about
Dual role for Saccharomyces cerevisiae Tel1 in the checkpoint response to double-strand breaks.Dephosphorylation of gamma H2A by Glc7/protein phosphatase 1 promotes recovery from inhibition of DNA replication.Association of new deletion/duplication region at chromosome 1p21 with intellectual disability, severe speech deficit and autism spectrum disorder-like behavior: an all-in approach to solving the DPYD enigmaTranscriptional profile of the homologous recombination machinery and characterization of the EhRAD51 recombinase in response to DNA damage in Entamoeba histolytica.Analysis of fine-scale mammalian evolutionary breakpoints provides new insight into their relation to genome organisation.Potential for genomic instability associated with retrotranspositionally-incompetent L1 loci.Rad9 interacts with Aft1 to facilitate genome surveillance in fragile genomic sites under non-DNA damage-inducing conditions in S. cerevisiaeSchistosoma comparative genomics: integrating genome structure, parasite biology and anthelmintic discovery.Evaluation of DNA double strand breaks repair efficiency in head and neck cancer.MRX-dependent DNA damage response to short telomeres.A Conserved DNA Repeat Promotes Selection of a Diverse Repertoire of Trypanosoma brucei Surface Antigens from the Genomic Archive.Role of the Saccharomyces cerevisiae Rad53 checkpoint kinase in signaling double-strand breaks during the meiotic cell cycleDNA damage response at functional and dysfunctional telomeresL1 mobile element expression causes multiple types of toxicity.Mechanisms and regulation of DNA end resection.The DNA damage checkpoint response to replication stress: A Game of Forks.Break dosage, cell cycle stage and DNA replication influence DNA double strand break response.Decondensation of chromosomal 45S rDNA sites in Lolium and Festuca genotypes does not result in karyotype instability.The MRX complex plays multiple functions in resection of Yku- and Rif2-protected DNA endsDominant TEL1-hy mutations compensate for Mec1 lack of functions in the DNA damage response.The Yku70-Yku80 complex contributes to regulate double-strand break processing and checkpoint activation during the cell cycleImpact of sodium arsenite on chromosomal aberrations with respect to polymorphisms of detoxification and DNA repair genes.Locus-specific control of DNA resection and suppression of subtelomeric VSG recombination by HAT3 in the African trypanosome.
P2860
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P2860
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
The cellular response to chromosome breakage.
@ast
The cellular response to chromosome breakage.
@en
type
label
The cellular response to chromosome breakage.
@ast
The cellular response to chromosome breakage.
@en
prefLabel
The cellular response to chromosome breakage.
@ast
The cellular response to chromosome breakage.
@en
P2860
P1476
The cellular response to chromosome breakage
@en
P2093
Davide Mantiero
Michela Clerici
P2860
P304
P356
10.1111/J.1365-2958.2006.05186.X
P407
P577
2006-06-01T00:00:00Z