The contribution of c-Jun N-terminal kinase activation and subsequent Bcl-2 phosphorylation to apoptosis induction in human B-cells is dependent on the mode of action of specific stresses
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Vinblastine induces acute, cell cycle phase-independent apoptosis in some leukemias and lymphomas and can induce acute apoptosis in others when Mcl-1 is suppressed.2-Phenyl-5-(pyrrolidin-1-yl)-1-(3,4,5-trimethoxybenzyl)-1H-benzimidazole, a benzimidazole derivative, inhibits growth of human prostate cancer cells by affecting tubulin and c-Jun N-terminal kinase.S-phase arrest after vincristine treatment may promote hepatitis B virus replicationVinblastine sensitizes leukemia cells to cyclin-dependent kinase inhibitors, inducing acute cell cycle phase-independent apoptosisRIPK1 regulates survival of human melanoma cells upon endoplasmic reticulum stress through autophagy.Phosphorylation of Glutathione S-Transferase P1 (GSTP1) by Epidermal Growth Factor Receptor (EGFR) Promotes Formation of the GSTP1-c-Jun N-terminal kinase (JNK) Complex and Suppresses JNK Downstream Signaling and Apoptosis in Brain Tumor Cells.Involvement of oxidative stress and caspase 2-mediated intrinsic pathway signaling in age-related increase in muscle cell apoptosis in mice.Anti-apoptotic signaling as a cytoprotective mechanism in mammalian hibernation.Microtubule destabilising agents: far more than just antimitotic anticancer drugs.AMP-activated protein kinase couples 3-bromopyruvate-induced energy depletion to apoptosis via activation of FoxO3a and upregulation of proapoptotic Bcl-2 proteins.Valproic acid sensitizes TRAIL-resistant anaplastic thyroid carcinoma cells to apoptotic cell death.Notoginsenoside R1 Protects against Neonatal Cerebral Hypoxic-Ischemic Injury through Estrogen Receptor-Dependent Activation of Endoplasmic Reticulum Stress Pathways.Glutathione S-transferase P1-1 as a target for mesothelioma treatment.Inactivation of Bcl-2 through IκB kinase (IKK)-dependent phosphorylation mediates apoptosis upon exposure to 4-hydroxynonenal (HNE).
P2860
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P2860
The contribution of c-Jun N-terminal kinase activation and subsequent Bcl-2 phosphorylation to apoptosis induction in human B-cells is dependent on the mode of action of specific stresses
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
The contribution of c-Jun N-te ...... of action of specific stresses
@ast
The contribution of c-Jun N-te ...... of action of specific stresses
@en
type
label
The contribution of c-Jun N-te ...... of action of specific stresses
@ast
The contribution of c-Jun N-te ...... of action of specific stresses
@en
prefLabel
The contribution of c-Jun N-te ...... of action of specific stresses
@ast
The contribution of c-Jun N-te ...... of action of specific stresses
@en
P2860
P1476
The contribution of c-Jun N-te ...... of action of specific stresses
@en
P2093
Donna E Muscarella
Stephen E Bloom
P2860
P304
P356
10.1016/J.TAAP.2007.11.032
P577
2007-12-14T00:00:00Z