Endogenous IRAK-M attenuates postinfarction remodeling through effects on macrophages and fibroblasts.
about
IL-1 Receptor-Associated Kinase Signaling and Its Role in Inflammation, Cancer Progression, and Therapy ResistanceCXCR3-independent actions of the CXC chemokine CXCL10 in the infarcted myocardium and in isolated cardiac fibroblasts are mediated through proteoglycansRegulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function.PPAR agonist-induced reduction of Mcp1 in atherosclerotic plaques of obese, insulin-resistant mice depends on adiponectin-induced Irak3 expression.IRAK-M promotes alternative macrophage activation and fibroproliferation in bleomycin-induced lung injury.The inflammatory response in myocardial injury, repair, and remodelling.Thrombospondin-1 induction in the diabetic myocardium stabilizes the cardiac matrix in addition to promoting vascular rarefaction through angiopoietin-2 upregulation.The role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction.Thrombospondin-1 regulates adiposity and metabolic dysfunction in diet-induced obesity enhancing adipose inflammation and stimulating adipocyte proliferationTargeting inflammatory pathways in myocardial infarction.The pathogenesis of cardiac fibrosis.Lack of specificity of fibroblast-specific protein 1 in cardiac remodeling and fibrosis.Fibroblasts in myocardial infarction: a role in inflammation and repair.Targeting the chemokines in cardiac repair.The immune system and the remodeling infarcted heart: cell biological insights and therapeutic opportunities.Non-coding RNAs as direct and indirect modulators of epigenetic mechanism regulation of cardiac fibrosis.Intracellular signaling of cardiac fibroblasts.Inorganic nitrite modulates miRNA signatures in acute myocardial in vivo ischemia/reperfusion.Inflammation as a therapeutic target in myocardial infarction: learning from past failures to meet future challenges.Interleukin-1 in cardiac injury, repair, and remodeling: pathophysiologic and translational conceptsPathophysiology of Myocardial Infarction.Specific α7 nicotinic acetylcholine receptor agonist ameliorates isoproterenol-induced cardiac remodeling in mice through TGF-β1/Smad3 pathway.Preconditioning with recombinant high-mobility group box 1 induces ischemic tolerance in a rat model of focal cerebral ischemia-reperfusion.The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.From Inflammation to Fibrosis-Molecular and Cellular Mechanisms of Myocardial Tissue Remodelling and Perspectives on Differential Treatment Opportunities.Effect of IRAK-M on Airway Inflammation Induced by Cigarette Smoking.The role of α-smooth muscle actin in fibroblast-mediated matrix contraction and remodeling.Critical Role of IRAK-M in Regulating Antigen-Induced Airway Inflammation.Anti-inflammatory therapies in myocardial infarction: failures, hopes, and challenges.Mechanisms of Fibroblast Activation in the Remodeling Myocardium.MCP-1-induced protein attenuates post-infarct cardiac remodeling and dysfunction through mitigating NF-κB activation and suppressing inflammation-associated microRNA expression.
P2860
Q26866390-A6828693-F097-4CD9-94A4-2830FCCCB441Q34106221-8939B40B-E819-42B9-B695-2A3D85F262CDQ34356195-78F3288C-C83F-4310-9058-F5B9D3E6AE95Q34689526-62DB87A2-DD94-4499-BFCB-85369B07734EQ35256069-52C04608-9CEA-4C97-9731-6AD6B60B6575Q35529656-CE543E4F-53EA-449A-B267-260EEE7D236AQ35534242-BF9A33C4-299F-49C0-9C62-D529122D5DF1Q36399896-9EDDE873-4BA7-4428-9DE8-1D1B0CF26CC8Q37093613-1F263AED-48F0-44A2-AF70-7B8538B65494Q37100838-371C9842-5D5C-4259-A311-21D9B82059C6Q37161329-CD05A47A-A8CC-4A67-A634-B8449C3D1D37Q37342086-BD8E51FD-6A47-4C1B-BC91-1F2479AECEC4Q37716851-5EE5D0B9-CF9D-40F6-8BDF-B71AAD1B933EQ38120799-8E145084-945A-4C38-A18B-EA9E03A7C5BAQ38144845-7B44DA1C-D30A-4517-A249-195499DA5B0EQ38344492-48326F8E-2E0E-4204-A310-3E606821887EQ38423864-807FDEB2-FA78-41AE-9154-689C47EA1F91Q38438100-FB5DD60A-F58B-4C9B-B824-61CCB61E30A1Q38560476-C2501BA4-4B5B-4DBB-98E7-378332B2CD79Q38566665-208D0E1D-DFE8-4FAF-9D2F-2C9E68817E90Q38596702-9198B5BC-FC92-4857-B7FE-88F4EB0E27E9Q38667881-E2FBD058-2FC5-42F6-BD51-B33B0E6B4EB8Q38785169-A04C13EE-1065-47B8-B376-05B1F73DD488Q38875774-089DBE49-FA0F-4791-9729-EFA7E9FC97F1Q39434474-EF0729F8-041D-48AA-9D14-ED2686EC31A2Q41689612-115AC4D2-5C00-4F36-B63B-41B59E012B89Q47171988-21B41970-4372-48C2-BEB4-655E93DC748BQ47847325-46625C22-B0AC-480C-B791-B492381CAE6EQ48113765-08D148E1-4342-4DA6-BC62-18386A03813EQ50066226-FA37D748-97D5-4A35-B14F-77DE99BEEEF7Q54168046-AB41E45B-012B-403F-AF12-754598854A1B
P2860
Endogenous IRAK-M attenuates postinfarction remodeling through effects on macrophages and fibroblasts.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Endogenous IRAK-M attenuates p ...... n macrophages and fibroblasts.
@ast
Endogenous IRAK-M attenuates p ...... n macrophages and fibroblasts.
@en
type
label
Endogenous IRAK-M attenuates p ...... n macrophages and fibroblasts.
@ast
Endogenous IRAK-M attenuates p ...... n macrophages and fibroblasts.
@en
prefLabel
Endogenous IRAK-M attenuates p ...... n macrophages and fibroblasts.
@ast
Endogenous IRAK-M attenuates p ...... n macrophages and fibroblasts.
@en
P2093
P2860
P1476
Endogenous IRAK-M attenuates p ...... on macrophages and fibroblasts
@en
P2093
Amit Gupta
Amit Saxena
Dong-Wook Lee
Marcin Dobaczewski
P2860
P304
P356
10.1161/ATVBAHA.112.300310
P407
P577
2012-09-20T00:00:00Z