Subunit positional effects revealed by novel heteromeric inwardly rectifying K+ channels.
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Inwardly rectifying K+ channel Kir7.1 is highly expressed in thyroid follicular cells, intestinal epithelial cells and choroid plexus epithelial cells: implication for a functional coupling with Na+,K+-ATPaseFunctional analysis of missense variants in the TRESK (KCNK18) K channelDifferential pH sensitivity of Kir4.1 and Kir4.2 potassium channels and their modulation by heteropolymerisation with Kir5.1Expression of a functional Kir4 family inward rectifier K+ channel from a gene cloned from mouse liverMyelinating satellite oligodendrocytes are integrated in a glial syncytium constraining neuronal high-frequency activityGain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy.A Structural Determinant for the Control of PIP2 Sensitivity in G Protein-gated Inward Rectifier K+ ChannelsAn inward rectifier K(+) channel at the basolateral membrane of the mouse distal convoluted tubule: similarities with Kir4-Kir5.1 heteromeric channelsFunctional Kir7.1 channels localized at the root of apical processes in rat retinal pigment epitheliumKCNJ10 gene mutations causing EAST syndrome (epilepsy, ataxia, sensorineural deafness, and tubulopathy) disrupt channel functionInwardly rectifying potassium channel Kir4.1 is responsible for the native inward potassium conductance of satellite glial cells in sensory gangliaIn vivo formation of a proton-sensitive K+ channel by heteromeric subunit assembly of Kir5.1 with Kir4.1Kir5.1 underlies long-lived subconductance levels in heteromeric Kir4.1/Kir5.1 channels from Xenopus tropicalisSilencing the Kir4.1 potassium channel subunit in satellite glial cells of the rat trigeminal ganglion results in pain-like behavior in the absence of nerve injuryGenetic inactivation of an inwardly rectifying potassium channel (Kir4.1 subunit) in mice: phenotypic impact in retinaInward rectifier potassium (Kir2.1) channels as end-stage boosters of endothelium-dependent vasodilators.K(+) channelepsy: progress in the neurobiology of potassium channels and epilepsy.MUPP1 complexes renal K+ channels to alter cell surface expression and whole cell currents.Small-molecule modulators of inward rectifier K+ channels: recent advances and future possibilities.Plant K+ channel alpha-subunits assemble indiscriminately.Modulation of the heteromeric Kir4.1-Kir5.1 channel by multiple neurotransmitters via Galphaq-coupled receptorsKCNJ10 determines the expression of the apical Na-Cl cotransporter (NCC) in the early distal convoluted tubule (DCT1).Trace amines depress D(2)-autoreceptor-mediated responses on midbrain dopaminergic cells.Astrocytes in the retrotrapezoid nucleus sense H+ by inhibition of a Kir4.1-Kir5.1-like current and may contribute to chemoreception by a purinergic mechanism.Genetic inactivation of Kcnj16 identifies Kir5.1 as an important determinant of neuronal PCO2/pH sensitivity.Molecular basis of decreased Kir4.1 function in SeSAME/EAST syndrome.Molecular dissection of the inward rectifier potassium current (IK1) in rabbit cardiomyocytes: evidence for heteromeric co-assembly of Kir2.1 and Kir2.2.H,K-ATPase protein localization and Kir4.1 function reveal concordance of three axes during early determination of left-right asymmetry.Molecular aspects of structure, gating, and physiology of pH-sensitive background K2P and Kir K+-transport channels.Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+ channel subunit contrasts with that observed in SeSAME/EAST syndrome.Ablation of Kcnj10 expression in retinal explants revealed pivotal roles for Kcnj10 in the proliferation and development of Müller glia.The disruption of central CO2 chemosensitivity in a mouse model of Rett syndrome.KCNJ10 (Kir4.1) is expressed in the basolateral membrane of the cortical thick ascending limb.Control of channel activity through a unique amino acid residue of a G protein-gated inwardly rectifying K+ channel subunitBasolateral membrane K+ channels in renal epithelial cellsIdentifying Candidate Genes that Underlie Cellular pH Sensitivity in Serotonin Neurons Using Transcriptomics: A Potential Role for Kir5.1 Channels.Inwardly rectifying potassium channels (Kir) in central nervous system glia: a special role for Kir4.1 in glial functions.Biophysical and molecular mechanisms underlying the modulation of heteromeric Kir4.1-Kir5.1 channels by CO2 and pH.Cloning and functional characterization of inward-rectifying potassium (Kir) channels from Malpighian tubules of the mosquito Aedes aegyptiNovel KCNJ10 Gene Variations Compromise Function of Inwardly Rectifying Potassium Channel 4.1.
P2860
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P2860
Subunit positional effects revealed by novel heteromeric inwardly rectifying K+ channels.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Subunit positional effects revealed by novel heteromeric inwardly rectifying K+ channels.
@ast
Subunit positional effects revealed by novel heteromeric inwardly rectifying K+ channels.
@en
type
label
Subunit positional effects revealed by novel heteromeric inwardly rectifying K+ channels.
@ast
Subunit positional effects revealed by novel heteromeric inwardly rectifying K+ channels.
@en
prefLabel
Subunit positional effects revealed by novel heteromeric inwardly rectifying K+ channels.
@ast
Subunit positional effects revealed by novel heteromeric inwardly rectifying K+ channels.
@en
P2093
P2860
P1433
P1476
Subunit positional effects revealed by novel heteromeric inwardly rectifying K+ channels.
@en
P2093
P2860
P304
P407
P577
1996-06-01T00:00:00Z