Phagosomal Acidification Prevents Macrophage Inflammatory Cytokine Production to Malaria, and Dendritic Cells Are the Major Source at the Early Stages of Infection: IMPLICATION FOR MALARIA PROTECTIVE IMMUNITY DEVELOPMENT
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Interaction between Plasmodium Glycosylphosphatidylinositol and the Host Protein Moesin Has No Implication in Malaria Pathology.Cytokines and Chemokines in Cerebral Malaria Pathogenesis.Small molecule-based inhibition of MEK1/2 proteins dampens inflammatory responses to malaria, reduces parasite load, and mitigates pathogenic outcomes.CD36 receptor regulates malaria-induced immune responses primarily at early blood stage infection contributing to parasitemia control and resistance to mortality.Cannabinoid Receptor 2 Modulates Susceptibility to Experimental Cerebral Malaria through a CCL17-dependent Mechanism.A malaria protein factor induces IL-4 production by dendritic cells via PI3K-Akt-NF-κB signaling independent of MyD88/TRIF and promotes Th2 response.
P2860
Phagosomal Acidification Prevents Macrophage Inflammatory Cytokine Production to Malaria, and Dendritic Cells Are the Major Source at the Early Stages of Infection: IMPLICATION FOR MALARIA PROTECTIVE IMMUNITY DEVELOPMENT
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2015 nî lūn-bûn
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2015年の論文
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Phagosomal Acidification Preve ...... ROTECTIVE IMMUNITY DEVELOPMENT
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Phagosomal Acidification Preve ...... ROTECTIVE IMMUNITY DEVELOPMENT
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type
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Phagosomal Acidification Preve ...... ROTECTIVE IMMUNITY DEVELOPMENT
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Phagosomal Acidification Preve ...... ROTECTIVE IMMUNITY DEVELOPMENT
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Phagosomal Acidification Preve ...... ROTECTIVE IMMUNITY DEVELOPMENT
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Phagosomal Acidification Preve ...... ROTECTIVE IMMUNITY DEVELOPMENT
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P2860
P921
P356
P1476
Phagosomal Acidification Preve ...... ROTECTIVE IMMUNITY DEVELOPMENT
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P2093
Nagaraj M Gowda
Xianzhu Wu
P2860
P304
23135-23147
P356
10.1074/JBC.M115.671065
P407
P577
2015-08-03T00:00:00Z