Initiation of prostate cancer in mice by Tp53R270H: evidence for an alternative molecular progression.
about
Mouse models of prostate cancer: picking the best model for the question.P53 deficiency-induced Smad1 upregulation suppresses tumorigenesis and causes chemoresistance in colorectal cancers.Diagnostic value of DNA alteration: loss of heterozygosity or allelic imbalance-promising for molecular staging of prostate cancers.The omega-3 polyunsaturated fatty acid DHA induces simultaneous apoptosis and autophagy via mitochondrial ROS-mediated Akt-mTOR signaling in prostate cancer cells expressing mutant p53ERG Activates the YAP1 Transcriptional Program and Induces the Development of Age-Related Prostate Tumors.Combined Loss of EAF2 and p53 Induces Prostate Carcinogenesis in Male Mice.Suppression of Akt1-β-catenin pathway in advanced prostate cancer promotes TGFβ1-mediated epithelial to mesenchymal transition and metastasis.
P2860
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P2860
Initiation of prostate cancer in mice by Tp53R270H: evidence for an alternative molecular progression.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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Initiation of prostate cancer ...... rnative molecular progression.
@ast
Initiation of prostate cancer ...... rnative molecular progression.
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type
label
Initiation of prostate cancer ...... rnative molecular progression.
@ast
Initiation of prostate cancer ...... rnative molecular progression.
@en
prefLabel
Initiation of prostate cancer ...... rnative molecular progression.
@ast
Initiation of prostate cancer ...... rnative molecular progression.
@en
P2093
P2860
P356
P1476
Initiation of prostate cancer ...... rnative molecular progression.
@en
P2093
Alexander D Borowsky
Jane Q Chen
Michael M Shen
Neil E Hubbard
Ralph W Devere White
Ruth L Vinall
Shola S Sulaimon
P2860
P304
P356
10.1242/DMM.008995
P577
2012-04-12T00:00:00Z