Abnormal glutamate homeostasis and impaired synaptic plasticity and learning in a mouse model of tuberous sclerosis complex. - Wikidata - awgldk - TriplyDB

Abnormal glutamate homeostasis and impaired synaptic plasticity and learning in a mouse model of tuberous sclerosis complex.

Abnormal glutamate homeostasis and impaired synaptic plasticity and learning in a mouse model of tuberous sclerosis complex.

http://www.wikidata.org/entity/Q36226071

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Phosphatase and tensin homolog, deleted on chromosome 10 deficiency in brain causes defects in synaptic structure, transmission and plasticity, and myelination abnormalities Emerging pharmacotherapies for neurodevelopmental disorders Synaptic Wnt/GSK3β Signaling Hub in Autism Chronic Glutamate Toxicity in Neurodegenerative Diseases-What is the Evidence?Dendritic spine dysgenesis in autism related disorders Behavioral and Neuroanatomical Phenotypes in Mouse Models of Autism Profile of everolimus in the treatment of tuberous sclerosis complex: an evidence-based review of its place in therapy Vigabatrin inhibits seizures and mTOR pathway activation in a mouse model of tuberous sclerosis complex Transgenic expression of Glud1 (glutamate dehydrogenase 1) in neurons: in vivo model of enhanced glutamate release, altered synaptic plasticity, and selective neuronal vulnerability.Modulation of astrocyte glutamate transporters decreases seizures in a mouse model of Tuberous Sclerosis Complex.Tsc2 gene inactivation causes a more severe epilepsy phenotype than Tsc1 inactivation in a mouse model of tuberous sclerosis complex.Single-cell Tsc1 knockout during corticogenesis generates tuber-like lesions and reduces seizure threshold in mice Rapamycin prevents epilepsy in a mouse model of tuberous sclerosis complex.A circuitry and biochemical basis for tuberous sclerosis symptoms: from epilepsy to neurocognitive deficits Chronic Hyponatremia Causes Neurologic and Psychologic Impairments Transcriptomic responses in mouse brain exposed to chronic excess of the neurotransmitter glutamate.From mTOR to cognition: molecular and cellular mechanisms of cognitive impairments in tuberous sclerosis.Metabolism changes during aging in the hippocampus and striatum of glud1 (glutamate dehydrogenase 1) transgenic mice.Gene expression analysis of tuberous sclerosis complex cortical tubers reveals increased expression of adhesion and inflammatory factors.Rapamycin for treating Tuberous sclerosis and Autism spectrum disorders Mechanisms of epileptogenesis: a convergence on neural circuit dysfunction.Ischemic tolerance in an in vivo model of glutamate preconditioning.Gene expression patterns in the hippocampus during the development and aging of Glud1 (Glutamate Dehydrogenase 1) transgenic and wild type mice Proteome Analysis of Rat Hippocampus Following Morphine-induced Amnesia and State-dependent Learning.Tuberous Sclerosis: A New Frontier in Targeted Treatment of Autism Neuroprotective effects of 20(S)-protopanaxadiol against glutamate-induced mitochondrial dysfunction in PC12 cells.LST8 regulates cell growth via target-of-rapamycin complex 2 (TORC2).Synaptic plasticity in mouse models of autism spectrum disorders.Modeling cognitive dysfunction in neurofibromatosis-1 Targeted treatment trials for tuberous sclerosis and autism: no longer a dream.TOR-mediated autophagy regulates cell death in Drosophila neurodegenerative disease.mTOR, a Potential Target to Treat Autism Spectrum Disorder.Animal models of focal cortical dysplasia and tuberous sclerosis complex: recent progress toward clinical applications.Mechanisms regulating neuronal excitability and seizure development following mTOR pathway hyperactivation.Autism spectrum disorders in tuberous sclerosis: pathogenetic pathways and implications for treatment.Epilepsy Mechanisms in Neurocutaneous Disorders: Tuberous Sclerosis Complex, Neurofibromatosis Type 1, and Sturge-Weber Syndrome.Dysfunctional mTORC1 Signaling: A Convergent Mechanism between Syndromic and Nonsyndromic Forms of Autism Spectrum Disorder?Is epilepsy a preventable disorder? New evidence from animal models.Mechanisms of neurocognitive dysfunction and therapeutic considerations in tuberous sclerosis complex.Tuberous sclerosis and epilepsy: role of astrocytes.

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P2860

Abnormal glutamate homeostasis and impaired synaptic plasticity and learning in a mouse model of tuberous sclerosis complex.

http://www.wikidata.org/entity/Q36226071

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Abnormal glutamate homeostasis ...... of tuberous sclerosis complex.

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Abnormal glutamate homeostasis ...... of tuberous sclerosis complex.

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Abnormal glutamate homeostasis ...... of tuberous sclerosis complex.

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J.NBD.2007.07.015

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Neurobiology of Disease

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Abnormal glutamate homeostasis ...... of tuberous sclerosis complex.

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David F Wozniak

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David H Gutmann

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John R Cirrito

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Kelvin A Yamada

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Kevin C Ess

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Laura A Jansen

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Ling-Hui Zeng

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Michael Wong

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Vered Gazit

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Yannan Ouyang

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P2860

Epilepsy and exacerbation of brain injury in mice lacking the glutamate transporter GLT-1

The tuberous sclerosis complex

Knockout of glutamate transporters reveals a major role for astroglial transport in excitotoxicity and clearance of glutamate

The hippocampus and contextual memory retrieval in Pavlovian conditioning.

Fluoro-Jade B: a high affinity fluorescent marker for the localization of neuronal degeneration.

Tuberous sclerosis complex.

Tuberous sclerosis: from tubers to mTOR.

Beta-lactam antibiotics offer neuroprotection by increasing glutamate transporter expression.

Cortical tuber count: a biomarker indicating neurologic severity of tuberous sclerosis complex.

The natural history and treatment of epilepsy in a murine model of tuberous sclerosis.

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184-196

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10.1016/J.NBD.2007.07.015

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2

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David M. Holtzman

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2007-07-21T00:00:00Z

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6124966

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17714952

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2117357

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