kinase dependent mechanism.Ultrastructural studies in APP/PS1 mice expressing human ApoE isoforms: implications for Alzheimer's diseaseSelective cytotoxicity of intracellular amyloid beta peptide1-42 through p53 and Bax in cultured primary human neurons.An approach to experimental synaptic pathology using green fluorescent protein-transgenic mice and gene knockout mice to show mitochondrial permeability transition pore-driven excitotoxicity in interneurons and motoneuronsBiology of mitochondria in neurodegenerative diseases.Extracellular deposition of beta-amyloid upon p53-dependent neuronal cell death in transgenic mice.Neuronal cell death in Alzheimer's disease correlates with apoE uptake and intracellular Abeta stabilization.Mitochondrial DNMT3A and DNA methylation in skeletal muscle and CNS of transgenic mouse models of ALS.Alzheimer's disease: experimental models and reality.Natural non-trasgenic animal models for research in Alzheimer's disease.
P2860
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P2860
Synaptic pathology and glial responses to neuronal injury precede the formation of senile plaques and amyloid deposits in the aging cerebral cortex.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Synaptic pathology and glial r ...... in the aging cerebral cortex.
@ast
Synaptic pathology and glial r ...... in the aging cerebral cortex.
@en
type
label
Synaptic pathology and glial r ...... in the aging cerebral cortex.
@ast
Synaptic pathology and glial r ...... in the aging cerebral cortex.
@en
prefLabel
Synaptic pathology and glial r ...... in the aging cerebral cortex.
@ast
Synaptic pathology and glial r ...... in the aging cerebral cortex.
@en
P2093
P2860
P1476
Synaptic pathology and glial r ...... in the aging cerebral cortex.
@en
P2093
P2860
P304
P407
P577
1994-12-01T00:00:00Z