ERG Activates the YAP1 Transcriptional Program and Induces the Development of Age-Related Prostate Tumors. - Wikidata - awgldk - TriplyDB

ERG Activates the YAP1 Transcriptional Program and Induces the Development of Age-Related Prostate Tumors.

ERG Activates the YAP1 Transcriptional Program and Induces the Development of Age-Related Prostate Tumors.

http://www.wikidata.org/entity/Q42049451

about

ETS (E26 transformation-specific) up-regulation of the transcriptional co-activator TAZ promotes cell migration and metastasis in prostate cancer.Fluid shear stress activates YAP1 to promote cancer cell motility ETS Related Gene mediated Androgen Receptor Aggregation and Endoplasmic Reticulum Stress in Prostate Cancer Development.Targeting YAP-Dependent MDSC Infiltration Impairs Tumor Progression.Histone demethylase JMJD2A drives prostate tumorigenesis through transcription factor ETV1.ETS transcription factor ERG cooperates with histone demethylase KDM4A.SIRT1 increases YAP- and MKK3-dependent p38 phosphorylation in mouse liver and human hepatocellular carcinoma.The ubiquitin ligase TRIM25 targets ERG for degradation in prostate cancer.Development of Peptidomimetic Inhibitors of the ERG Gene Fusion Product in Prostate Cancer.The Germ Cell Gene TDRD1 as an ERG Target Gene and a Novel Prostate Cancer Biomarker.SOX9 drives WNT pathway activation in prostate cancer.YAP/TAZ at the Roots of Cancer.Drugging Ras GTPase: a comprehensive mechanistic and signaling structural view.Role of YAP/TAZ transcriptional regulators in resistance to anti-cancer therapies.Hippo vs. Crab: tissue-specific functions of the mammalian Hippo pathway.Photosensitizers in prostate cancer therapy.α3β1 Integrin Suppresses Prostate Cancer Metastasis via Regulation of the Hippo Pathway.Drug development against the hippo pathway in mesothelioma Independent and core pathways in oncogenic KRAS signaling.Platelets reduce anoikis and promote metastasis by activating YAP1 signaling.Mapping the STK4/Hippo signaling network in prostate cancer cell.Discovery and characterization of small molecules targeting the DNA-binding ETS domain of ERG in prostate cancer.miR-302/367/LATS2/YAP pathway is essential for prostate tumor-propagating cells and promotes the development of castration resistance.YAP triggers the Wnt/β-catenin signalling pathway and promotes enterocyte self-renewal, regeneration and tumorigenesis after DSS-induced injury.YAP1 regulates prostate cancer stem cell-like characteristics to promote castration resistant growth.DNA binding partners of YAP/TAZ.The deubiquitinase USP9X promotes tumor cell survival and confers chemoresistance through YAP1 stabilization.New possibilities for urinary molecular diagnostics.YAP and TAZ in Lung Cancer: Oncogenic Role and Clinical Targeting.Genetics and biology of prostate cancer Recent advances in prostate cancer research: large-scale genomic analyses reveal novel driver mutations and DNA repair defects

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ERG Activates the YAP1 Transcriptional Program and Induces the Development of Age-Related Prostate Tumors.

http://www.wikidata.org/entity/Q42049451

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

@zh-tw

2015年论文

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2015年论文

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2015年论文

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name

ERG Activates the YAP1 Transcr ...... f Age-Related Prostate Tumors.

@en

ERG Activates the YAP1 Transcr ...... f Age-Related Prostate Tumors.

@nl

type

label

ERG Activates the YAP1 Transcr ...... f Age-Related Prostate Tumors.

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ERG Activates the YAP1 Transcr ...... f Age-Related Prostate Tumors.

@nl

prefLabel

ERG Activates the YAP1 Transcr ...... f Age-Related Prostate Tumors.

@en

ERG Activates the YAP1 Transcr ...... f Age-Related Prostate Tumors.

@nl

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ERG Activates the YAP1 Transcr ...... of Age-Related Prostate Tumors

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Colm Morrissey

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Hamid Bolouri

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Ilsa Coleman

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Jared Lucas

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Lawrence D True

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Liem T Nguyen

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Maria S Tretiakova

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Mark R Silvis

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Olga Klezovitch

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Peter S Nelson

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P2860

TopHat2: accurate alignment of transcriptomes in the presence of insertions, deletions and gene fusions

The pro-apoptotic kinase Mst1 and its caspase cleavage products are direct inhibitors of Akt1

Mechanistic rationale for inhibition of poly(ADP-ribose) polymerase in ETS gene fusion-positive prostate cancer

An integrated network of androgen receptor, polycomb, and TMPRSS2-ERG gene fusions in prostate cancer progression

TEAD mediates YAP-dependent gene induction and growth control

Genetic and pharmacological disruption of the TEAD-YAP complex suppresses the oncogenic activity of YAP

The hippo signaling pathway in development and cancer

Transcript assembly and quantification by RNA-Seq reveals unannotated transcripts and isoform switching during cell differentiation

Expression of Yes-associated protein in common solid tumors

Transforming properties of YAP, a candidate oncogene on the chromosome 11q22 amplicon

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