Transgenic inhibition of astroglial NF-κB protects from optic nerve damage and retinal ganglion cell loss in experimental optic neuritis. - Wikidata - awgldk - TriplyDB

Transgenic inhibition of astroglial NF-κB protects from optic nerve damage and retinal ganglion cell loss in experimental optic neuritis.

Transgenic inhibition of astroglial NF-κB protects from optic nerve damage and retinal ganglion cell loss in experimental optic neuritis.

http://www.wikidata.org/entity/Q36376697

about

Retinal Cell Degeneration in Animal Models Systemic simvastatin rescues retinal ganglion cells from optic nerve injury possibly through suppression of astroglial NF-κB activation Astrocytic TGF-β signaling limits inflammation and reduces neuronal damage during central nervous system Toxoplasma infection.Tumor necrosis factor-alpha mediates activation of NF-κB and JNK signaling cascades in retinal ganglion cells and astrocytes in opposite ways.IKKβ-mediated inflammatory myeloid cell activation exacerbates experimental autoimmune encephalomyelitis by potentiating Th1/Th17 cell activation and compromising blood brain barrier.Molecular pathways of pannexin1-mediated neurotoxicity.NF-κB signaling regulates myelination in the CNS.Insight into astrocyte activation after optic nerve injury.Interrelation of oxidative stress and inflammation in neurodegenerative disease: role of TNF.Withaferin A Inhibits Nuclear Factor-κB-Dependent Pro-Inflammatory and Stress Response Pathways in the Astrocytes.Control of immune-mediated pathology via the aryl hydrocarbon receptor.Virally delivered, constitutively active NFκB improves survival of injured retinal ganglion cells.Up-regulation of Wip1 involves in neuroinflammation of retinal astrocytes after optic nerve crush via NF-κB signaling pathway.Neuroprotective effect of melatonin in experimental optic neuritis in rats.Automated Axon Counting in Rodent Optic Nerve Sections with AxonJ.Anti-Inflammatory and Antioxidative Stress Effects of Oryzanol in Glaucomatous Rabbits.Astrocytes play a key role in EAE pathophysiology by orchestrating in the CNS the inflammatory response of resident and peripheral immune cells and by suppressing remyelination.Central Administration of 5Z-7-Oxozeaenol Protects Experimental Autoimmune Encephalomyelitis Mice by Inhibiting Microglia Activation.Mitochondrial DNA Double-Strand Breaks in Oligodendrocytes Cause Demyelination, Axonal Injury, and CNS Inflammation.Role for mammalian chitinase 3-like protein 1 in traumatic brain injury.Fingolimod may support neuroprotection via blockade of astrocyte nitric oxide.Optic Neuritis: A Model for the Immuno-pathogenesis of Central Nervous System Inflammatory Demyelinating Diseases.Sex chromosome contributions to sex differences in multiple sclerosis susceptibility and progression.Laquinimod protects the optic nerve and retina in an experimental autoimmune encephalomyelitis model.Role of nuclear factor κB in multiple sclerosis and experimental autoimmune encephalomyelitis

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Transgenic inhibition of astroglial NF-κB protects from optic nerve damage and retinal ganglion cell loss in experimental optic neuritis.

http://www.wikidata.org/entity/Q36376697

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Transgenic inhibition of astro ...... n experimental optic neuritis.

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Transgenic inhibition of astro ...... n experimental optic neuritis.

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type

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Transgenic inhibition of astro ...... n experimental optic neuritis.

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Transgenic inhibition of astro ...... n experimental optic neuritis.

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Journal of Neuroinflammation

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Transgenic inhibition of astro ...... in experimental optic neuritis

@en

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Galina Dvoriantchikova

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John R Bethea

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Roberta Brambilla

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Valery I Shestopalov

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P2860

Clinical practice. Optic neuritis.

Adhesion molecule expression and regulation on cells of the central nervous system.

IkappaB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-kappaB in the central nervous system

Transgenic inhibition of glial NF-kappa B reduces pain behavior and inflammation after peripheral nerve injury.

Regulation of intercellular adhesion molecule-1 (CD54) gene expression.

Transgenic inhibition of astroglial NF-kappa B leads to increased axonal sparing and sprouting following spinal cord injury.

Nuclear factor-κB activation in Schwann cells regulates regeneration and remyelination.

Vision in multiple sclerosis: the story, structure-function correlations, and models for neuroprotection.

Transgenic inhibition of astroglial NF-kappa B improves functional outcome in experimental autoimmune encephalomyelitis by suppressing chronic central nervous system inflammation

Glial NF-κB inhibition alters neuropeptide expression after sciatic nerve injury in mice.

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