beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure - Wikidata - awgldk - TriplyDB

beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

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Inhibition of the Hantavirus Fusion Process by Predicted Domain III and Stem Peptides from Glycoprotein Gc The Role of Phlebovirus Glycoproteins in Viral Entry, Assembly and Release What Do We Know about How Hantaviruses Interact with Their Different Hosts?Early Bunyavirus-Host Cell Interactions Hemorrhagic Fever with Renal Syndrome: Pathogenesis and Clinical Picture Hantaviral Proteins: Structure, Functions, and Role in Hantavirus Infection The Syrian hamster model of hantavirus pulmonary syndrome Hantavirus infection induces the expression of RANTES and IP-10 without causing increased permeability in human lung microvascular endothelial cells Thottapalayam Virus, a Prototype Shrewborne Hantavirus Kaposi's Sarcoma-Associated Herpesvirus Forms a Multimolecular Complex of Integrins ( V 5, V 3, and 3 1) and CD98-xCT during Infection of Human Dermal Microvascular Endothelial Cells, and CD98-xCT Is Essential for the Postentry Stage of Infection A global perspective on hantavirus ecology, epidemiology, and disease Human Parechovirus 1 Infection Occurs via αVβ1 Integrin Endocytic Pathways Used by Andes Virus to Enter Primary Human Lung Endothelial Cells Hantavirus protein interactions regulate cellular functions and signaling responses.Basolateral entry and release of Crimean-Congo hemorrhagic fever virus in polarized MDCK-1 cells Meeting report: Tenth International Conference on Hantaviruses.Electron cryotomography of Tula hantavirus suggests a unique assembly paradigm for enveloped viruses.Peptide antagonists that inhibit Sin Nombre virus and hantaan virus entry through the beta3-integrin receptor [Hantavirus pulmonary and cardiovascular syndrome: epidemiology, clinical presentation, laboratory diagnosis and treatment]Pathogenic hantaviruses direct the adherence of quiescent platelets to infected endothelial cells.Severe fever with thrombocytopenia virus glycoproteins are targeted by neutralizing antibodies and can use DC-SIGN as a receptor for pH-dependent entry into human and animal cell lines Hypoxia induces permeability and giant cell responses of Andes virus-infected pulmonary endothelial cells by activating the mTOR-S6K signaling pathway.Systematic review of severe fever with thrombocytopenia syndrome: virology, epidemiology, and clinical characteristics.Vascular events in viral hemorrhagic fevers: a comparative study of dengue and hantaviruses.Platelets interact with Coxsackieviruses B and have a critical role in the pathogenesis of virus-induced myocarditis.Evidence that Processing of the Severe Fever with Thrombocytopenia Syndrome Virus Gn/Gc Polyprotein Is Critical for Viral Infectivity and Requires an Internal Gc Signal Peptide.Phage display selection of cyclic peptides that inhibit Andes virus infection.Identification of oligopeptides mimicking the receptor-binding domain of Hantaan virus envelope glycoprotein from a phage-displayed peptide library.Andes virus recognition of human and Syrian hamster beta3 integrins is determined by an L33P substitution in the PSI domain.Immunogenetic factors affecting susceptibility of humans and rodents to hantaviruses and the clinical course of hantaviral disease in humans.Role of alpha(v) integrins in adenovirus cell entry and gene delivery.The role of viral genomic RNA and nucleocapsid protein in the autophagic clearance of hantavirus glycoprotein Gn.Hantavirus interferon regulation and virulence determinants Pathogenic hantaviruses bind plexin-semaphorin-integrin domains present at the apex of inactive, bent alphavbeta3 integrin conformers Temporal and spatial analysis of Sin Nombre virus quasispecies in naturally infected rodents β2 integrin mediates hantavirus-induced release of neutrophil extracellular traps.Cell adhesion molecules in the pathogenesis of and host defence against microbial infection.Pathogenic hantaviruses Andes virus and Hantaan virus induce adherens junction disassembly by directing vascular endothelial cadherin internalization in human endothelial cells Hantaviruses induce cell type- and viral species-specific host microRNA expression signatures.Genome-wide small interfering RNA screens reveal VAMP3 as a novel host factor required for Uukuniemi virus late penetration

P2860

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P2860

beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

http://www.wikidata.org/entity/Q36507782

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beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

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beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

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beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

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beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

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beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

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beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

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beta3 Integrins mediate the cellular entry of hantaviruses that cause respiratory failure

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E R Mackow

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I N Gavrilovskaya

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M H Ginsberg

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M Shepley

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R Shaw

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P2860

Identification of the alpha6 integrin as a candidate receptor for papillomaviruses

Integrins: versatility, modulation, and signaling in cell adhesion

Integrins: emerging paradigms of signal transduction

Inside-out signaling through integrins

Integrins and signal transduction pathways: the road taken

Integrins alpha v beta 3 and alpha v beta 5 promote adenovirus internalization but not virus attachment

Lymphocyte homing and homeostasis

Hantavirus pulmonary syndrome in the United States: a pathological description of a disease caused by a new agent.

Hantavirus pulmonary syndrome. Pathogenesis of an emerging infectious disease.

The integrin VLA-2 binds echovirus 1 and extracellular matrix ligands by different mechanisms.

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