Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice - Wikidata - awgldk - TriplyDB

Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

http://www.wikidata.org/entity/Q36231826

about

Death pathways in T cell homeostasis and their role in autoimmune diabetes.Fas-mediated apoptosis regulates the composition of peripheral alphabeta T cell repertoire by constitutively purging out double negative T cells From T to B and back again: positive feedback in systemic autoimmune disease.The significance of immunohistochemistry in the diagnosis and therapy of neoplasms.Correction of accelerated autoimmune disease by early replacement of the mutated lpr gene with the normal Fas apoptosis gene in the T cells of transgenic MRL-lpr/lpr mice.CD95 (Fas) may control the expansion of activated T cells after elimination of bacteria in murine listeriosis.T-helper cell tolerance to ubiquitous nuclear antigens The potential of Fas ligand (apoptosis-inducing molecule) as an unconventional therapeutic target in type 1 diabetes.Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide.Breakdown of B cell tolerance in a mouse model of systemic lupus erythematosus.T cells with gamma/delta T cell receptors (TCR) of intestinal type are preferentially expanded in TCR-alpha-deficient lpr mice.Prevention of age-related T cell apoptosis defect in CD2-fas-transgenic mice.Inhibition of Nur77/Nurr1 leads to inefficient clonal deletion of self-reactive T cells The genetic basis of autoimmune disease in MRL-lpr/lpr mice.Apoptosis defects analyzed in TcR transgenic and fas transgenic lpr mice.A role for Fas in negative selection of thymocytes in vivo.

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P2860

Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

http://www.wikidata.org/entity/Q36231826

description

1992 nî lūn-bûn

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1992年の論文

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1992年論文

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1992年論文

@zh-hant

1992年論文

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1992年論文

@zh-mo

1992年論文

@zh-tw

1992年论文

@wuu

1992年论文

@zh

1992年论文

@zh-cn

name

Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

@ast

Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

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type

label

Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

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Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

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prefLabel

Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

@ast

Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

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P1433

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P1433

Journal of Experimental Medicine

P1476

Defective maintenance of T cell tolerance to a superantigen in MRL-lpr/lpr mice

@en

P2093

C K Edwards

http://www.w3.org/2001/XMLSchema#string

H Bluethmann

http://www.w3.org/2001/XMLSchema#string

J D Mountz

http://www.w3.org/2001/XMLSchema#string

J Zhang

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T Zhou

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P2860

The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

The cDNA structure, expression, and chromosomal assignment of the mouse Fas antigen.

Programmed cell death and extrathymic reduction of Vbeta8+ CD4+ T cells in mice tolerant to Staphylococcus aureus enterotoxin B.

Lpr and gld: single gene models of systemic autoimmunity and lymphoproliferative disease.

The kinetics of T cell antigen receptor expression by subgroups of CD4+8+ thymocytes: delineation of CD4+8+3(2+) thymocytes as post-selection intermediates leading to mature T cells.

Reduction of lupus nephritis in MRL/lpr mice by a bacterial superantigen treatment

In vivo induction of anergy in peripheral V beta 8+ T cells by staphylococcal enterotoxin B.

Transgenic rearranged T cell receptor gene inhibits lymphadenopathy and accumulation of CD4-CD8-B220+ T cells in lpr/lpr mice

The complex pattern of cytokines in serum from patients with meningococcal septic shock. Association between interleukin 6, interleukin 1, and fatal outcome.

P304

1063-1072

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scholarly article

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10.1084/JEM.176.4.1063

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176

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1402652

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P932

2119382

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