The role of the persistent Na(+) current during cardiac ischemia and hypoxia.
about
Na⁺ transport in the normal and failing heart - remember the balanceThe role of late I Na in development of cardiac arrhythmiasRegulation of intracellular Na(+) in health and disease: pathophysiological mechanisms and implications for treatmentCa2+ Sparks and Ca2+ waves are the subcellular events underlying Ca2+ overload during ischemia and reperfusion in perfused intact hearts.Background sodium current underlying respiratory rhythm regularity.Icariin, a Novel Blocker of Sodium and Calcium Channels, Eliminates Early and Delayed Afterdepolarizations, As Well As Triggered Activity, in Rabbit CardiomyocytesResveratrol attenuates the Na(+)-dependent intracellular Ca(2+) overload by inhibiting H(2)O(2)-induced increase in late sodium current in ventricular myocytesNF-kappaB-dependent transcriptional regulation of the cardiac scn5a sodium channel by angiotensin II.Extracellular proton depression of peak and late Na⁺ current in the canine left ventricle.Inhibition of late sodium current to reduce electrical and mechanical dysfunction of ischaemic myocardium.The cardiac persistent sodium current: an appealing therapeutic target?F 15845 inhibits persistent sodium current in the heart and prevents angina in animal modelsThe arrhythmogenic consequences of increasing late INa in the cardiomyocyte.Regulation of cellular gas exchange, oxygen sensing, and metabolic control.Acute myocardial ischemia: cellular mechanisms underlying ST segment elevation.Inhibition of the cardiac Na⁺ channel α-subunit Nav1.5 by propofol and dexmedetomidine.NaV Channels: Assaying Biosynthesis, Trafficking, Function.Persistent sodium current and Na+/H+ exchange contributes to the augmentation of the reverse Na+/Ca2+ exchange during hypoxia or acute ischemia in ventricular myocytes.Calmodulin kinase II and protein kinase C mediate the effect of increased intracellular calcium to augment late sodium current in rabbit ventricular myocytes.Mechanism of Action Potential Prolongation During Metabolic Inhibition in the Whole Rabbit Heart
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P2860
The role of the persistent Na(+) current during cardiac ischemia and hypoxia.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
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2006年學術文章
@zh-hant
name
The role of the persistent Na(+) current during cardiac ischemia and hypoxia.
@ast
The role of the persistent Na(+) current during cardiac ischemia and hypoxia.
@en
type
label
The role of the persistent Na(+) current during cardiac ischemia and hypoxia.
@ast
The role of the persistent Na(+) current during cardiac ischemia and hypoxia.
@en
prefLabel
The role of the persistent Na(+) current during cardiac ischemia and hypoxia.
@ast
The role of the persistent Na(+) current during cardiac ischemia and hypoxia.
@en
P2860
P1476
The role of the persistent Na(+) current during cardiac ischemia and hypoxia.
@en
P2093
David A Saint
P2860
P304
P356
10.1111/J.1540-8167.2006.00390.X
P478
17 Suppl 1
P577
2006-05-01T00:00:00Z