Myelin basic protein-specific T helper 2 (Th2) cells cause experimental autoimmune encephalomyelitis in immunodeficient hosts rather than protect them from the disease.
about
Mast cells in the pathogenesis of multiple sclerosis and experimental autoimmune encephalomyelitisTertiary Lymphoid Organs in Central Nervous System AutoimmunityChallenging cytokine redundancy: inflammatory cell movement and clinical course of experimental autoimmune encephalomyelitis are normal in lymphotoxin-deficient, but not tumor necrosis factor-deficient, miceThe heat-stable antigen determines pathogenicity of self-reactive T cells in experimental autoimmune encephalomyelitisAbsence of monocyte chemoattractant protein 1 in mice leads to decreased local macrophage recruitment and antigen-specific T helper cell type 1 immune response in experimental autoimmune encephalomyelitisRegulatory T Cells Control Th2-Dominant Murine Autoimmune GastritisIntestinal parasitism terminates self tolerance and enhances neonatal induction of autoimmune disease and memory.Models of autoimmune demyelination in the central nervous system: on the way to translational medicine.An unexpected version of horror autotoxicus: anaphylactic shock to a self-peptide.Phenotypes of Th lineages generated by the commonly used activation with anti-CD3/CD28 antibodies differ from those generated by the physiological activation with the specific antigenSLAT/Def6 plays a critical role in the development of Th17 cell-mediated experimental autoimmune encephalomyelitis.Dissecting the role of CD4+ T cells in autoimmune diabetes through the use of TCR transgenic mice.Mechanisms of central and peripheral T-cell tolerance: lessons from experimental models of multiple sclerosis.Tolerance and autoimmunity in TCR transgenic mice specific for myelin basic protein.Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.Induction of CD4+CD25+ regulatory T cells by copolymer-I through activation of transcription factor Foxp3Treatment with anti-FcεRIα antibody exacerbates EAE and T-cell immunity against myelin.The role of TNFalpha and lymphotoxin in demyelinating disease.Allergen-specific Th1 cells fail to counterbalance Th2 cell-induced airway hyperreactivity but cause severe airway inflammation.Pathways of T cell pathology in models of chronic intestinal inflammation.Spontaneous chronic colitis in TCR alpha-mutant mice; an experimental model of human ulcerative colitis.Treatment of autoimmune premature ovarian failure.Characterization of the KRN cell transfer model of rheumatoid arthritis (KRN-CTM), a chronic yet synchronized version of the K/BxN mouseMixed periodontal Th1-Th2 cytokine profile in Actinobacillus actinomycetemcomitans-specific osteoprotegerin ligand (or RANK-L)- mediated alveolar bone destruction in vivo.Experimental autoimmune encephalitis and inflammation in the absence of interleukin-12Mast cells in autoimmune disease.Unlike Th1, Th17 cells mediate sustained autoimmune inflammation and are highly resistant to restimulation-induced cell deathSelective immunointervention in autoimmune diseases: lessons from multiple sclerosis.Myelin-specific CD8 T cells in the pathogenesis of experimental allergic encephalitis and multiple sclerosis.Th17 cells can provide B cell help in autoantibody induced arthritis.Peptide-based immunotherapy of autoimmunity: a path of puzzles, paradoxes and possibilities.Multiple elements of the allergic arm of the immune response modulate autoimmune demyelination.Microglia as liaisons between the immune and central nervous systems: functional implications for multiple sclerosis.Th1 and Th2 responses in pathogenesis and regulation of experimental autoimmune uveoretinitis.Inflammation and therapeutic vaccination in CNS diseases.Effector and regulatory T-cell subsets in autoimmunity and tissue inflammation.A tumor necrosis factor receptor 1-dependent conversation between central nervous system-specific T cells and the central nervous system is required for inflammatory infiltration of the spinal cord.Massive apoptosis in lymphoid organs in animal models for primary and secondary progressive multiple sclerosisTH1-TH2: a procrustean paradigm.Th1 and Th2 mediate acute graft-versus-host disease, each with distinct end-organ targets.
P2860
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P2860
Myelin basic protein-specific T helper 2 (Th2) cells cause experimental autoimmune encephalomyelitis in immunodeficient hosts rather than protect them from the disease.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
Myelin basic protein-specific ...... protect them from the disease.
@ast
Myelin basic protein-specific ...... protect them from the disease.
@en
type
label
Myelin basic protein-specific ...... protect them from the disease.
@ast
Myelin basic protein-specific ...... protect them from the disease.
@en
prefLabel
Myelin basic protein-specific ...... protect them from the disease.
@ast
Myelin basic protein-specific ...... protect them from the disease.
@en
P2093
P2860
P356
P1476
Myelin basic protein-specific ...... protect them from the disease.
@en
P2093
J J Lafaille
S Tonegawa
P2860
P304
P356
10.1084/JEM.186.2.307
P407
P577
1997-07-01T00:00:00Z