Myasthenia gravis-like syndrome induced by expression of interferon gamma in the neuromuscular junction.
about
IFN-gamma transgenic mice: clues to the pathogenesis of systemic lupus erythematosus?Oral administration of an immunodominant T-cell epitope downregulates Th1/Th2 cytokines and prevents experimental myasthenia gravis.The nature of the active suppression of responses associated with experimental autoimmune myasthenia gravis by a dual altered peptide ligand administered by different routesTherapeutic vaccines in autoimmunity.Both CD4+ and CD8+ T cells are essential to induce experimental autoimmune myasthenia gravis.Mechanisms of cytokine-mediated localized immunoprotectionAntinuclear autoantibodies and lupus nephritis in transgenic mice expressing interferon gamma in the epidermisHypothyroidism in transgenic mice expressing IFN-gamma in the thyroidCCR2-64I and CCR5Delta32 Polymorphisms in Korean Patients with Myasthenia Gravis.T cell-independent rescue of B lymphocytes from peripheral immune tolerance.Protein tyrosine phosphatases PTP-1B and TC-PTP play nonredundant roles in macrophage development and IFN-gamma signaling.IL-4 receptor as a bridge between the immune system and muscle in experimental myasthenia gravis I: up-regulation of muscle IL-15 by IL-4Viruses, cytokines, antigens, and autoimmunity.Thymic remodeling associated with hyperplasia in myasthenia gravis.Lipopeptides of Borrelia burgdorferi outer surface proteins induce Th1 phenotype development in alphabeta T-cell receptor transgenic mice.A partially humanized monoclonal antibody to human IFN-gamma inhibits cytokine effects both in vitro and in vivo.Interferon gamma (IFN-gamma) is necessary for the genesis of acetylcholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice.Disequilibrium of T helper type 1, 2 and 17 cells and regulatory T cells during the development of experimental autoimmune myasthenia gravis.IFN-gamma is required to induce experimental autoimmune myasthenia gravis.Th1 cells of myasthenia gravis patients recognize multiple epitopes on the muscle acetylcholine receptor alpha subunit.T cell recognition of the acetylcholine receptor in myasthenia gravis.Neutralization of interleukin-9 ameliorates symptoms of experimental autoimmune myasthenia gravis in rats by decreasing effector T cells and altering humoral responses.BM stromal cells ameliorate experimental autoimmune myasthenia gravis by altering the balance of Th cells through the secretion of IDO.Modulation of immune responses and suppression of experimental autoimmune myasthenia gravis by surgical denervation of the spleen.
P2860
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P2860
Myasthenia gravis-like syndrome induced by expression of interferon gamma in the neuromuscular junction.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
Myasthenia gravis-like syndrom ...... in the neuromuscular junction.
@ast
Myasthenia gravis-like syndrom ...... in the neuromuscular junction.
@en
type
label
Myasthenia gravis-like syndrom ...... in the neuromuscular junction.
@ast
Myasthenia gravis-like syndrom ...... in the neuromuscular junction.
@en
prefLabel
Myasthenia gravis-like syndrom ...... in the neuromuscular junction.
@ast
Myasthenia gravis-like syndrom ...... in the neuromuscular junction.
@en
P2093
P2860
P356
P1476
Myasthenia gravis-like syndrom ...... in the neuromuscular junction
@en
P2093
H C Powell
J Lindstrom
J P Merlie
L Wogensen
N A Calcutt
N Sarvetnick
P2860
P304
P356
10.1084/JEM.181.2.547
P407
P577
1995-02-01T00:00:00Z