Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.
about
BAFF, a novel ligand of the tumor necrosis factor family, stimulates B cell growthFas-Fas Ligand: Checkpoint of T Cell Functions in Multiple SclerosisBcl-2 and Fas/APO-1 regulate distinct pathways to lymphocyte apoptosisDeath pathways in T cell homeostasis and their role in autoimmune diabetes.Mutation in the Fas pathway impairs CD8+ T cell memory.A role for perforin in downregulating T-cell responses during chronic viral infection.Galectin-1, an alternative signal for T cell death, is increased in activated macrophages.Cell death induced by the Fas/Fas ligand pathway and its role in pathology.Structure and function of Fas/Fas ligand.Effects of anti-Fas antibodies on lymphocytes and other organs: preparation of original and new monoclonal antibodies and amelioration of systemic autoimmune disease.The molecular basis for apoptotic defects in patients with CD95 (Fas/Apo-1) mutationsMolecular and cellular mechanisms regulating T and B cell apoptosis through Fas/FasL interaction.The power and the promise of restimulation-induced cell death in human immune diseases.A versatile system for rapid multiplex genome-edited CAR T cell generationThe many roles of FAS receptor signaling in the immune systemMolecular mechanisms for apoptosis induced by signaling through the B cell antigen receptor.'Activation-induced cell death': a special program able to preserve the homeostasis of the skin?Correction of accelerated autoimmune disease by early replacement of the mutated lpr gene with the normal Fas apoptosis gene in the T cells of transgenic MRL-lpr/lpr mice.Intertwined pathways of programmed cell death in immunity.Double negative (DN) αβ T cells: misperception and overdue recognitionCD95 (Fas) may control the expansion of activated T cells after elimination of bacteria in murine listeriosis.The HSP90 Inhibitor Ganetespib Alleviates Disease Progression and Augments Intermittent Cyclophosphamide Therapy in the MRL/lpr Mouse Model of Systemic Lupus Erythematosus.Antigen-presenting cell engineering. The molecular toolboxInvolvement of soluble CD95 in Churg-Strauss syndrome.Involvement of Fas in regression of vaginal epithelia after ovariectomy and during an estrous cycle.CrmA expression in T lymphocytes of transgenic mice inhibits CD95 (Fas/APO-1)-transduced apoptosis, but does not cause lymphadenopathy or autoimmune disease.Increased protection from vaccinia virus infection in mice genetically prone to lymphoproliferative disorders.Significantly reduced lymphadenopathy, salivary gland infiltrates and proteinuria in MRL-lpr/lpr mice treated with ultrasoluble curcumin/turmeric: increased survival with curcumin treatmentRadiation and stress-induced apoptosis: a role for Fas/Fas ligand interactions.Differential ability of T cell subsets to undergo activation-induced cell death.TCR/CD3 coupling to Fas-based cytotoxicity.Fas and activation-induced Fas ligand mediate apoptosis of T cell hybridomas: inhibition of Fas ligand expression by retinoic acid and glucocorticoids.T cells with gamma/delta T cell receptors (TCR) of intestinal type are preferentially expanded in TCR-alpha-deficient lpr mice.Fas and the art of lymphocyte maintenance.Cytotoxic T cells deficient in both functional fas ligand and perforin show residual cytolytic activity yet lose their capacity to induce lethal acute graft-versus-host diseaseA subclass of dendritic cells kills CD4 T cells via Fas/Fas-ligand-induced apoptosis.CD28-dependent activation of protein kinase B/Akt blocks Fas-mediated apoptosis by preventing death-inducing signaling complex assembly.Tolerance induction and autoimmune encephalomyelitis amelioration after administration of myelin basic protein-derived peptideConversion of membrane-bound Fas(CD95) ligand to its soluble form is associated with downregulation of its proapoptotic activity and loss of liver toxicity.Spontaneous development of plasmacytoid tumors in mice with defective Fas-Fas ligand interactions.
P2860
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P2860
Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.
@ast
Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.
@en
type
label
Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.
@ast
Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.
@en
prefLabel
Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.
@ast
Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.
@en
P2093
P2860
P356
P1476
Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.
@en
P2093
P2860
P304
P356
10.1073/PNAS.90.10.4409
P407
P577
1993-05-01T00:00:00Z