Oxidative stress induces itch via activation of transient receptor potential subtype ankyrin 1 in mice.
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Oxidative Stress in Atopic DermatitisNew insights into the mechanisms of itch: are pain and itch controlled by distinct mechanisms?Neuroimmune interactions in itch: Do chronic itch, chronic pain, and chronic cough share similar mechanisms?Neural processing of itch.Activity-dependent silencing reveals functionally distinct itch-generating sensory neurons.Transient receptor potential (TRP) channels: a clinical perspectiveAlpha-lipoic Acid suppresses P2X receptor activities and visceral hypersensitivity to colorectal distention in diabetic rats.A dynamic set point for thermal adaptation requires phospholipase C-mediated regulation of TRPM8 in vivoMolecular and cellular mechanisms that initiate pain and itch.Trp channels and itch.TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.The transient receptor potential channel TRPA1: from gene to pathophysiology.Molecular signaling and targets from itch: lessons for cough.Investigational drugs for pruritus.Targeting TRP ion channels for itch relief.Transient receptor potential ankyrin 1 (TRPA1) antagonists.Antioxidants Attenuate Acute and Chronic Itch: Peripheral and Central Mechanisms of Oxidative Stress in Pruritus.Preclinical and human surrogate models of itch.A subpopulation of itch-sensing neurons marked by Ret and somatostatin expression.Mechanistic link between diesel exhaust particles and respiratory reflexes.TNF-α/TNFR1 Signaling is Required for the Full Expression of Acute and Chronic Itch in Mice via Peripheral and Central Mechanisms.Recent progress in understanding the mechanisms of pain and itch.Vitamin E modulates oxidative stress and protein kinase C activator (PMA)-induced TRPM2 channel gate in dorsal root ganglion of rats.Elevated H2 O2 levels in trinitrobenzene sulfate-induced colitis rats contributes to visceral hyperalgesia through interaction with the transient receptor potential ankyrin 1 cation channel.TRPA1 channels: molecular sentinels of cellular stress and tissue damage.Regulation of Pain and Itch by TRP Channels.Antipruritic Effects of Botulinum Neurotoxins.Itch induced by peripheral mu opioid receptors is dependent on TRPV1-expressing neurons and alleviated by channel activation
P2860
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P2860
Oxidative stress induces itch via activation of transient receptor potential subtype ankyrin 1 in mice.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Oxidative stress induces itch ...... ial subtype ankyrin 1 in mice.
@ast
Oxidative stress induces itch ...... ial subtype ankyrin 1 in mice.
@en
type
label
Oxidative stress induces itch ...... ial subtype ankyrin 1 in mice.
@ast
Oxidative stress induces itch ...... ial subtype ankyrin 1 in mice.
@en
prefLabel
Oxidative stress induces itch ...... ial subtype ankyrin 1 in mice.
@ast
Oxidative stress induces itch ...... ial subtype ankyrin 1 in mice.
@en
P2860
P1476
Oxidative stress induces itch ...... ial subtype ankyrin 1 in mice.
@en
P2093
P2860
P2888
P304
P356
10.1007/S12264-012-1207-9
P50
P577
2012-04-01T00:00:00Z