Increased susceptibility to colitis and colorectal tumors in mice lacking core 3-derived O-glycans. - Wikidata - awgldk - TriplyDB

Increased susceptibility to colitis and colorectal tumors in mice lacking core 3-derived O-glycans.

Increased susceptibility to colitis and colorectal tumors in mice lacking core 3-derived O-glycans.

http://www.wikidata.org/entity/Q36229430

about

Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice Mucosal glycan foraging enhances fitness and transmission of a saccharolytic human gut bacterial symbiont Association of cell surface mucins with galectin-3 contributes to the ocular surface epithelial barrier Therapeutic Potential to Modify the Mucus Barrier in Inflammatory Bowel Disease Regulation of the Intestinal Barrier Function by Host Defense Peptides Mucin-type O-glycosylation during development A refined palate: bacterial consumption of host glycans in the gut Keeping bugs in check: The mucus layer as a critical component in maintaining intestinal homeostasis Bacteria penetrate the inner mucus layer before inflammation in the dextran sulfate colitis model Helminthic therapy: improving mucosal barrier function Aberrant mucin assembly in mice causes endoplasmic reticulum stress and spontaneous inflammation resembling ulcerative colitis Glycosyltransferase function in core 2-type protein O glycosylation.CCR6 regulation of the actin cytoskeleton orchestrates human beta defensin-2- and CCL20-mediated restitution of colonic epithelial cells The inner of the two Muc2 mucin-dependent mucus layers in colon is devoid of bacteria Loss of the actin remodeler Eps8 causes intestinal defects and improved metabolic status in mice Fucosyltransferase 2 (FUT2) non-secretor status is associated with Crohn's disease Multiple members of the UDP-GalNAc: polypeptide N-acetylgalactosaminyltransferase family are essential for viability in Drosophila Initiation of protein O glycosylation by the polypeptide GalNAcT-1 in vascular biology and humoral immunity.Metagenomic approaches for defining the pathogenesis of inflammatory bowel diseases.O-glycosylation regulates polarized secretion by modulating Tango1 stability.Sulfation of colonic mucins by N-acetylglucosamine 6-O-sulfotransferase-2 and its protective function in experimental colitis in mice C1GALT1 overexpression promotes the invasive behavior of colon cancer cells through modifying O-glycosylation of FGFR2.Structure and biological roles of mucin-type O-glycans at the ocular surface.Mechanisms of intestinal inflammation and development of associated cancers: lessons learned from mouse models.Adenoma formation following limited ablation of p120-catenin in the mouse intestine.An O-glycosyltransferase promotes cell adhesion during development by influencing secretion of an extracellular matrix integrin ligand.Critical roles of TIPE2 protein in murine experimental colitis The role of mucin-type O-glycans in eukaryotic development Analysis of carbohydrates and glycoconjugates by matrix-assisted laser desorption/ionization mass spectrometry: an update for 2007-2008.Glycosylation potential of human prostate cancer cell lines.Human intestinal MUC17 mucin augments intestinal cell restitution and enhances healing of experimental colitis Intestinal goblet cells and mucins in health and disease: recent insights and progress.Immune and genetic gardening of the intestinal microbiome.Targeted disruption of core 1 β1,3-galactosyltransferase (C1galt1) induces apical endocytic trafficking in human corneal keratinocytes.Multifarious roles of sialic acids in immunity.High-sensitivity O-glycomic analysis of mice deficient in core 2 {beta}1,6-N-acetylglucosaminyltransferases.Mucin-type O-glycosylation is critical for vascular integrity.B cells as a critical node in the microbiota-host immune system network.Epsin is required for Dishevelled stability and Wnt signalling activation in colon cancer development Mucin-bacterial interactions in the human oral cavity and digestive tract

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P2860

Increased susceptibility to colitis and colorectal tumors in mice lacking core 3-derived O-glycans.

http://www.wikidata.org/entity/Q36229430

description

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Increased susceptibility to co ...... king core 3-derived O-glycans.

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Increased susceptibility to co ...... king core 3-derived O-glycans.

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Increased susceptibility to co ...... king core 3-derived O-glycans.

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Increased susceptibility to co ...... king core 3-derived O-glycans.

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Baoyun Xia

http://www.w3.org/2001/XMLSchema#string

Bo Wei

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Guangyu An

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J Michael McDaniel

http://www.w3.org/2001/XMLSchema#string

Jonathan Braun

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Lijun Xia

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Richard D Cummings

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Tongzhong Ju

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P2860

Cloning and expression of human core 1 beta1,3-galactosyltransferase

Molecular cloning and characterization of a novel UDP-GlcNAc:GalNAc-peptide beta1,3-N-acetylglucosaminyltransferase (beta 3Gn-T6), an enzyme synthesizing the core 3 structure of O-glycans

A unique molecular chaperone Cosmc required for activity of the mammalian core 1 beta 3-galactosyltransferase

Contamination and sensitivity issues with a real-time universal 16S rRNA PCR

The inflammatory infiltrate in the acute stage of the dextran sulphate sodium induced colitis: B cell response differs depending on the percentage of DSS used to induce it

Colorectal cancer in mice genetically deficient in the mucin Muc2

Defective angiogenesis and fatal embryonic hemorrhage in mice lacking core 1-derived O-glycans.

Recognition of commensal microflora by toll-like receptors is required for intestinal homeostasis

Muc2-deficient mice spontaneously develop colitis, indicating that MUC2 is critical for colonic protection

Linking colorectal cancer to Wnt signaling

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10.1084/JEM.20061929

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17517967

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2118614

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