CCAAT/enhancer binding protein (C/EBP) sites are required for HIV-1 replication in primary macrophages but not CD4(+) T cells.
about
Genome-wide association study identifies single nucleotide polymorphism in DYRK1A associated with replication of HIV-1 in monocyte-derived macrophagesTranscription factor binding sites in the pol gene intragenic regulatory region of HIV-1 are important for virus infectivityThe macrophage: a therapeutic target in HIV-1 infectionAnti-CD45RO suppresses human immunodeficiency virus type 1 replication in microglia: role of Hck tyrosine kinase and implications for AIDS dementia.Novel function of prothymosin alpha as a potent inhibitor of human immunodeficiency virus type 1 gene expression in primary macrophagesCell-type-specific proteome and interactome: using HIV-1 Tat as a test case.Coordinated regulation of SIV replication and immune responses in the CNS.A novel HIV-1-encoded microRNA enhances its viral replication by targeting the TATA box region.Regulation of SIV mac 239 basal long terminal repeat activity and viral replication in macrophages: functional roles of two CCAAT/enhancer-binding protein beta sites in activation and interferon beta-mediated suppression.Functional interactions between C/EBP, Sp1, and COUP-TF regulate human immunodeficiency virus type 1 gene transcription in human brain cells.Interleukin-16 inhibits human immunodeficiency virus type 1 entry and replication in macrophages and in dendritic cellsGenetic variation and HIV-associated neurologic disease.Host hindrance to HIV-1 replication in monocytes and macrophages.Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineageTranscriptional correlates of disease outcome in anticoagulant-treated non-human primates infected with ebolavirus.Neuropathogenic SIVsmmFGb genetic diversity and selection-induced tissue-specific compartmentalization during chronic infection and temporal evolution of viral genes in lymphoid tissues and regions of the central nervous system.Effects of forskolin on Kupffer cell production of interleukin-10 and tumor necrosis factor alpha differ from those of endogenous adenylyl cyclase activators: possible role for adenylyl cyclase 9.Defining differential genetic signatures in CXCR4- and the CCR5-utilizing HIV-1 co-linear sequences.Structural and functional studies of CCAAT/enhancer binding sites within the human immunodeficiency virus type 1 subtype C LTR.Early emergence and selection of a SIV-LTR C/EBP site variant in SIV-infected macaques that increases virus infectivityInnate and adaptive factors regulating human immunodeficiency virus type 1 genomic activation.Development of co-selected single nucleotide polymorphisms in the viral promoter precedes the onset of human immunodeficiency virus type 1-associated neurocognitive impairment.MicroRNA regulation of IFN-beta protein expression: rapid and sensitive modulation of the innate immune response.HIV-1 and bacterial pneumonia in the era of antiretroviral therapy.Rev-dependent lentiviral expression vectorArc of a vicious circle: pathways activated by Mycobacterium tuberculosis that target the HIV-1 long terminal repeat.Impact of Tat Genetic Variation on HIV-1 DiseaseCSF-induced and HIV-1-mediated distinct regulation of Hck and C/EBPbeta represent a heterogeneous susceptibility of monocyte-derived macrophages to M-tropic HIV-1 infection.Type I interferon induces inhibitory 16-kD CCAAT/ enhancer binding protein (C/EBP)beta, repressing the HIV-1 long terminal repeat in macrophages: pulmonary tuberculosis alters C/EBP expression, enhancing HIV-1 replication.CCAAT/enhancer binding protein beta is a major mediator of inflammation and viral replication in the gastrointestinal tract of simian immunodeficiency virus-infected rhesus macaquesErythromycin derivatives inhibit HIV-1 replication in macrophages through modulation of MAPK activity to induce small isoforms of C/EBPbetaHIV-1 Tat mediates degradation of RON receptor tyrosine kinase, a regulator of inflammation.Thioredoxin reductase-1 negatively regulates HIV-1 transactivating protein Tat-dependent transcription in human macrophagesDeployment of the human immunodeficiency virus type 1 protein arsenal: combating the host to enhance viral transcription and providing targets for therapeutic developmentInterleukin-10 induces inhibitory C/EBPbeta through STAT-3 and represses HIV-1 transcription in macrophagesCCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.Role of macrophages in HIV infection and persistence.Interaction between CCAAT/enhancer binding protein and cyclic AMP response element binding protein 1 regulates human immunodeficiency virus type 1 transcription in cells of the monocyte/macrophage lineage.Endothelial cells enhance human immunodeficiency virus type 1 replication in macrophages through a C/EBP-dependent mechanism.Molecular Understanding of HIV-1 Latency.
P2860
Q21135536-6D41BAA4-6767-473A-82F4-3FDE81B0D9E6Q24815940-0CECF19D-E7F5-437D-B2D9-6C02C1142167Q26823236-B1029305-A669-41B4-A6C4-B3E52243F287Q30160016-4DC48957-ACC1-423B-8598-B1873D1698D6Q30445480-CB202D5E-E828-4941-8D45-790AF4E2052CQ33347964-FB51CBB0-21E8-4FE7-84AE-355CF92D6A51Q33519523-02B6AB28-DA3C-4D13-B73E-550F7F292CB1Q33556837-F2A266FF-366C-4FC8-95DB-AC6F15FCCDEEQ33593821-6B4F3874-ECA0-4372-97B2-B10E4FBDEF58Q33794329-99D69362-5020-4213-98A5-E19A9CB96AA2Q33818097-5C10DC93-92C2-4B30-901B-32939A50D4E5Q33819189-E8FCB310-FC89-4649-B90B-B072751B66C8Q33846376-51A9A746-0D33-48F5-B196-191C230C80CEQ33864438-A9CE07D7-0CB6-46D0-9BED-C7FF7D6C5F24Q33981963-EB314961-8E6E-4E6E-93BE-9225EF78CB77Q34100200-DA4F8697-7AEA-469F-94BB-6E0871C7A757Q34111956-54E19973-0DD7-4CE2-BF29-F2345D23C4BCQ34267043-F887FF1B-6A32-4BE1-810B-E8E54F31828DQ34387337-EB139128-4FDC-4DF8-8C16-AD7449F3B80CQ34404828-2489FD45-63D6-46BC-8825-63F25960D4D3Q34671609-7B751D56-9535-47DC-88E6-B91EF744EF2AQ34671618-CDBA9B69-F291-4D09-B5F6-B08658540E20Q34793627-BEC1997D-24CD-4536-99C2-40EFF72E7564Q35095031-4DAD1B96-F365-4A78-A8BC-1ED248FD502BQ35634169-7A87DD3E-06E2-45E9-BF2A-C0A4596ABBB8Q35684805-0D10CBB8-86BD-458F-85AB-7B1A45FBBD49Q36148507-CCC9F83D-D391-4CFC-8816-F4CB7C3F85C0Q36371384-F116F521-9D0E-48B2-8D20-D8421806A1E4Q36401561-A5772357-673F-4F66-8636-0F90784085D6Q36734869-CF8BEFB9-6630-4E9A-AE1C-76AE10984DF5Q36858639-87DC0898-9DBA-4F7C-9E16-54DDB80302B2Q36877203-3FBEA03D-A69F-4471-AD8B-55F3B599471EQ36980967-81034CE4-25F5-4A9F-AF0C-0938D049D7E8Q37127522-E85E3854-5ECA-4CC2-AA09-ECF367AD21DDQ37273948-D3522EDC-A87C-458B-B439-00A2DE44C318Q37425262-C169D3F3-0F44-446A-8FF8-97399F809407Q37755250-5AD9BC5E-BC24-46F0-A5DC-9C0C234B4013Q39602178-C2266856-0002-402B-81E7-8A8803212F20Q39604381-07F5FF49-5C06-442D-961B-9BB74EA6F1B4Q39622803-723726AB-258A-42DC-872C-29A924F7861E
P2860
CCAAT/enhancer binding protein (C/EBP) sites are required for HIV-1 replication in primary macrophages but not CD4(+) T cells.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
CCAAT/enhancer binding protein ...... phages but not CD4(+) T cells.
@ast
CCAAT/enhancer binding protein ...... phages but not CD4(+) T cells.
@en
type
label
CCAAT/enhancer binding protein ...... phages but not CD4(+) T cells.
@ast
CCAAT/enhancer binding protein ...... phages but not CD4(+) T cells.
@en
prefLabel
CCAAT/enhancer binding protein ...... phages but not CD4(+) T cells.
@ast
CCAAT/enhancer binding protein ...... phages but not CD4(+) T cells.
@en
P2860
P356
P1476
CCAAT/enhancer binding protein ...... ophages but not CD4(+) T cells
@en
P2093
K L Calame
P2860
P304
P356
10.1073/PNAS.94.16.8714
P407
P577
1997-08-01T00:00:00Z