Blockade of inflammation and airway hyperresponsiveness in immune-sensitized mice by dominant-negative phosphoinositide 3-kinase-TAT - Wikidata - awgldk - TriplyDB

Blockade of inflammation and airway hyperresponsiveness in immune-sensitized mice by dominant-negative phosphoinositide 3-kinase-TAT

Blockade of inflammation and airway hyperresponsiveness in immune-sensitized mice by dominant-negative phosphoinositide 3-kinase-TAT

http://www.wikidata.org/entity/Q36371501

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The epithelial anion transporter pendrin is induced by allergy and rhinovirus infection, regulates airway surface liquid, and increases airway reactivity and inflammation in an asthma model Inhibitors of mitogen-activated protein kinases differentially regulate costimulated T cell cytokine production and mouse airway eosinophilia.Use of Cell-Penetrating Peptides in Dendritic Cell-Based Vaccination TCR-dependent translational control of GATA-3 enhances Th2 differentiation Anti-malarial drug artesunate attenuates experimental allergic asthma via inhibition of the phosphoinositide 3-kinase/Akt pathway Pulmonary mastocytosis and enhanced lung inflammation in mice heterozygous null for the Foxf1 gene Derivation and validation of murine histologic alterations resembling asthma, with two proposed histologic grade parameters.Intelectin is required for IL-13-induced monocyte chemotactic protein-1 and -3 expression in lung epithelial cells and promotes allergic airway inflammation.Inhibition of Pyk2 blocks airway inflammation and hyperresponsiveness in a mouse model of asthma.Role of SHIP-1 in the adaptive immune responses to aeroallergen in the airway Inhibition of myristoylated alanine-rich C kinase substrate (MARCKS) protein inhibits ozone-induced airway neutrophilia and inflammation.Tat-Mediated Peptide Intervention in Analgesia and Anesthesia.Synthesized OVA323-339MAP octamers mitigate OVA-induced airway inflammation by regulating Foxp3 T regulatory cells The AGC kinase inhibitor H89 attenuates airway inflammation in mouse models of asthma Key role of group v secreted phospholipase A2 in Th2 cytokine and dendritic cell-driven airway hyperresponsiveness and remodeling.Echinococcus granulosus infection reduces airway inflammation of mice likely through enhancing IL-10 and down-regulation of IL-5 and IL-17A The antioxidative effect of heat-shock protein 70 in dendritic cells.Characterisation of cell-penetrating peptide-mediated peptide delivery TAT-protein blockade during ischemia/reperfusion reveals critical role for p85 PI3K-PTEN interaction in cardiomyocyte injury.Neonatal Streptococcus pneumoniae infection may aggravate adulthood allergic airways disease in association with IL-17A.Resveratrol-mediated gamma interferon reduction prevents airway inflammation and airway hyperresponsiveness in respiratory syncytial virus-infected immunocompromised mice.FOXP3(+)Treg/Th17 cell imbalance in lung tissues of mice with asthma.Epinephrine Activation of the β2-Adrenoceptor Is Required for IL-13-Induced Mucin Production in Human Bronchial Epithelial Cells.Inhibition of Pyk2 blocks lung inflammation and injury in a mouse model of acute lung injury Intranasal Administration of Recombinant Mycobacterium smegmatis Inducing IL-17A Autoantibody Attenuates Airway Inflammation in a Murine Model of Allergic Asthma The periarterial space in the lung: its important role in lung edema, transplantation, and microbial or allergic inflammation.Caerulomycin A inhibits Th2 cell activity: a possible role in the management of asthma.Importance of group X-secreted phospholipase A2 in allergen-induced airway inflammation and remodeling in a mouse asthma model.Break on through to the other side-biophysics and cell biology shed light on cell-penetrating peptides.Recent advances in the use of protein transduction domains for the delivery of peptides, proteins and nucleic acids in vivo.Src homology 2 domain-containing inositol 5-phosphatase 1 deficiency leads to a spontaneous allergic inflammation in the murine lung.Immune Modulation of the T Cell Response in Asthma through Wnt10b.Phosphoinositide 3-kinase signalling in lung disease: leucocytes and beyond.Signal relay by CC chemokine receptor 2 (CCR2) and formylpeptide receptor 2 (Fpr2) in the recruitment of monocyte-derived dendritic cells in allergic airway inflammation Roles of integrin activation in eosinophil function and the eosinophilic inflammation of asthma.T cell activation threshold regulated by E3 ubiquitin ligase Cbl-b determines fate of inducible regulatory T cells.PI3K gamma-deficient mice have reduced levels of allergen-induced eosinophilic inflammation and airway remodeling.Cell-penetrating peptides and proteins: new inhibitors of allergic airways disease.A critical role of SHP-1 in regulation of type 2 inflammation in the lung.Investigation of the anti‑asthmatic activity of Oridonin on a mouse model of asthma.

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P2860

Blockade of inflammation and airway hyperresponsiveness in immune-sensitized mice by dominant-negative phosphoinositide 3-kinase-TAT

http://www.wikidata.org/entity/Q36371501

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2003年学术文章

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2003年學術文章

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2003年學術文章

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Blockade of inflammation and a ...... phosphoinositide 3-kinase-TAT

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Alan R Leff

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Angelo Y Meliton

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Evan Boetticher

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Jiankun Tong

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Jie Liu

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Nilda M Munoz

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Saori Myo

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Shigeharu Myou

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P2860

Type I phosphatidylinositol kinase makes a novel inositol phospholipid, phosphatidylinositol-3-phosphate

Protein kinase C isotypes controlled by phosphoinositide 3-kinase through the protein kinase PDK1

Regulation of protein kinase C zeta by PI 3-kinase and PDK-1

In vivo protein transduction: delivery of a biologically active protein into the mouse

Regulation of human eosinophil degranulation and activation by endogenous phospholipase A2.

Expression of mRNA for interleukin-5 in mucosal bronchial biopsies from asthma.

IL-4 induces mucin gene expression and goblet cell metaplasia in vitro and in vivo.

Involvement of PTEN in airway hyperresponsiveness and inflammation in bronchial asthma.

Demethoxyviridin and wortmannin block phospholipase C and D activation in the human neutrophil

The presence of interleukin 4 during in vitro priming determines the lymphokine-producing potential of CD4+ T cells from T cell receptor transgenic mice.

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10.1084/JEM.20030298

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198

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14623911

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