Varicella-zoster virus (VZV) open reading frame 10 protein, the homolog of the essential herpes simplex virus protein VP16, is dispensable for VZV replication in vitro.
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Herpes simplex virus and varicella zoster virus, the house guests who never leaveHigh-level expression of Marek's disease virus glycoprotein C is detrimental to virus growth in vitroImproving the yeast two-hybrid system with permutated fusions proteins: the Varicella Zoster Virus interactome.Varicella-zoster virusA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationGenome-wide mutagenesis reveals that ORF7 is a novel VZV skin-tropic factorRole of the varicella-zoster virus gene product encoded by open reading frame 35 in viral replication in vitro and in differentiated human skin and T cells in vivo.Evidence that herpes simplex virus VP16 is required for viral egress downstream of the initial envelopment event.Characterization of Marek's disease virus serotype 1 (MDV-1) deletion mutants that lack UL46 to UL49 genes: MDV-1 UL49, encoding VP22, is indispensable for virus growth.Varicella-zoster virus open reading frame 10 is a virulence determinant in skin cells but not in T cells in vivoVaricella zoster virus latency.Analysis of the interaction between the essential herpes simplex virus 1 tegument proteins VP16 and VP1/2.Cloning the simian varicella virus genome in E. coli as an infectious bacterial artificial chromosomeIdentification of non-essential loci within the Meleagrid herpesvirus 1 genome.The ubiquitous cellular transcriptional factor USF targets the varicella-zoster virus open reading frame 10 promoter and determines virulence in human skin xenografts in SCIDhu mice in vivo.The transcriptional regulatory proteins encoded by varicella-zoster virus open reading frames (ORFs) 4 and 63, but not ORF 61, are associated with purified virus particles.Herpes simplex virus VP16 rescues viral mRNA from destruction by the virion host shutoff functionImmunization with recombinant varicella-zoster virus expressing herpes simplex virus type 2 glycoprotein D reduces the severity of genital herpes in guinea pigs.Identification and characterization of a small modular domain in the herpes simplex virus host shutoff protein sufficient for interaction with VP16.Horizontal transmission of Marek's disease virus requires US2, the UL13 protein kinase, and gCRodent models of varicella-zoster virus neurotropism.The varicella-zoster virus genomeMarek's disease virus expresses multiple UL44 (gC) variants through mRNA splicing that are all required for efficient horizontal transmission.Varicella-zoster virus ORF47 protein kinase, which is required for replication in human T cells, and ORF66 protein kinase, which is expressed during latency, are dispensable for establishment of latency.ORF9p phosphorylation by ORF47p is crucial for the formation and egress of varicella-zoster virus viral particles.The Marek's disease virus (MDV) protein encoded by the UL17 ortholog is essential for virus growth.Genetic profile of an Oka varicella vaccine virus variant isolated from an infant with zoster.Functions of the ORF9-to-ORF12 gene cluster in varicella-zoster virus replication and in the pathogenesis of skin infectionEssential role played by the C-terminal domain of glycoprotein I in envelopment of varicella-zoster virus in the trans-Golgi network: interactions of glycoproteins with tegument.The UL48 tegument protein of pseudorabies virus is critical for intracytoplasmic assembly of infectious virionsThe pseudorabies virus VP22 homologue (UL49) is dispensable for virus growth in vitro and has no effect on virulence and neuronal spread in rodents.Relevance of the interaction between alphaherpesvirus UL3.5 and UL48 proteins for virion maturation and neuroinvasion.A pseudorabies virus recombinant simultaneously lacking the major tegument proteins encoded by the UL46, UL47, UL48, and UL49 genes is viable in cultured cells.The varicella-zoster virus (VZV) ORF9 protein interacts with the IE62 major VZV transactivator.The alpha-TIF (VP16) homologue (ETIF) of equine herpesvirus 1 is essential for secondary envelopment and virus egress.Phosphorylation of the varicella-zoster virus (VZV) major transcriptional regulatory protein IE62 by the VZV open reading frame 66 protein kinase.Complete DNA sequences of two oka strain varicella-zoster virus genomes.
P2860
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P2860
Varicella-zoster virus (VZV) open reading frame 10 protein, the homolog of the essential herpes simplex virus protein VP16, is dispensable for VZV replication in vitro.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Varicella-zoster virus (VZV) o ...... for VZV replication in vitro.
@ast
Varicella-zoster virus (VZV) o ...... for VZV replication in vitro.
@en
type
label
Varicella-zoster virus (VZV) o ...... for VZV replication in vitro.
@ast
Varicella-zoster virus (VZV) o ...... for VZV replication in vitro.
@en
prefLabel
Varicella-zoster virus (VZV) o ...... for VZV replication in vitro.
@ast
Varicella-zoster virus (VZV) o ...... for VZV replication in vitro.
@en
P2860
P1433
P1476
Varicella-zoster virus (VZV) o ...... e for VZV replication in vitro
@en
P2093
P2860
P304
P407
P577
1994-12-01T00:00:00Z