Increased substance P responses in dorsal root ganglia and intestinal macrophages during Clostridium difficile toxin A enteritis in rats. - Wikidata - awgldk - TriplyDB

Increased substance P responses in dorsal root ganglia and intestinal macrophages during Clostridium difficile toxin A enteritis in rats.

Increased substance P responses in dorsal root ganglia and intestinal macrophages during Clostridium difficile toxin A enteritis in rats.

http://www.wikidata.org/entity/Q36147340

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Clostridium difficile toxins: mechanism of action and role in disease.Fulminant Clostridium difficile: an underappreciated and increasing cause of death and complications Saccharomyces boulardii protease inhibits the effects of Clostridium difficile toxins A and B in human colonic mucosa The Neuromodulation of the Intestinal Immune System and Its Relevance in Inflammatory Bowel Disease Biological and Pharmacological Aspects of the NK1-Receptor Substance P antagonist (CP-96,345) inhibits HIV-1 replication in human mononuclear phagocytes Intestinal inflammation and activation of sensory nerve pathways: a functional and morphological study in the nematode infected rat Corticotropin-releasing hormone (CRH) requirement in Clostridium difficile toxin A-mediated intestinal inflammation Pathophysiology and impact of enteric bacterial and protozoal infections: new approaches to therapy.Substance P signaling mediates BMP-dependent heterotopic ossification Substance P-evoked Cl(-) secretion in guinea pig distal colonic epithelia: interaction with PGE(2).Anatomical evidence for enteric neuroimmune interactions in Peyer's patches Human neuronal cells (NT2-N) express functional substance P and neurokinin-1 receptor coupled to MIP-1 beta expression Colocalization of substance P with tumor necrosis factor-α in the lymphocytes and mast cells in gastritis in experimental rats.Neurotensin is a proinflammatory neuropeptide in colonic inflammation Microbes and microbial toxins: paradigms for microbial-mucosal interactions II. The integrated response of the intestine to Clostridium difficile toxins.Substance P modulates colitis-associated fibrosis Metalloproteinases and transforming growth factor-alpha mediate substance P-induced mitogen-activated protein kinase activation and proliferation in human colonocytes.Neuropeptide neurotensin stimulates intestinal wound healing following chronic intestinal inflammation.Neurokinin-1 receptor antagonist treatment in polymicrobial sepsis: molecular insights Theodore E. Woodward Award. How bacterial enterotoxins work: insights from in vivo studies.Induction of colitis causes inflammatory responses in fat depots: evidence for substance P pathways in human mesenteric preadipocytes The roles of host and pathogen factors and the innate immune response in the pathogenesis of Clostridium difficile infection.Melanin-concentrating hormone (MCH) modulates C difficile toxin A-mediated enteritis in mice.Regulation of the NK-1 receptor gene expression in human macrophage cells via an NF-kappa B site on its promoter.Expression of Clostridium difficile toxins A and B and their sigma factor TcdD is controlled by temperature.Bone marrow transplantation reveals an essential synergy between neuronal and hemopoietic cell neurokinin production in pulmonary inflammation.Protective effects of neurokinin-1 receptor during colitis in mice: role of the epidermal growth factor receptor.Clostridium difficile toxin A excites enteric neurones and suppresses sympathetic neurotransmission in the guinea pig.Neuropeptides, mesenteric fat, and intestinal inflammation.Substance P (neurokinin-1) and neurokinin A (neurokinin-2) receptor gene and protein expression in the healthy and inflamed human intestine Irritable bowel syndrome: methods, mechanisms, and pathophysiology. Neural and neuro-immune mechanisms of visceral hypersensitivity in irritable bowel syndrome.Substance P mediates pro-inflammatory cytokine release form mesenteric adipocytes in Inflammatory Bowel Disease patients Role of substance P and the neurokinin 1 receptor in acute pancreatitis and pancreatitis-associated lung injury.Protease-activated receptor 2, dipeptidyl peptidase I, and proteases mediate Clostridium difficile toxin A enteritis.Immunomodulatory properties of substance P: the gastrointestinal system as a model.Role of neuropeptides in inflammatory bowel disease.Interleukin-12 (IL-12) and IL-23 induction of substance p synthesis in murine T cells and macrophages is subject to IL-10 and transforming growth factor beta regulation.Substance P stimulates cyclooxygenase-2 and prostaglandin E2 expression through JAK-STAT activation in human colonic epithelial cells.Contributions to the study of Trichinella spiralis TSL-1 antigens in host immunity.

P2860

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P2860

Increased substance P responses in dorsal root ganglia and intestinal macrophages during Clostridium difficile toxin A enteritis in rats.

http://www.wikidata.org/entity/Q36147340

description

1997 nî lūn-bûn

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1997年の論文

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1997年論文

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1997年論文

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1997年論文

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1997年論文

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1997年論文

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1997年论文

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1997年论文

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1997年论文

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Increased substance P response ...... ile toxin A enteritis in rats.

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Increased substance P response ...... ile toxin A enteritis in rats.

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Increased substance P response ...... ile toxin A enteritis in rats.

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Increased substance P response ...... ile toxin A enteritis in rats.

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Increased substance P response ...... ile toxin A enteritis in rats.

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Increased substance P response ...... ile toxin A enteritis in rats.

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Proceedings of the National Academy of Sciences of the United States of America

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Increased substance P response ...... cile toxin A enteritis in rats

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A C Keates

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B Qiu

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C P Kelly

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C Pothoulakis

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S E Leeman

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S Nikulasson

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P2860

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Isolation and characterization of rat and human glyceraldehyde-3-phosphate dehydrogenase cDNAs: genomic complexity and molecular evolution of the gene

Receptor binding sites for substance P, but not substance K or neuromedin K, are expressed in high concentrations by arterioles, venules, and lymph nodules in surgical specimens obtained from patients with ulcerative colitis and Crohn disease.

Tumour necrosis factor alpha changes porcine intestinal ion transport through a paracrine mechanism involving prostaglandins.

Role of interleukin 1 in inflammatory bowel disease--enhanced production during active disease.

Substance P and calcitonin gene-related peptide (CGRP) in gastrointestinal inflammation.

Local secretion of corticotropin-releasing hormone in the joints of Lewis rats with inflammatory arthritis.

CP-96,345, a substance P antagonist, inhibits rat intestinal responses to Clostridium difficile toxin A but not cholera toxin.

Capsaicin-sensitive nerves in the gastrointestinal tract.

Current concepts: immunology. Monocytes and macrophages.

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4788-4793

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scholarly article

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10.1073/PNAS.94.9.4788

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9

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94

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Ignazio Castagliuolo

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1997-04-01T00:00:00Z

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24451725

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9114070

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Clostridium difficile

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20803

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