The involvement of the tyrosine kinase c-Src in the regulation of reactive oxygen species generation mediated by NADPH oxidase-1.
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Novel p47(phox)-related organizers regulate localized NADPH oxidase 1 (Nox1) activityDirect interaction between Tks proteins and the N-terminal proline-rich region (PRR) of NoxA1 mediates Nox1-dependent ROS generationBiochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular systemPathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2Store-operated calcium entry and diabetic complicationsCanonical transient receptor potential 6 (TRPC6), a redox-regulated cation channelTks5-dependent, nox-mediated generation of reactive oxygen species is necessary for invadopodia formationPeroxiredoxin 6 (Prdx6) supports NADPH oxidase1 (Nox1)-based superoxide generation and cell migration.NADPH oxidase 1 controls the persistence of directed cell migration by a Rho-dependent switch of alpha2/alpha3 integrins.Signaling components of redox active endosomes: the redoxosomes.Emerging evidence for the importance of phosphorylation in the regulation of NADPH oxidases.A novel and specific NADPH oxidase-1 (Nox1) small-molecule inhibitor blocks the formation of functional invadopodia in human colon cancer cellsActivation of c-Src: a hub for exogenous pro-oxidant-mediated activation of Toll-like receptor 4 signalingRedox modifier genes and pathways in amyotrophic lateral sclerosis.Src activates HIF-1α not through direct phosphorylation of HIF-1α specific prolyl-4 hydroxylase 2 but through activation of the NADPH oxidase/Rac pathway.Scaffold proteins IRSp53 and spinophilin regulate localized Rac activation by T-lymphocyte invasion and metastasis protein 1 (TIAM1).Interleukin-4 and interleukin-13 increase NADPH oxidase 1-related proliferation of human colon cancer cells.Regulation of cancer invasion by reactive oxygen species and Tks family scaffold proteins.c-Src-mediated phosphorylation of NoxA1 and Tks4 induces the reactive oxygen species (ROS)-dependent formation of functional invadopodia in human colon cancer cells.Macrophage-specific NOX2 contributes to the development of lung emphysema through modulation of SIRT1/MMP-9 pathways.Redox regulation of protein kinases.Global proteomic assessment of the classical protein-tyrosine phosphatome and "Redoxome"Mitochondrial energetics and therapeutics.Regulation of Nox enzymes expression in vascular pathophysiology: Focusing on transcription factors and epigenetic mechanisms.Methods to monitor classical protein-tyrosine phosphatase oxidation.Glucotoxicity induces glucose-6-phosphatase catalytic unit expression by acting on the interaction of HIF-1α with CREB-binding proteinThe role of Src kinase in macrophage-mediated inflammatory responses.Reactive oxygen species in vascular formation and development.NADPH oxidase biology and the regulation of tyrosine kinase receptor signaling and cancer drug cytotoxicityEffects of iodonium-class flavin dehydrogenase inhibitors on growth, reactive oxygen production, cell cycle progression, NADPH oxidase 1 levels, and gene expression in human colon cancer cells and xenografts.Inside-Out Signaling Pathways from Nuclear Reactive Oxygen Species Control Pulmonary Innate Immunity.Prooxidant-induced c-Src/nuclear factor kappa B-coupled signalling in sensory ganglia mediates cutaneous hyperalgesia.Invasive cells in animals and plants: searching for LECA machineries in later eukaryotic lifeCyclic adenosine monophosphate and brain-derived neurotrophic factor decreased oxidative stress and apoptosis in developing hypothalamic neuronal cells: role of microgliaHepatocytes produce TNF-α following hypoxia-reoxygenation and liver ischemia-reperfusion in a NADPH oxidase- and c-Src-dependent manner.Subanesthetic isoflurane relieves zymosan-induced neutrophil inflammatory response by targeting NMDA glutamate receptor and Toll-like receptor 2 signalingModulation of cellular redox state underlies antagonism between oxaliplatin and cetuximab in human colorectal cancer cell linesNox proteins in signal transductionSelective recapitulation of conserved and nonconserved regions of putative NOXA1 protein activation domain confers isoform-specific inhibition of Nox1 oxidase and attenuation of endothelial cell migration.Reactive oxygen species in cancer.
P2860
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P2860
The involvement of the tyrosine kinase c-Src in the regulation of reactive oxygen species generation mediated by NADPH oxidase-1.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
The involvement of the tyrosin ...... n mediated by NADPH oxidase-1.
@ast
The involvement of the tyrosin ...... n mediated by NADPH oxidase-1.
@en
type
label
The involvement of the tyrosin ...... n mediated by NADPH oxidase-1.
@ast
The involvement of the tyrosin ...... n mediated by NADPH oxidase-1.
@en
prefLabel
The involvement of the tyrosin ...... n mediated by NADPH oxidase-1.
@ast
The involvement of the tyrosin ...... n mediated by NADPH oxidase-1.
@en
P2093
P2860
P356
P1476
The involvement of the tyrosin ...... n mediated by NADPH oxidase-1.
@en
P2093
Davide Gianni
Gary M Bokoch
Sara A Courtneidge
P2860
P304
P356
10.1091/MBC.E08-02-0138
P577
2008-05-07T00:00:00Z