The Epstein-Barr virus nuclear protein 1 promoter active in type I latency is autoregulated.
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IRF-7, a new interferon regulatory factor associated with Epstein-Barr virus latencyExpression of Epstein-Barr virus nuclear antigen-1 induces B cell neoplasia in transgenic miceThe cellular proteins that bind specifically to the Epstein-Barr virus origin of plasmid DNA replication belong to a gene familyDual stimulation of Epstein-Barr Virus (EBV)-specific CD4+- and CD8+-T-cell responses by a chimeric antigen construct: potential therapeutic vaccine for EBV-positive nasopharyngeal carcinomaA conserved domain of the Epstein-Barr virus nuclear antigens 3A and 3C binds to a discrete domain of JkappaGamma-herpesvirus latency requires T cell evasion during episome maintenancePotential cellular functions of Epstein-Barr Nuclear Antigen 1 (EBNA1) of Epstein-Barr VirusEpstein-Barr virus latency switch in human B-cells: a physico-chemical model.Epstein-Barr virus independent dysregulation of UBP43 expression alters interferon-stimulated gene expression in Burkitt lymphoma.Epstein-Barr virus-encoded EBNA1 inhibits the canonical NF-kappaB pathway in carcinoma cells by inhibiting IKK phosphorylation.Mechanisms that regulate Epstein-Barr virus EBNA-1 gene transcription during restricted latency are conserved among lymphocryptoviruses of Old World primates.Transcription profiling of Epstein-Barr virus nuclear antigen (EBNA)-1 expressing cells suggests targeting of chromatin remodeling complexesThe Epstein-Barr virus major latent promoter Qp is constitutively active, hypomethylated, and methylation sensitiveFunctional analyses of the EBNA1 origin DNA binding protein of Epstein-Barr virus.Interferon regulatory factor 7 is induced by Epstein-Barr virus latent membrane protein 1Repression of Epstein-Barr virus EBNA-1 gene transcription by pRb during restricted latencyProtein-DNA binding and CpG methylation at nucleotide resolution of latency-associated promoters Qp, Cp, and LMP1p of Epstein-Barr virusThe Epstein-Barr virus and its association with human cancers.Human DNA replication initiation factors, ORC and MCM, associate with oriP of Epstein-Barr virus.Interferon regulatory factor 2 represses the Epstein-Barr virus BamHI Q latency promoter in type III latency.Host factors LR1 and Sp1 regulate the Fp promoter of Epstein-Barr virus.Viral latency and its regulation: lessons from the gamma-herpesviruses.Constitutive binding of EBNA1 protein to the Epstein-Barr virus replication origin, oriP, with distortion of DNA structure during latent infection.Identification of spliced gammaherpesvirus 68 LANA and v-cyclin transcripts and analysis of their expression in vivo during latent infectionTranscriptional activation by EBV nuclear antigen 1 is essential for the expression of EBV's transforming genes.trans-Repression of protein expression dependent on the Epstein-Barr virus promoter Wp during latency.The Epstein-Barr virus latency BamHI-Q promoter is positively regulated by STATs and Zta interference with JAK/STAT activation leads to loss of BamHI-Q promoter activityGastric carcinoma: monoclonal epithelial malignant cells expressing Epstein-Barr virus latent infection protein.A subpopulation of normal B cells latently infected with Epstein-Barr virus resembles Burkitt lymphoma cells in expressing EBNA-1 but not EBNA-2 or LMP1.Transcription start sites downstream of the Epstein-Barr virus (EBV) Fp promoter in early-passage Burkitt lymphoma cells define a fourth promoter for expression of the EBV EBNA-1 proteinTranscription of the Epstein-Barr virus nuclear antigen 1 (EBNA1) gene occurs before induction of the BCR2 (Cp) EBNA gene promoter during the initial stages of infection in B cells.The Epstein-Barr virus EBNA-1 promoter Qp requires an initiator-like element.Epstein-Barr virus latent membrane protein 2 associates with and is a substrate for mitogen-activated protein kinase.Interferon-independent and -induced regulation of Epstein-Barr virus EBNA-1 gene transcription in Burkitt lymphoma.Live-cell imaging reveals multiple interactions between Epstein-Barr virus nuclear antigen 1 and cellular chromatin during interphase and mitosisCell cycle association of the retinoblastoma protein Rb and the histone demethylase LSD1 with the Epstein-Barr virus latency promoter Cp.Host-cell-determined methylation of specific Epstein-Barr virus promoters regulates the choice between distinct viral latency programsSeparation of the complex DNA binding domain of EBNA-1 into DNA recognition and dimerization subdomains of novel structure.Reciprocal regulation of the Epstein-Barr virus BamHI-F promoter by EBNA-1 and an E2F transcription factor.The Epstein-Barr virus (EBV) nuclear antigen 1 BamHI F promoter is activated on entry of EBV-transformed B cells into the lytic cycle.
P2860
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P2860
The Epstein-Barr virus nuclear protein 1 promoter active in type I latency is autoregulated.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
The Epstein-Barr virus nuclear ...... pe I latency is autoregulated.
@ast
The Epstein-Barr virus nuclear ...... pe I latency is autoregulated.
@en
type
label
The Epstein-Barr virus nuclear ...... pe I latency is autoregulated.
@ast
The Epstein-Barr virus nuclear ...... pe I latency is autoregulated.
@en
prefLabel
The Epstein-Barr virus nuclear ...... pe I latency is autoregulated.
@ast
The Epstein-Barr virus nuclear ...... pe I latency is autoregulated.
@en
P2093
P2860
P1433
P1476
The Epstein-Barr virus nuclear ...... pe I latency is autoregulated.
@en
P2093
P2860
P304
P407
P577
1992-08-01T00:00:00Z