Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils. - Wikidata - awgldk - TriplyDB

Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

http://www.wikidata.org/entity/Q35978720

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Genome-wide analysis of group a streptococci reveals a mutation that modulates global phenotype and disease specificity.Neutrophils in innate host defense against Staphylococcus aureus infections Gene expression in systemic lupus erythematosus: Bone marrow analysis differentiates active from inactive disease and reveals apoptosis and granulopoiesis signatures Cleavage of CXCR1 on neutrophils disables bacterial killing in cystic fibrosis lung disease How do microbes evade neutrophil killing?G1-4A, a Polysaccharide from Tinospora cordifolia Inhibits the Survival of Mycobacterium tuberculosis by Modulating Host Immune Responses in TLR4 Dependent Manner Transcriptome analysis of murine macrophages in response to infection with Streptococcus pyogenes reveals an unusual activation program Gene expression signatures diagnose influenza and other symptomatic respiratory viral infections in humans Antimicrobial activity of community-associated methicillin-resistant Staphylococcus aureus is caused by phenol-soluble modulin derivatives.Yersinia pestis survival and replication within human neutrophil phagosomes and uptake of infected neutrophils by macrophages.A meta-analysis of multiple matched copy number and transcriptomics data sets for inferring gene regulatory relationships.Integration of clinical data, pathology, and cDNA microarrays in influenza virus-infected pigtailed macaques (Macaca nemestrina)An unbiased systems genetics approach to mapping genetic loci modulating susceptibility to severe streptococcal sepsis.SalK/SalR, a two-component signal transduction system, is essential for full virulence of highly invasive Streptococcus suis serotype 2.Incompetence of neutrophils to invasive group A streptococcus is attributed to induction of plural virulence factors by dysfunction of a regulator.Phagocytosis of Staphylococcus aureus by macrophages exerts cytoprotective effects manifested by the upregulation of antiapoptotic factors.The SaeR/S gene regulatory system is essential for innate immune evasion by Staphylococcus aureus.Yersinia pestis two-component gene regulatory systems promote survival in human neutrophils Influence of Microbes on Neutrophil Life and Death.Are proteinase 3 and cathepsin C enzymes related to pathogenesis of periodontitis?Epic Immune Battles of History: Neutrophils vs. Staphylococcus aureus.Harnessing Neutrophil Survival Mechanisms during Chronic Infection by Pseudomonas aeruginosa: Novel Therapeutic Targets to Dampen Inflammation in Cystic Fibrosis Bacterial and host determinants of MAL activation upon EPEC infection: the roles of Tir, ABRA, and FLRT3 Transcriptomic analysis of milk somatic cells in mastitis resistant and susceptible sheep upon challenge with Staphylococcus epidermidis and Staphylococcus aureus Mutational analysis of the group A streptococcal operon encoding streptolysin S and its virulence role in invasive infection.Phagocytosis and killing of Staphylococcus aureus by human neutrophils.Early transcriptional response of human neutrophils to Anaplasma phagocytophilum infection Neutrophil apoptosis: relevance to the innate immune response and inflammatory disease.MicroRNA expression profile in RAW264.7 cells in response to Brucella melitensis infection Characterization of a Staphylococcus aureus surface virulence factor that promotes resistance to oxidative killing and infectious endocarditis.Simultaneous Host-Pathogen Transcriptome Analysis during Granulibacter bethesdensis Infection of Neutrophils from Healthy Subjects and Patients with Chronic Granulomatous Disease Global analysis of community-associated methicillin-resistant Staphylococcus aureus exoproteins reveals molecules produced in vitro and during infection.Dextromethorphan efficiently increases bactericidal activity, attenuates inflammatory responses, and prevents group a streptococcal sepsis.Staphylococcus aureus biofilms prevent macrophage phagocytosis and attenuate inflammation in vivo.RNA profiling in host-pathogen interactions.Toward a genome-wide systems biology analysis of host-pathogen interactions in group A Streptococcus.Analysis of the transcriptome of group A Streptococcus in mouse soft tissue infection Host responses to group a streptococcus: cell death and inflammation Mesenchymal Stromal/Stem Cell and Minocycline-Loaded Hydrogels Inhibit the Growth of Staphylococcus aureus that Evades Immunomodulation of Blood-Derived Leukocytes.Rapid neutrophil destruction following phagocytosis of Staphylococcus aureus

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Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

http://www.wikidata.org/entity/Q35978720

description

2003 nî lūn-bûn

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2003年の論文

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2003年論文

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name

Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

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Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

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type

label

Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

@ast

Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

@en

prefLabel

Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

@ast

Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils.

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P2093

Adeline R Whitney

http://www.w3.org/2001/XMLSchema#string

Jovanka M Voyich

http://www.w3.org/2001/XMLSchema#string

Kevin R Braughton

http://www.w3.org/2001/XMLSchema#string

Scott D Kobayashi

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Tom G Schwan

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P2860

Cell activation and apoptosis by bacterial lipoproteins through toll-like receptor-2

The apoptotic signaling pathway activated by Toll-like receptor-2

Isolation of mononuclear cells and granulocytes from human blood. Isolation of monuclear cells by one centrifugation, and of granulocytes by combining centrifugation and sedimentation at 1 g

Identification of genes differentially regulated by interferon alpha, beta, or gamma using oligonucleotide arrays

AP-1 as a regulator of cell life and death

Points of control in inflammation

Identification of Rgg-regulated exoproteins of Streptococcus pyogenes.

Stereotyped and specific gene expression programs in human innate immune responses to bacteria.

Human macrophage activation programs induced by bacterial pathogens.

Global changes in gene expression by human polymorphonuclear leukocytes during receptor-mediated phagocytosis: cell fate is regulated at the level of gene expression.

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