The E6/E7 promoter of human papillomavirus type 16 is activated in the absence of E2 proteins by a sequence-aberrant Sp1 distal element.
about
Cloning and functional analysis of spliced isoforms of human nuclear factor I-X: interference with transcriptional activation by NFI/CTF in a cell-type specific mannerGenomic diversity and interspecies host infection of alpha12 Macaca fascicularis papillomaviruses (MfPVs).Variant upstream regulatory region sequences differentially regulate human papillomavirus type 16 DNA replication throughout the viral life cycle.Overexpression of C/EBPbeta represses human papillomavirus type 18 upstream regulatory region activity in HeLa cells by interfering with the binding of TATA-binding protein.Temporal usage of multiple promoters during the life cycle of human papillomavirus type 31b.Isolation of three novel rat and mouse papillomaviruses and their genomic characterizationCharacterization of transcription factor binding to human papillomavirus type 16 DNA during cellular differentiation.Control of HPV 18 DNA replication by cellular and viral transcription factors.During negative regulation of the human papillomavirus-16 E6 promoter, the viral E2 protein can displace Sp1 from a proximal promoter element.Transcriptional activation of the human papillomavirus type 5 and 16 long control region in cells from cutaneous and mucosal originDelineation of human papillomavirus type 18 enhancer binding proteins: the intracellular distribution of a novel octamer binding protein p92 is cell cycle regulated.Human papillomavirus (HPV) types 101 and 103 isolated from cervicovaginal cells lack an E6 open reading frame (ORF) and are related to gamma-papillomaviruses.Negative regulation of the bovine papillomavirus E5, E6, and E7 oncogenes by the viral E1 and E2 genes.Suppression of cellular proliferation by the papillomavirus E2 protein.Replication and assembly of human papillomaviruses.YY1 represses human papillomavirus type 16 transcription by quenching AP-1 activity.A novel C/EBP beta-YY1 complex controls the cell-type-specific activity of the human papillomavirus type 18 upstream regulatory region.Transcription activities of human papillomavirus type 11 E6 promoter-proximal elements in raft and submerged cultures of foreskin keratinocytes.Activation of human papillomavirus type 18 E6-E7 oncogene expression by transcription factor Sp1.Differential suppression of the tumorigenicity of HeLa and SiHa cells by adeno-associated virus.IGF1 mRNA isoform expression in the cervix of HPV-positive women with pre-cancerous and cancer lesionsRecent advances in the search for antiviral agents against human papillomaviruses.Human papillomavirus type 11 E2 proteins repress the homologous E6 promoter by interfering with the binding of host transcription factors to adjacent elements.The human papillomavirus type 16 E2 transcription factor binds with low cooperativity to two flanking sites and represses the E6 promoter through displacement of Sp1 and TFIID.The E2 transcriptional repressor can compensate for Sp1 activation of the human papillomavirus type 18 early promoter.Identification and characterization of novel promoters in the genome of human papillomavirus type 18.Cell-type-specific activity of the human papillomavirus type 18 upstream regulatory region in transgenic mice and its modulation by tetradecanoyl phorbol acetate and glucocorticoids.Two AP1 sites binding JunB are essential for human papillomavirus type 18 transcription in keratinocytesThe enhancer of human papillomavirus type 16: binding sites for the ubiquitous transcription factors oct-1, NFA, TEF-2, NF1, and AP-1 participate in epithelial cell-specific transcription.Disruption of either the E1 or the E2 regulatory gene of human papillomavirus type 16 increases viral immortalization capacityThe E6/E7 promoter of extrachromosomal HPV16 DNA in cervical cancers escapes from cellular repression by mutation of target sequences for YY1.The integration of HPV-18 DNA in cervical carcinoma.Identification and characterization of two novel human papillomaviruses (HPVs) by overlapping PCR: HPV102 and HPV106Orientation of a novel DNA binding site affects human papillomavirus-mediated transcription and replication.A novel YY1-independent silencer represses the activity of the human papillomavirus type 16 enhancer.Both viral E2 protein and the cellular factor PEBP2 regulate transcription via E2 consensus sites within the bovine papillomavirus type 4 long control region.Different mechanisms contribute to the E2-mediated transcriptional repression of human papillomavirus type 18 viral oncogenes.Repression of the human papillomavirus type 18 enhancer by the cellular transcription factor Oct-1.Kinetic and equilibrium binding studies of the human papillomavirus type-16 transcription regulatory protein E2 interacting with core enhancer elements.Control of human papillomavirus type 11 origin of replication by the E2 family of transcription regulatory proteins.
P2860
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P2860
The E6/E7 promoter of human papillomavirus type 16 is activated in the absence of E2 proteins by a sequence-aberrant Sp1 distal element.
description
1990 nî lūn-bûn
@nan
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
1990年论文
@zh
1990年论文
@zh-cn
name
The E6/E7 promoter of human pa ...... e-aberrant Sp1 distal element.
@ast
The E6/E7 promoter of human pa ...... e-aberrant Sp1 distal element.
@en
type
label
The E6/E7 promoter of human pa ...... e-aberrant Sp1 distal element.
@ast
The E6/E7 promoter of human pa ...... e-aberrant Sp1 distal element.
@en
prefLabel
The E6/E7 promoter of human pa ...... e-aberrant Sp1 distal element.
@ast
The E6/E7 promoter of human pa ...... e-aberrant Sp1 distal element.
@en
P2860
P1433
P1476
The E6/E7 promoter of human pa ...... e-aberrant Sp1 distal element.
@en
P2093
P2860
P304
P407
P577
1990-11-01T00:00:00Z