Electrophysiological effects of AFD-21 and AFD-19, new antiarrhythmic compounds on papillary muscles and single ventricular myocytes isolated from guinea-pig hearts.

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Electrophysiological effects of AFD-21 and AFD-19, new antiarrhythmic compounds on papillary muscles and single ventricular myocytes isolated from guinea-pig hearts.

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1990 nî lūn-bûn

@nan

1990年の論文

@ja

1990年論文

@yue

1990年論文

@zh-hant

1990年論文

@zh-hk

1990年論文

@zh-mo

1990年論文

@zh-tw

1990年论文

@wuu

1990年论文

@zh

1990年论文

@zh-cn

name

Electrophysiological effects o ...... olated from guinea-pig hearts.

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Electrophysiological effects o ...... olated from guinea-pig hearts.

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type

Item

label

Electrophysiological effects o ...... olated from guinea-pig hearts.

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Electrophysiological effects o ...... olated from guinea-pig hearts.

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Electrophysiological effects o ...... olated from guinea-pig hearts.

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Electrophysiological effects o ...... olated from guinea-pig hearts.

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P1476

Electrophysiological effects o ...... olated from guinea-pig hearts.

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P2093

Kamiya K

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Kawamura T

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Kodama I

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Suzuki R

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Toyama J

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P2860

Effects of dimethylpropranolol (UM-272) on the electrophysiological properties of guinea-pig ventricular muscles

Effects of AFD-21, a new class I antiarrhythmic agent, and AFD-19, its active metabolite, on the maximal rate of rise of action potentials in guinea pig papillary muscles: dependence on time, voltage, and action potential duration

A classification of antiarrhythmic actions reassessed after a decade of new drugs.

Fast sodium current in cardiac muscle. A quantitative description.

Kinetics of onset of rate-dependent effects of Class I antiarrhythmic drugs are important in determining their effects on refractoriness in guinea-pig ventricle, and provide a theoretical basis for their subclassification.

Importance of physico-chemical properties in determining the kinetics of the effects of Class I antiarrhythmic drugs on maximum rate of depolarization in guinea-pig ventricle

Time- and voltage-dependent interactions of antiarrhythmic drugs with cardiac sodium channels.

Antiarrhythmic drug action. Blockade of the inward sodium current.

Antiarrhythmic agents: the modulated receptor mechanism of action of sodium and calcium channel-blocking drugs.

Why do some drugs preferentially block open sodium channels?

P304

803-808

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scholarly article

P356

10.1111/J.1476-5381.1990.TB14161.X

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English

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P478

101

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P577

1990-12-01T00:00:00Z

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P5875

21069735

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2085705

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P932

1917845

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