Nitro-linoleic acid inhibits vascular smooth muscle cell proliferation via the Keap1/Nrf2 signaling pathway - Wikidata - awgldk - TriplyDB

Nitro-linoleic acid inhibits vascular smooth muscle cell proliferation via the Keap1/Nrf2 signaling pathway

Nitro-linoleic acid inhibits vascular smooth muscle cell proliferation via the Keap1/Nrf2 signaling pathway

http://www.wikidata.org/entity/Q36319949

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Nitro-fatty acids: novel anti-inflammatory lipid mediators Redox-dependent anti-inflammatory signaling actions of unsaturated fatty acids NF-E2-related factor 2 regulates the stress response to UVA-1-oxidized phospholipids in skin cells.Oxidases and peroxidases in cardiovascular and lung disease: new concepts in reactive oxygen species signaling Dysfunction of Nrf-2 in CF epithelia leads to excess intracellular H2O2 and inflammatory cytokine production RAGE mediates accelerated diabetic vein graft atherosclerosis induced by combined mechanical stress and AGEs via synergistic ERK activation Transduction of redox signaling by electrophile-protein reactions Nitrated fatty acids modulate the physical properties of model membranes and the structure of transmembrane proteins.Human haem oxygenase-1 induction by nitro-linoleic acid is mediated by cAMP, AP-1 and E-box response element interactions.Nitro-fatty acids and cyclopentenone prostaglandins share strategies to activate the Keap1-Nrf2 system: a study using green fluorescent protein transgenic zebrafish Cellular uptake and antiproliferative effects of 11-oxo-eicosatetraenoic acid.Dihydro-CDDO-trifluoroethyl amide (dh404), a novel Nrf2 activator, suppresses oxidative stress in cardiomyocytes.Oxidative risk for atherothrombotic cardiovascular disease.Activation of vascular endothelial nitric oxide synthase and heme oxygenase-1 expression by electrophilic nitro-fatty acids.Electrophilic nitro-fatty acids: anti-inflammatory mediators in the vascular compartment.Covalent peroxisome proliferator-activated receptor gamma adduction by nitro-fatty acids: selective ligand activity and anti-diabetic signaling actions.Nitro-fatty acids reduce atherosclerosis in apolipoprotein E-deficient mice.Age-related changes in redox signaling and VSMC function.Protective effects of 10-nitro-oleic acid in a hypoxia-induced murine model of pulmonary hypertension Activation of the AMP-activated protein kinase (AMPK) by nitrated lipids in endothelial cells.Vascular smooth muscle cell peroxisome proliferator-activated receptor-γ deletion promotes abdominal aortic aneurysms 15-Oxoeicosatetraenoic acid is a 15-hydroxyprostaglandin dehydrogenase-derived electrophilic mediator of inflammatory signaling pathways.Electrophilic nitro-fatty acids activate NRF2 by a KEAP1 cysteine 151-independent mechanism.The ubiquitin-conjugating enzyme UBE2E3 and its import receptor importin-11 regulate the localization and activity of the antioxidant transcription factor NRF2.Electrophilic fatty acids regulate matrix metalloproteinase activity and expression.Signaling actions of electrophiles: anti-inflammatory therapeutic candidates 15-Hydroxyprostaglandin dehydrogenase generation of electrophilic lipid signaling mediators from hydroxy ω-3 fatty acids.Nitroalkene fatty acids mediate activation of Nrf2/ARE-dependent and PPARγ-dependent transcription by distinct signaling pathways and with significantly different potencies.Regulation of keratinocyte expression of stress proteins and antioxidants by the electrophilic nitrofatty acids 9- and 10-nitrooleic acid.Macrophage activation induces formation of the anti-inflammatory lipid cholesteryl-nitrolinoleate.Nitric oxide in liver diseases.Generation and esterification of electrophilic fatty acid nitroalkenes in triacylglycerides Nitro-fatty acids in cardiovascular regulation and diseases: characteristics and molecular mechanisms.Nitro-oleic acid modulates classical and regulatory activation of macrophages and their involvement in pro-fibrotic responses.Electrophilic nitro-fatty acids inhibit vascular inflammation by disrupting LPS-dependent TLR4 signalling in lipid rafts.Nitro-fatty acid formation and signaling.Nitroalkenes suppress lipopolysaccharide-induced signal transducer and activator of transcription signaling in macrophages: a critical role of mitogen-activated protein kinase phosphatase 1.Electrophilic nitro-fatty acids prevent astrocyte-mediated toxicity to motor neurons in a cell model of familial amyotrophic lateral sclerosis via nuclear factor erythroid 2-related factor activation Nrf2/Keap1 system regulates vascular smooth muscle cell apoptosis for vascular homeostasis: role in neointimal formation after vascular injury Noncatalytic interactions between glutathione S-transferases and nitroalkene fatty acids modulate nitroalkene-mediated activation of peroxisomal proliferator-activated receptor gamma

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Nitro-linoleic acid inhibits vascular smooth muscle cell proliferation via the Keap1/Nrf2 signaling pathway

http://www.wikidata.org/entity/Q36319949

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2007 nî lūn-bûn

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American Journal of Physiology - Heart and Circulatory Physiology

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Nitro-linoleic acid inhibits v ...... e Keap1/Nrf2 signaling pathway

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Bruce A Freeman

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Jifeng Zhang

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Minerva T Garcia-Barrio

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Xi-lin Chen

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Yuqing E Chen

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P2860

Differential responses of the Nrf2-Keap1 system to laminar and oscillatory shear stresses in endothelial cells

Nitration of unsaturated fatty acids by nitric oxide-derived reactive nitrogen species peroxynitrite, nitrous acid, nitrogen dioxide, and nitronium ion

Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway

Transcription factor Nrf2 coordinately regulates a group of oxidative stress-inducible genes in macrophages

Oxidative and electrophilic stresses activate Nrf2 through inhibition of ubiquitination activity of Keap1

Molecular mechanisms of leukocyte recruitment in the inflammatory process.

Nitrated fatty acids: Endogenous anti-inflammatory signaling mediators

Reversible post-translational modification of proteins by nitrated fatty acids in vivo.

Reactions of prostaglandin endoperoxide synthase with nitric oxide and peroxynitrite.

Effects of nitric oxide and nitric oxide-derived species on prostaglandin endoperoxide synthase and prostaglandin biosynthesis.

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