Regulation of the C/EBP-related gene gadd153 by glucose deprivation. - Wikidata - awgldk - TriplyDB

Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

http://www.wikidata.org/entity/Q36694493

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CHOP enhancement of gene transcription by interactions with Jun/Fos AP-1 complex proteins Endoplasmic reticulum stress-induced apoptosis: multiple pathways and activation of p53-up-regulated modulator of apoptosis (PUMA) and NOXA by p53 Ig/EBP (C/EBP gamma) is a transdominant negative inhibitor of C/EBP family transcriptional activators Analysis of ATF3, a transcription factor induced by physiological stresses and modulated by gadd153/Chop10 Secretory defect and cytotoxicity: the potential disease mechanisms for the retinitis pigmentosa (RP)-associated interphotoreceptor retinoid-binding protein (IRBP)Identification of novel stress-induced genes downstream of chop The gadd and MyD genes define a novel set of mammalian genes encoding acidic proteins that synergistically suppress cell growth Japanese encephalitis virus infection initiates endoplasmic reticulum stress and an unfolded protein response A novel effector domain from the RNA-binding protein TLS or EWS is required for oncogenic transformation by CHOP Cellular iron depletion and the mechanisms involved in the iron-dependent regulation of the growth arrest and DNA damage family of genes CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum Stress-induced binding of the transcriptional factor CHOP to a novel DNA control element Involvement of GADD153 and cardiac ankyrin repeat protein in hypoxia-induced apoptosis of H9c2 cells Gadd153 sensitizes cells to endoplasmic reticulum stress by down-regulating Bcl2 and perturbing the cellular redox state Involvement of GADD153 and cardiac ankyrin repeat protein in cardiac ischemia-reperfusion injury Expression of the pro-apoptotic gene gadd153/chop is elevated in liver with aging and sensitizes cells to oxidant injury Cloning of mammalian Ire1 reveals diversity in the ER stress responses CHOP-Dependent stress-inducible expression of a novel form of carbonic anhydrase VI ATF6 activated by proteolysis binds in the presence of NF-Y (CBF) directly to the cis-acting element responsible for the mammalian unfolded protein response Liver X receptor agonists augment human islet function through activation of anaplerotic pathways and glycerolipid/free fatty acid cycling.Autocrine human growth hormone (hGH) regulation of human mammary carcinoma cell gene expression. Identification of CHOP as a mediator of hGH-stimulated human mammary carcinoma cell survival.GADD153 expression does not necessarily correlate with changes in culture behavior of hybridoma cells.DDIT3/CHOP and the sarcoma fusion oncoprotein FUS-DDIT3/TLS-CHOP bind cyclin-dependent kinase 2.beta-adrenergic receptor-induced activation of nerve growth factor gene transcription in rat cerebral cortex involves CCAAT/enhancer-binding protein delta The C/EBP family of transcription factors in the liver and other organs.Transcriptional regulation of the human growth hormone receptor (hGHR) gene V2 promoter by transcriptional activators and repressor.Protective function of von Hippel-Lindau protein against impaired protein processing in renal carcinoma cells Nitric oxide-induced apoptosis in pancreatic beta cells is mediated by the endoplasmic reticulum stress pathway.Inhibition of CHOP translation by a peptide encoded by an open reading frame localized in the chop 5'UTR A mitochondrial specific stress response in mammalian cells Is there a common upstream link for autophagic and apoptotic cell death in human high-grade gliomas?Upregulation of the coagulation factor VII gene during glucose deprivation is mediated by activating transcription factor 4.CCAAT/Enhancer-binding protein homologous protein (CHOP) decreases bone formation and causes osteopenia.Expression profiling of pancreatic beta cells: glucose regulation of secretory and metabolic pathway genes Differential regulation of CHOP-10/GADD153 gene expression by MAPK signaling in pancreatic beta-cells.Legionella suppresses the host unfolded protein response via multiple mechanisms.Induction of a secreted protein by the myxoid liposarcoma oncogene Evidence for posttranscriptional regulation of C/EBPalpha and C/EBPbeta isoform expression during the lipopolysaccharide-mediated acute-phase response.Signals from the stressed endoplasmic reticulum induce C/EBP-homologous protein (CHOP/GADD153).gadd153/Chop10, a potential target gene of the transcriptional repressor ATF3.

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P2860

Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

http://www.wikidata.org/entity/Q36694493

description

1993 nî lūn-bûn

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1993年の論文

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1993年論文

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1993年論文

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1993年論文

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1993年論文

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1993年論文

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1993年论文

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1993年论文

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1993年论文

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name

Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

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Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

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type

label

Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

@ast

Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

@en

prefLabel

Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

@ast

Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

@en

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Molecular and Cellular Biology

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Regulation of the C/EBP-related gene gadd153 by glucose deprivation.

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Bartlett JD

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Bernier M

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Carlson SG

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Fawcett TW

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Holbrook NJ

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P2860

Isolation, characterization and chromosomal localization of the human GADD153 gene

Genomic sequencing

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Isolation of biologically active ribonucleic acid from sources enriched in ribonuclease

Rearrangement of the transcription factor gene CHOP in myxoid liposarcomas with t(12;16)(q13;p11)

Regulated expression of three C/EBP isoforms during adipose conversion of 3T3-L1 cells

Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogene

Genetic and biochemical definition of the bovine papillomavirus E5 transforming protein.

Induction of fatty acid synthetase synthesis in differentiating 3T3-L1 preadipocytes.

Is CCAAT/enhancer-binding protein a central regulator of energy metabolism?

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4736-4744

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10.1128/MCB.13.8.4736

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360099

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