Cell autonomous expression of inflammatory genes in biologically aged fibroblasts associated with elevated NF-kappaB activity.
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Rule-based cell systems model of aging using feedback loop motifs mediated by stress responsesBetter Living through Chemistry: Caloric Restriction (CR) and CR Mimetics Alter Genome Function to Promote Increased Health and LifespanComparative Meta-Analysis of Transcriptomics Data during Cellular Senescence and In Vivo Tissue Ageing.Functional comparison between genes dysregulated in ulcerative colitis and colorectal carcinoma.Comparing the yeast retrograde response and NF-κB stress responses: implications for aging.Alive and well? Exploring disease by studying lifespan.Loss of Nfkb1 leads to early onset agingEpithelial cell senescence impairs repair process and exacerbates inflammation after airway injury.Embryonic expression of the common progeroid lamin A splice mutation arrests postnatal skin developmentSignaling pathway activation drift during aging: Hutchinson-Gilford Progeria Syndrome fibroblasts are comparable to normal middle-age and old-age cellsOverexpression of Lamin B Receptor Results in Impaired Skin Differentiation.NF-κB in Aging and DiseaseInfluence of non-steroidal anti-inflammatory drugs on Drosophila melanogaster longevityTranscriptome analysis of human ageing in male skin shows mid-life period of variability and central role of NF-κB.Alterations in transcriptional responses associated with vascular aging.Computational systems biology of aging.The role of the ubiquitin-editing enzyme A20 in diseases of the central nervous system and other pathological processes.Deciphering the role of nuclear factor-κB in cellular senescence.Activation of nuclear factor-kappa B signalling promotes cellular senescenceDistinct Cell Stress Responses Induced by ATP Restriction in Quiescent Human Fibroblasts.NF-κB inhibition delays DNA damage-induced senescence and aging in mice.Chronic NF-kappaB activation delays RasV12-induced premature senescence of human fibroblasts by suppressing the DNA damage checkpoint response.Poly(ADP-ribose)polymerase 1 inhibition protects against age-dependent endothelial dysfunction.Global mapping of transcription factor motifs in human aging.Embryonic Stem Cell-Derived Mesenchymal Stem Cells (MSCs) Have a Superior Neuroprotective Capacity Over Fetal MSCs in the Hypoxic-Ischemic Mouse Brain.
P2860
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P1343
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P2860
Cell autonomous expression of inflammatory genes in biologically aged fibroblasts associated with elevated NF-kappaB activity.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Cell autonomous expression of ...... h elevated NF-kappaB activity.
@ast
Cell autonomous expression of ...... h elevated NF-kappaB activity.
@en
type
label
Cell autonomous expression of ...... h elevated NF-kappaB activity.
@ast
Cell autonomous expression of ...... h elevated NF-kappaB activity.
@en
prefLabel
Cell autonomous expression of ...... h elevated NF-kappaB activity.
@ast
Cell autonomous expression of ...... h elevated NF-kappaB activity.
@en
P2093
P2860
P4510
P921
P356
P1433
P1476
Cell autonomous expression of ...... h elevated NF-kappaB activity.
@en
P2093
Andres Kriete
Csaba Kari
Kelli L Mayo
Nirupama Yalamanchili
Patrick Bender
Ulrich Rodeck
William Beggs
P2860
P2888
P356
10.1186/1742-4933-5-5
P577
2008-07-16T00:00:00Z
P5875
P6179
1027612736