Resting and anergic B cells are defective in CD28-dependent costimulation of naive CD4+ T cells.
about
Separable functions of Nef disrupt two aspects of T cell receptor machinery: CD4 expression and CD3 signalingThemis is a member of a new metazoan gene family and is required for the completion of thymocyte positive selectionBarcoding T cell calcium response diversity with methods for automated and accurate analysis of cell signals (MAAACS)Enhanced B cell expansion, survival, and humoral responses by targeting death receptor 6A mutation in a chromosome condensin II subunit, kleisin beta, specifically disrupts T cell developmentDendritic cells induce peripheral T cell unresponsiveness under steady state conditions in vivoThe SH3 domain-binding surface and an acidic motif in HIV-1 Nef regulate trafficking of class I MHC complexes.A role for LFA-1 in delaying T-lymphocyte egress from lymph nodes.Why do B cell lymphoma fail to elicit clinically sufficient T cell immune responses?The role of B7 costimulation in T-cell immunity.Production, specificity, and functionality of monoclonal antibodies to specific peptide-major histocompatibility complex class II complexes formed by processing of exogenous protein.Immune tolerance split between hepatitis B virus precore and core proteins.Restricted islet-cell reactive T cell repertoire of early pancreatic islet infiltrates in NOD mice.Using carbon magnetic nanoparticles to target, track, and manipulate dendritic cellsInducible resistance to Fas-mediated apoptosis in B cells.Unlike Th1, Th17 cells mediate sustained autoimmune inflammation and are highly resistant to restimulation-induced cell deathBystander killing of cancer requires the cooperation of CD4(+) and CD8(+) T cells during the effector phase.B cells solicit their own help from T cells.Early growth response-1 is required for CD154 transcription.All-trans retinoic acid mediates enhanced T reg cell growth, differentiation, and gut homing in the face of high levels of co-stimulationA two-step, two-signal model for the primary activation of precursor helper T cellsAntigen presentation by keratinocytes directs autoimmune skin disease.Anti-islet autoantibodies trigger autoimmune diabetes in the presence of an increased frequency of islet-reactive CD4 T cellsQuantitative reduction of the TCR adapter protein SLP-76 unbalances immunity and immune regulation.Opposing functions of the T cell receptor kinase ZAP-70 in immunity and tolerance differentially titrate in response to nucleotide substitutions.Alloimmunization to transfused platelets requires priming of CD4+ T cells in the splenic microenvironment in a murine modelLymphocyte apoptosis during early phase of Listeria infection in miceGenetic lesions in T-cell tolerance and thresholds for autoimmunity.Block and tackle: CTLA4Ig takes on lupus.The Fas/CD95 Receptor Regulates the Death of Autoreactive B Cells and the Selection of Antigen-Specific B Cells.Conformational isomers of a peptide-class II major histocompatibility complex.CD4 T cell-induced, bid-dependent apoptosis of cutaneous dendritic cells regulates T cell expansion and immune responses.Modulation of T cell development by an endogenous altered peptide ligandIg-specific T cell receptor-transgenic T cells are not deleted in the thymus and are functional in vivo.The formation of immunogenic major histocompatibility complex class II-peptide ligands in lysosomal compartments of dendritic cells is regulated by inflammatory stimuli.A kinetic threshold between negative and positive selection based on the longevity of the T cell receptor-ligand complex.Interferon-producing cells fail to induce proliferation of naive T cells but can promote expansion and T helper 1 differentiation of antigen-experienced unpolarized T cells.Failure to censor forbidden clones of CD4 T cells in autoimmune diabetes.The strategy of T cell antigen-presenting cell encounter in antigen-draining lymph nodes revealed by imaging of initial T cell activation.The strength of persistent antigenic stimulation modulates adaptive tolerance in peripheral CD4+ T cells.
P2860
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P2860
Resting and anergic B cells are defective in CD28-dependent costimulation of naive CD4+ T cells.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
1994年學術文章
@zh
1994年學術文章
@zh-hant
name
Resting and anergic B cells ar ...... ulation of naive CD4+ T cells.
@ast
Resting and anergic B cells ar ...... ulation of naive CD4+ T cells.
@en
type
label
Resting and anergic B cells ar ...... ulation of naive CD4+ T cells.
@ast
Resting and anergic B cells ar ...... ulation of naive CD4+ T cells.
@en
prefLabel
Resting and anergic B cells ar ...... ulation of naive CD4+ T cells.
@ast
Resting and anergic B cells ar ...... ulation of naive CD4+ T cells.
@en
P2093
P2860
P356
P1476
Resting and anergic B cells ar ...... mulation of naive CD4+ T cells
@en
P2093
P2860
P304
P356
10.1084/JEM.179.5.1539
P407
P577
1994-05-01T00:00:00Z