Critical role of soluble amyloid-β for early hippocampal hyperactivity in a mouse model of Alzheimer's disease.
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Emerging Link between Alzheimer's Disease and Homeostatic Synaptic PlasticityImaging Multimodalities for Dissecting Alzheimer's Disease: Advanced Technologies of Positron Emission Tomography and Fluorescence ImagingCortical odor processing in health and diseaseIntraneuronal Aβ accumulation induces hippocampal neuron hyperexcitability through A-type K(+) current inhibition mediated by activation of caspases and GSK-3.Mitochondrial functional state impacts spontaneous neocortical activity and resting state FMRIInterictal spikes during sleep are an early defect in the Tg2576 mouse model of β-amyloid neuropathologyWhy looking at the whole hippocampus is not enough-a critical role for anteroposterior axis, subfield and activation analyses to enhance predictive value of hippocampal changes for Alzheimer's disease diagnosis.Analyzing dendritic spine pathology in Alzheimer's disease: problems and opportunitiesPathogenic PS1 phosphorylation at Ser367Posttraumatic stress disorder-like induction elevates β-amyloid levels, which directly activates corticotropin-releasing factor neurons to exacerbate stress responses.Seizures and epileptiform activity in the early stages of Alzheimer diseaseMapping synaptic glutamate transporter dysfunction in vivo to regions surrounding Aβ plaques by iGluSnFR two-photon imaging.Early and progressive deficit of neuronal activity patterns in a model of local amyloid pathology in mouse prefrontal cortexBiomarkers in the Diagnosis and Prognosis of Alzheimer's Disease.Spatial learning and memory impairments are associated with increased neuronal activity in 5XFAD mouse as measured by manganese-enhanced magnetic resonance imaging.The evolution of cost-efficiency in neural networks during recovery from traumatic brain injuryAltered Kv2.1 functioning promotes increased excitability in hippocampal neurons of an Alzheimer's disease mouse model.Ligand-based and structure-based investigation for Alzheimer's disease from traditional chinese medicine.Intrinsic brain activity of cognitively normal older persons resembles more that of patients both with and at risk for Alzheimer's disease than that of healthy younger persons.Age-Dependent Modifications of AMPA Receptor Subunit Expression Levels and Related Cognitive Effects in 3xTg-AD Mice.Degenerative alterations in noradrenergic neurons of the locus coeruleus in Alzheimer's diseaseLipocalin 2 modulates the cellular response to amyloid beta.Abeta(1-42) enhances neuronal excitability in the CA1 via NR2B subunit-containing NMDA receptorsModifications of hippocampal circuits and early disruption of adult neurogenesis in the tg2576 mouse model of Alzheimer's disease.Early hyperactivity in lateral entorhinal cortex is associated with elevated levels of AβPP metabolites in the Tg2576 mouse model of Alzheimer's disease.Brain amyloid-β burden is associated with disruption of intrinsic functional connectivity within the medial temporal lobe in cognitively normal elderlyImpaired firing properties of dentate granule neurons in an Alzheimer's disease animal model are rescued by PPARγ agonism.Sleep interacts with aβ to modulate intrinsic neuronal excitabilityTau-dependent Kv4.2 depletion and dendritic hyperexcitability in a mouse model of Alzheimer's disease.Linking aβ42-induced hyperexcitability to neurodegeneration, learning and motor deficits, and a shorter lifespan in an Alzheimer's modelη-Secretase processing of APP inhibits neuronal activity in the hippocampus.Selective GABA(A) α5 positive allosteric modulators improve cognitive function in aged rats with memory impairment.Differential calcium alterations in animal models of neurodegenerative disease: Reversal by FK506"Untangling" Alzheimer's disease and epilepsy.DNA repair factor BRCA1 depletion occurs in Alzheimer brains and impairs cognitive function in mice.Tau secretion is correlated to an increase of Golgi dynamics.Soluble Aβ oligomers impair hippocampal LTP by disrupting glutamatergic/GABAergic balance.Elevated Neuronal Excitability Due to Modulation of the Voltage-Gated Sodium Channel Nav1.6 by Aβ1-42.Dysregulation of oxygen hemodynamic responses to synaptic train stimulation in a rat hippocampal model of subarachnoid hemorrhage.Abnormal Population Responses in the Somatosensory Cortex of Alzheimer's Disease Model Mice.
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P2860
Critical role of soluble amyloid-β for early hippocampal hyperactivity in a mouse model of Alzheimer's disease.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Critical role of soluble amylo ...... model of Alzheimer's disease.
@ast
Critical role of soluble amylo ...... model of Alzheimer's disease.
@en
type
label
Critical role of soluble amylo ...... model of Alzheimer's disease.
@ast
Critical role of soluble amylo ...... model of Alzheimer's disease.
@en
prefLabel
Critical role of soluble amylo ...... model of Alzheimer's disease.
@ast
Critical role of soluble amylo ...... model of Alzheimer's disease.
@en
P2093
P2860
P50
P356
P1476
Critical role of soluble amylo ...... model of Alzheimer's disease.
@en
P2093
Horst A Henning
Julia Reichwald
Xiaowei Chen
P2860
P304
P356
10.1073/PNAS.1206171109
P407
P577
2012-05-16T00:00:00Z