Herpes simplex virus thymidine kinase and specific stages of latency in murine trigeminal ganglia.
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Absence of rapid selection for acyclovir or penciclovir resistance following suboptimal oral prodrug therapy of HSV-infected miceDiverse herpes simplex virus type 1 thymidine kinase mutants in individual human neurons and Ganglia.Human thymidine kinase can functionally replace herpes simplex virus type 1 thymidine kinase for viral replication in mouse sensory ganglia and reactivation from latency upon explant.The latent herpes simplex virus type 1 genome copy number in individual neurons is virus strain specific and correlates with reactivationRecent progress in herpes simplex virus immunobiology and vaccine researchResistance of herpes simplex viruses to nucleoside analogues: mechanisms, prevalence, and managementHerpes simplex virus DNA synthesis is not a decisive regulatory event in the initiation of lytic viral protein expression in neurons in vivo during primary infection or reactivation from latency.Failure of thymidine kinase-negative herpes simplex virus to reactivate from latency following efficient establishment.Phosphorylation of herpes simplex virus 1 dUTPase regulates viral virulence and genome integrity by compensating for low cellular dUTPase activity in the central nervous systemEffects of thyroid hormone on HSV-1 gene regulation: implications in the control of viral latency and reactivation.A net +1 frameshift permits synthesis of thymidine kinase from a drug-resistant herpes simplex virus mutant.Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleosideCorrelation between precolonization of trigeminal ganglia by attenuated strains of pseudorabies virus and resistance to wild-type virus latency.Phosphorylation of a herpes simplex virus 1 dUTPase by a viral protein kinase, Us3, dictates viral pathogenicity in the central nervous system but not at the periphery.Difference in incidence of spontaneous mutations between Herpes simplex virus types 1 and 2.Characterization of herpes simplex viruses selected in culture for resistance to penciclovir or acyclovir.Accumulation of viral transcripts and DNA during establishment of latency by herpes simplex virus.Characterization of herpes simplex virus type 1 thymidine kinase mutants selected under a single round of high-dose brivudinThe herpes simplex virus type 1 latency associated transcript locus is required for the maintenance of reactivation competent latent infections.
P2860
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P2860
Herpes simplex virus thymidine kinase and specific stages of latency in murine trigeminal ganglia.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Herpes simplex virus thymidine ...... in murine trigeminal ganglia.
@ast
Herpes simplex virus thymidine ...... in murine trigeminal ganglia.
@en
type
label
Herpes simplex virus thymidine ...... in murine trigeminal ganglia.
@ast
Herpes simplex virus thymidine ...... in murine trigeminal ganglia.
@en
prefLabel
Herpes simplex virus thymidine ...... in murine trigeminal ganglia.
@ast
Herpes simplex virus thymidine ...... in murine trigeminal ganglia.
@en
P2093
P2860
P1433
P1476
Herpes simplex virus thymidine ...... in murine trigeminal ganglia.
@en
P2093
J G Jacobson
K L Ruffner
M Kosz-Vnenchak
P2860
P304
P407
P577
1993-11-01T00:00:00Z