Oxidative stress, mitochondrial dysfunction and cellular stress response in Friedreich's ataxia.
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Neuroprotective, neurotherapeutic, and neurometabolic effects of carbon monoxideEvidence linking oxidative stress, mitochondrial dysfunction, and inflammation in the brain of individuals with autismDynamics, stability and iron-binding activity of frataxin clinical mutantsMitochondrial dysfunction in autism spectrum disorders: a systematic review and meta-analysisFree radical scavengers vitamins A, C, and E plus magnesium reduce noise traumaAbeta, oxidative stress in Alzheimer disease: evidence based on proteomics studiesCharacterization of a novel dithiocarbamate glutathione reductase inhibitor and its use as a tool to modulate intracellular glutathione.Mitochondrial transfer: Implications for assisted reproductive technologiesOxidative Stress and Carbonyl Lesions in Ulcerative Colitis and Associated Colorectal CancerPrimary Mitochondrial Disease and Secondary Mitochondrial Dysfunction: Importance of Distinction for Diagnosis and TreatmentIron behaving badly: inappropriate iron chelation as a major contributor to the aetiology of vascular and other progressive inflammatory and degenerative diseasesLymphoblast Oxidative Stress Genes as Potential Biomarkers of Disease Severity and Drug Effect in Friedreich's AtaxiaThe tricarboxylic acid cycle, an ancient metabolic network with a novel twist.Altered gene expression and DNA damage in peripheral blood cells from Friedreich's ataxia patients: cellular model of pathology.The change in serum Thiol/Disulphide homeostasis after transrectal ultrasound guided prostate biopsy.Functional characterization of Friedreich ataxia iPS-derived neuronal progenitors and their integration in the adult brain.Differential expression of PGC-1α and metabolic sensors suggest age-dependent induction of mitochondrial biogenesis in Friedreich ataxia fibroblastsMesenchymal stem cells restore frataxin expression and increase hydrogen peroxide scavenging enzymes in Friedreich ataxia fibroblastsGenetic variation in iron metabolism is associated with neuropathic pain and pain severity in HIV-infected patients on antiretroviral therapy.Friedreich ataxia: molecular mechanisms, redox considerations, and therapeutic opportunities.Defects in mitochondrial axonal transport and membrane potential without increased reactive oxygen species production in a Drosophila model of Friedreich ataxia(1)H MR spectroscopy in Friedreich's ataxia and ataxia with oculomotor apraxia type 2.Loss of Frataxin induces iron toxicity, sphingolipid synthesis, and Pdk1/Mef2 activation, leading to neurodegeneration.Small molecules affecting transcription in Friedreich ataxiaCo-precipitation of phosphate and iron limits mitochondrial phosphate availability in Saccharomyces cerevisiae lacking the yeast frataxin homologue (YFH1).Stem cells from wildtype and Friedreich's ataxia mice present similar neuroprotective properties in dorsal root ganglia cells.A major role for nonenzymatic antioxidant processes in the radioresistance of Halobacterium salinarum.Metabolic and functional differences between brain and spinal cord mitochondria underlie different predisposition to pathology.Mitochondrial approaches to protect against cardiac ischemia and reperfusion injury.Degeneration modulates retinal response to transient exogenous oxidative injury.Base excision repair of chemotherapeutically-induced alkylated DNA damage predominantly causes contractions of expanded GAA repeats associated with Friedreich's ataxia.Gene expression profiling of mitochondrial oxidative phosphorylation (OXPHOS) complex I in Friedreich ataxia (FRDA) patients.Early Steroid-Induced Osteonecrosis of Rabbit Femoral Head and Panax notoginseng Saponins: Mechanism and Protective EffectsMesenchymal stem cells improve motor functions and decrease neurodegeneration in ataxic mice.Zebrafish phosvitin is an antioxidant with non-cytotoxic activity.X-ray absorption spectroscopy at the sulfur K-edge: a new tool to investigate the biochemical mechanisms of neurodegeneration.Effects of CoQ10 Supplementation on Lipid Profiles and Glycemic Control in Patients with Type 2 Diabetes: a randomized, double blind, placebo-controlled trial.Stable isotopes and LC-MS for monitoring metabolic disturbances in Friedreich's ataxia platelets.PGE2 EP1 receptor deletion attenuates 6-OHDA-induced Parkinsonism in mice: old switch, new target.Redox processes in neurodegenerative disease involving reactive oxygen species
P2860
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P2860
Oxidative stress, mitochondrial dysfunction and cellular stress response in Friedreich's ataxia.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Oxidative stress, mitochondria ...... sponse in Friedreich's ataxia.
@ast
Oxidative stress, mitochondria ...... sponse in Friedreich's ataxia.
@en
type
label
Oxidative stress, mitochondria ...... sponse in Friedreich's ataxia.
@ast
Oxidative stress, mitochondria ...... sponse in Friedreich's ataxia.
@en
prefLabel
Oxidative stress, mitochondria ...... sponse in Friedreich's ataxia.
@ast
Oxidative stress, mitochondria ...... sponse in Friedreich's ataxia.
@en
P2093
P1476
Oxidative stress, mitochondria ...... sponse in Friedreich's ataxia.
@en
P2093
A M Giuffrida Stella
Andrea Mangiameli
Caterina Tonon
Giovanni Pennisi
Giovanni Scapagnini
Maria Sapienza
Raffaele Lodi
Velia D'Agata
Vittorio Calabrese
P304
P356
10.1016/J.JNS.2005.03.012
P577
2005-06-01T00:00:00Z