Correction or transfer of immunodeficiency due to TNF-LT alpha deletion by bone marrow transplantation
about
Membrane TNF confers protection to acute mycobacterial infection.Both functional LTbeta receptor and TNF receptor 2 are required for the development of experimental cerebral malariaToll-like receptor and tumour necrosis factor dependent endotoxin-induced acute lung injuryMurine cerebral malaria development is independent of toll-like receptor signalingStructural Relationship of the Lipid A Acyl Groups to Activation of Murine Toll-Like Receptor 4 by Lipopolysaccharides from Pathogenic Strains of Burkholderia mallei, Acinetobacter baumannii, and Pseudomonas aeruginosa.Membrane tumor necrosis factor confers partial protection to Listeria infection.Lymphotoxins: from cytotoxicity to lymphoid organogenesisMutant mice without B lymphocyte follicles.Lymphotoxin-alpha (LTalpha) supports development of splenic follicular structure that is required for IgG responses.Interleukin 4 gene-defective mice reconstituted with wild-type bone marrow fail to produce normal immunoglobulin E levels.Protective role of membrane tumour necrosis factor in the host's resistance to mycobacterial infection.Neutralisation of the interleukin-33/ST2 pathway ameliorates experimental colitis through enhancement of mucosal healing in mice.Fatal Mycobacterium tuberculosis infection despite adaptive immune response in the absence of MyD88.The NLRP3 inflammasome is activated by nanoparticles through ATP, ADP and adenosine.Crucial role of tumor necrosis factor receptor 1 expression on nonhematopoietic cells for B cell localization within the splenic white pulp.IL-1R1/MyD88 signaling and the inflammasome are essential in pulmonary inflammation and fibrosis in mice.Long-term control of Mycobacterium bovis BCG infection in the absence of Toll-like receptors (TLRs): investigation of TLR2-, TLR6-, or TLR2-TLR4-deficient miceToll-like receptor 2 is required for optimal control of Listeria monocytogenes infectionRelative contribution of IL-1α, IL-1β and TNF to the host response to Mycobacterium tuberculosis and attenuated M. bovis BCG.Hypoacylated LPS from Foodborne Pathogen Campylobacter jejuni Induces Moderate TLR4-Mediated Inflammatory Response in Murine Macrophages.GM-CSF targeted immunomodulation affects host response to M. tuberculosis infection.Cell iron status influences macrophage polarization.
P2860
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P2860
Correction or transfer of immunodeficiency due to TNF-LT alpha deletion by bone marrow transplantation
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Correction or transfer of immu ...... by bone marrow transplantation
@ast
Correction or transfer of immu ...... by bone marrow transplantation
@en
type
label
Correction or transfer of immu ...... by bone marrow transplantation
@ast
Correction or transfer of immu ...... by bone marrow transplantation
@en
prefLabel
Correction or transfer of immu ...... by bone marrow transplantation
@ast
Correction or transfer of immu ...... by bone marrow transplantation
@en
P2093
P2860
P1433
P1476
Correction or transfer of immu ...... by bone marrow transplantation
@en
P2093
Di Padova F
Eugster HP
Quesniaux VF
P2860
P304
P407
P577
1996-03-01T00:00:00Z