Increased chondroitin sulfate proteoglycan expression in denervated brainstem targets following spinal cord injury creates a barrier to axonal regeneration overcome by chondroitinase ABC and neurotrophin-3.
about
Astrocytes as a source for extracellular matrix molecules and cytokinesTransplantation of specific human astrocytes promotes functional recovery after spinal cord injuryThe proteoglycan brevican binds to fibronectin after proteolytic cleavage and promotes glioma cell motilityMyelin damage and repair in pathologic CNS: challenges and prospectsPrecursor cell biology and the development of astrocyte transplantation therapies: lessons from spinal cord injuryTranslational spinal cord injury research: preclinical guidelines and challengesElucidating the Role of Injury-Induced Electric Fields (EFs) in Regulating the Astrocytic Response to Injury in the Mammalian Central Nervous SystemChondroitin sulphate N-acetylgalactosaminyl-transferase-1 inhibits recovery from neural injuryThe laser lesion of the mouse visual cortex as a model to study neural extracellular matrix remodeling during degeneration, regeneration and plasticity of the CNS.In vivo diffusion tensor imaging of thoracic and cervical rat spinal cord at 7 TPeripheral nerve grafts after cervical spinal cord injury in adult cats.Immature astrocytes promote CNS axonal regeneration when combined with chondroitinase ABCNeurotrophin treatment to promote regeneration after traumatic CNS injury.Plasticity after spinal cord injury: relevance to recovery and approaches to facilitate itA systematic review of directly applied biologic therapies for acute spinal cord injury.Grafted neural progenitors integrate and restore synaptic connectivity across the injured spinal cordCombined delivery of Nogo-A antibody, neurotrophin-3 and the NMDA-NR2d subunit establishes a functional 'detour' in the hemisected spinal cord.Local Delivery of High-Dose Chondroitinase ABC in the Sub-Acute Stage Promotes Axonal Outgrowth and Functional Recovery after Complete Spinal Cord Transection.Chondroitinase ABC promotes selective reactivation of somatosensory cortex in squirrel monkeys after a cervical dorsal column lesionThe yellow fluorescent protein (YFP-H) mouse reveals neuroprotection as a novel mechanism underlying chondroitinase ABC-mediated repair after spinal cord injury.An inhibitor of chondroitin sulfate proteoglycan synthesis promotes central nervous system remyelination.GDNF selectively promotes regeneration of injury-primed sensory neurons in the lesioned spinal cord.Axonal regeneration induced by blockade of glial inhibitors coupled with activation of intrinsic neuronal growth pathwaysLoss and spontaneous recovery of forelimb evoked potentials in both the adult rat cuneate nucleus and somatosensory cortex following contusive cervical spinal cord injuryThe assessment of adeno-associated vectors as potential intrinsic treatments for brainstem axon regeneration.Expression and activation of ephexin is altered after spinal cord injury.Role of chondroitin sulfate proteoglycans in axonal conduction in Mammalian spinal cord.Alterations in chondroitin sulfate proteoglycan expression occur both at and far from the site of spinal contusion injury.Chondroitinase ABC promotes compensatory sprouting of the intact corticospinal tract and recovery of forelimb function following unilateral pyramidotomy in adult mice.Rolipram attenuates acute oligodendrocyte death in the adult rat ventrolateral funiculus following contusive cervical spinal cord injury.Infarct-derived chondroitin sulfate proteoglycans prevent sympathetic reinnervation after cardiac ischemia-reperfusion injuryCortical and subcortical plasticity in the brains of humans, primates, and rats after damage to sensory afferents in the dorsal columns of the spinal cord.Growth factors and combinatorial therapies for CNS regenerationChondroitinase ABC combined with neurotrophin NT-3 secretion and NR2D expression promotes axonal plasticity and functional recovery in rats with lateral hemisection of the spinal cordAlterations in sulfated chondroitin glycosaminoglycans following controlled cortical impact injury in mice.Matrix metalloproteinase-9 facilitates glial scar formation in the injured spinal cordChondroitinase ABC-mediated plasticity of spinal sensory function.Synaptic plasticity, neurogenesis, and functional recovery after spinal cord injury.Combination of engineered Schwann cell grafts to secrete neurotrophin and chondroitinase promotes axonal regeneration and locomotion after spinal cord injury.Construction of pathways to promote axon growth within the adult central nervous system.
P2860
Q21129284-90D88DFB-4EB4-4239-BB5D-9E2436B7C7E8Q21135533-B9E68185-8AEB-4515-ADF0-A84A271431D1Q24607682-CF60DAFE-BF0D-4F6A-8EC6-80EE837F773DQ26797407-CEB16188-6106-49D3-95D0-2511FB5ED21DQ27004382-9EC466EE-8259-491B-A74F-55A73ED7F067Q27025285-D3A979B4-4F82-4CB9-B594-D4E9C97AAC85Q27344252-54985FBE-B693-4035-8391-313E88BA60F1Q27682773-2B7F7E6A-4BCD-4705-A890-CEED4203679BQ31046527-DA6A5437-110E-4F3F-A072-28503B35B9BCQ33466632-12C7FD86-F84B-438A-BBB4-EFFD246ED4A0Q34069201-9E3F88DC-5F2E-4D96-9725-F459828672A7Q34134519-426DFBFF-0A88-4256-B129-51B27A9360A3Q34547330-91ADEA73-2D24-406D-98CA-A67B8FFA51F7Q35006452-62C6EDDF-1637-48B1-A83D-249392512221Q35127473-12B2C3B1-5740-4CB9-8561-374FC1A3D036Q35142642-EBC2276A-B345-4E46-9389-6ADF210C31BFQ35375651-BB1AFF9C-60DB-4FF1-A92D-084A500378E0Q35783366-1A9977F9-022C-4D7D-A299-AF81EABF1D84Q35786927-4C2F3EB3-40AB-4346-A5E4-C69C416953BCQ35923626-87FB5EF8-9B08-4A4C-BCC3-6373E063B746Q35999574-79AE8598-B0BC-400D-BDDB-A5D45E74934EQ36082822-9816F405-CC84-47D6-8AC7-E35FE96D8932Q36162213-5E482896-53CE-4710-A179-0B24FEF67209Q36276528-B1088EB5-7E1D-479E-99B2-5B5580574866Q36337994-A932B37F-C9F0-4493-BDF0-FE708BD95AAEQ36445643-96651298-3CBA-426B-B3E7-A2FD8CB6404AQ36493205-01651F8A-2072-452E-B754-60871862B980Q36807055-1F7ED251-03C4-4817-A6C2-3CC740879D26Q36852309-7C00F626-DB8F-48F6-9ACF-1555E4BE4B49Q36864853-A069DAE3-498B-449E-8503-72F82E5896EFQ36900816-8ACA89B6-B9AB-404E-828C-E9BE12FFC3F9Q36907946-F12233AD-7095-48FD-98C5-D519A6259F44Q36967422-71883ECA-2EFE-4B46-B6FD-66B009E6B5C7Q37135719-DD4A7DB5-5948-4D6C-B897-8ECCDF7584C9Q37249700-4BA3DFE6-35C4-4786-85D0-434C2CF84A01Q37265996-09B0AC88-F706-4612-9021-FE0C28AEF1D0Q37352467-28942E9F-00AB-4CDB-9032-3923A7F2B427Q37421067-EE4699FD-2032-4127-826D-88EB1BA00284Q37526111-FB7ED3B2-E60A-4EBD-9D3D-53DC95435BEFQ37765694-661E90DB-A3FB-44CF-9324-D615047F8599
P2860
Increased chondroitin sulfate proteoglycan expression in denervated brainstem targets following spinal cord injury creates a barrier to axonal regeneration overcome by chondroitinase ABC and neurotrophin-3.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Increased chondroitin sulfate ...... tinase ABC and neurotrophin-3.
@ast
Increased chondroitin sulfate ...... tinase ABC and neurotrophin-3.
@en
type
label
Increased chondroitin sulfate ...... tinase ABC and neurotrophin-3.
@ast
Increased chondroitin sulfate ...... tinase ABC and neurotrophin-3.
@en
prefLabel
Increased chondroitin sulfate ...... tinase ABC and neurotrophin-3.
@ast
Increased chondroitin sulfate ...... tinase ABC and neurotrophin-3.
@en
P2093
P2860
P1476
Increased chondroitin sulfate ...... tinase ABC and neurotrophin-3.
@en
P2093
Alicia L Yonkof
Christopher M Whitaker
James M Massey
Jeremy Amps
Jerry Silver
Mariano S Viapiano
Michelle R Wagoner
Nigel G F Cooper
Russell T Matthews
Stephen M Onifer
P2860
P304
P356
10.1016/J.EXPNEUROL.2007.03.029
P407
P577
2007-04-12T00:00:00Z