Experimental autoimmune myasthenia: A model of myasthenia gravis in rats and guinea pigs. - Wikidata - awgldk - TriplyDB

Experimental autoimmune myasthenia: A model of myasthenia gravis in rats and guinea pigs.

Experimental autoimmune myasthenia: A model of myasthenia gravis in rats and guinea pigs.

http://www.wikidata.org/entity/Q36358130

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Autoimmune neuromuscular disorders Strain differences in susceptibility to 2-acetylaminofluorene and phenobarbital promotion of rat hepatocarcinogenesis in a medium-term assay system: quantitation of glutathione S-transferase P-positive foci development Mechanisms of Autoantibody-Induced Pathology.Passive transfer of autoimmune autonomic neuropathy to mice.Immunization of rats with polypeptide chains from torpedo acetylcholine receptor causes an autoimmune response to receptors in rat muscle.Clinical correlations of antibodies that bind, block, or modulate human acetylcholine receptors in myasthenia gravis.Antigen-specific immunotherapeutic vaccine for experimental autoimmune myasthenia gravis Immunization with neuronal nicotinic acetylcholine receptor induces neurological autoimmune disease.Experimental myasthenia in Balb/c mice immunized with rat acetylcholine receptor from rat denervated muscle Direct method for detection and characterization of cell surface receptors for insulin by means of 125I-labeled autoantibodies against the insulin receptor.Experimental autoimmune myasthenia gravis in the mouse.Standardization of the experimental autoimmune myasthenia gravis (EAMG) model by immunization of rats with Torpedo californica acetylcholine receptors--Recommendations for methods and experimental designs Overexpression of rapsyn in rat muscle increases acetylcholine receptor levels in chronic experimental autoimmune myasthenia gravis.The thymus in myasthenia gravis. Changes typical for the human disease are absent in experimental autoimmune myasthenia gravis of the Lewis rat.Role of complement in the pathogenesis of experimental autoimmune myasthenia gravis.Experimental myasthenia gravis. A murine system.Monoclonal antibodies used to probe acetylcholine receptor structure: localization of the main immunogenic region and detection of similarities between subunits.Passive transfer of experimental autoimmune myasthenia by lymph node cells in inbred guinea pigs.Pathological mechanisms in experimental autoimmune myasthenia gravis. I. Immunogenicity of syngeneic muscle acetylcholine receptor and quantitative extraction of receptor and antibody-receptor complexes from muscles of rats with experimental automim Pathological mechanisms in experimental autoimmune myasthenia gravis. II. Passive transfer of experimental autoimmune myasthenia gravis in rats with anti-acetylcholine recepotr antibodies.Myasthenia gravis-like syndrome induced by expression of interferon gamma in the neuromuscular junction.Antigen-specific modulation of experimental myasthenia gravis: nasal tolerization with recombinant fragments of the human acetylcholine receptor alpha-subunit.Prevention of experimental autoimmune myasthenia gravis by manipulation of the immune network with a complementary peptide for the acetylcholine receptor.Experimental autoimmune myasthenia gravis.Immune disease and HLA associations with myasthenia gravis.Nonspecific lymphocyte responses in F344 and LEW rats: susceptibility to murine respiratory mycoplasmosis and examination of cellular basis for strain differences.Paraneoplastic and other autoimmune disorders of the central nervous system.Antibodies from patients with myasthenia gravis recognize determinants unique to extrajunctional acetylcholine receptors C57BL/6 mice genetically deficient in IL-12/IL-23 and IFN-gamma are susceptible to experimental autoimmune myasthenia gravis, suggesting a pathogenic role of non-Th1 cells.Antibodies against low-density lipoprotein receptor-related protein 4 induce myasthenia gravis.MuSK IgG4 autoantibodies cause myasthenia gravis by inhibiting binding between MuSK and Lrp4.Region of peptide 125-147 of acetylcholine receptor alpha subunit is exposed at neuromuscular junction and induces experimental autoimmune myasthenia gravis, T-cell immunity, and modulating autoantibodies.Anti-acetylcholine receptor antibodies directed against the alpha-bungarotoxin binding site induce a unique form of experimental myasthenia.Antibody effector mechanisms in myasthenia gravis-pathogenesis at the neuromuscular junction.The pathogenesis of myasthenia gravis.Frequency of autoimmune diseases in myasthenia gravis: a systematic review.Paraneoplastic encephalomyelopathies: pathology and mechanisms.Acetylcholine receptor-induced experimental myasthenia gravis: what have we learned from animal models after three decades?Animal models for metabolic, neuromuscular and ophthalmological rare diseases.Pathogenic immune mechanisms at the neuromuscular synapse: the role of specific antibody-binding epitopes in myasthenia gravis.

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Experimental autoimmune myasthenia: A model of myasthenia gravis in rats and guinea pigs.

http://www.wikidata.org/entity/Q36358130

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1975 nî lūn-bûn

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1975年學術文章

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Journal of Experimental Medicine

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Lennon VA

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Lindstrom JM

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Seybold ME

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P2860

Studies on purified eel acetylcholine receptor and anti-acetylcholine receptor antibody

Electrophysiological study of neuromuscular blocking action of cobra neurotoxin.

Myasthenia gravis in the dog.

Isolation of the principal neurotoxins of two Naja naja subspecies.

Neuromuscular junction in myasthenia gravis: decreased acetylcholine receptors.

Autoimmune response to acetylcholine receptor.

Immunopharmacological disease: a break in tolerance to receptor sites.

Gradually increasing doses of prednisone in myasthenia gravis. Reducing the hazards of treatment.

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1365-1375

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myasthenia gravis

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