IFN-γ receptor and STAT1 signaling in B cells are central to spontaneous germinal center formation and autoimmunity. - Wikidata - awgldk - TriplyDB

IFN-γ receptor and STAT1 signaling in B cells are central to spontaneous germinal center formation and autoimmunity.

IFN-γ receptor and STAT1 signaling in B cells are central to spontaneous germinal center formation and autoimmunity.

http://www.wikidata.org/entity/Q36863144

about

Follicular Helper T (Tfh) Cells in Autoimmune Diseases and Allograft Rejection Ectopic lymphoid neogenesis in rheumatic autoimmune diseases.Pathways leading to an immunological disease: systemic lupus erythematosus.Altered B cell signalling in autoimmunity.Developing connections amongst B lymphocytes and deregulated pathways in autoimmunity.IFN type I and II induce BAFF secretion from human decidual stromal cells.BRILIA: Integrated Tool for High-Throughput Annotation and Lineage Tree Assembly of B-Cell Repertoires.B cells expressing the transcription factor T-bet drive lupus-like autoimmunity.Small Molecule Inhibition of Rab7 Impairs B Cell Class Switching and Plasma Cell Survival To Dampen the Autoantibody Response in Murine Lupus.Cutting Edge: IL-10 Is Essential for the Generation of Germinal Center B Cell Responses and Anti-Plasmodium Humoral Immunity.B cell contribution of the CD4+ T cell inflammatory phenotypes in systemic lupus erythematosus.Mold and Human Health: a Reality Check.Role of germinal centers for the induction of broadly-reactive memory B cells.B-cell tolerance and autoimmunity.Spontaneous germinal centers and autoimmunity.Plasmodium DNA-mediated TLR9 activation of T-bet+ B cells contributes to autoimmune anaemia during malaria.The interplay of type I and type II interferons in murine autoimmune cholangitis as a basis for sex-biased autoimmunity.Mer Receptor Tyrosine Kinase Signaling Prevents Self-Ligand Sensing and Aberrant Selection in Germinal Centers.B cell-derived IL-6 initiates spontaneous germinal center formation during systemic autoimmunity.Distinct Expression and Function of FcεRII in Human B Cells and Monocytes.Bruton's Tyrosine Kinase, a Component of B Cell Signaling Pathways, Has Multiple Roles in the Pathogenesis of Lupus.In Vitro-Induced Germinal Center B Cell Culture System.Relative Contributions of B Cells and Dendritic Cells from Lupus-Prone Mice to CD4+ T Cell Polarization.Tailoring Immune Responses toward Autoimmunity: Transcriptional Regulators That Drive the Creation and Collusion of Autoreactive Lymphocytes.Galectin-3 deficiency drives lupus-like disease by promoting spontaneous germinal centers formation via IFN-γ.Inhibition of glucose metabolism selectively targets autoreactive follicular helper T cells A splenic IgM memory subset with antibacterial specificities is sustained from persistent mucosal responses Expansion and Sub-Classification of T Cell-Dependent Antibody Responses to Encompass the Role of Innate-Like T Cells in Antibody Responses Innate and adaptive signals enhance differentiation and expansion of dual-antibody autoreactive B cells in lupus

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P2860

IFN-γ receptor and STAT1 signaling in B cells are central to spontaneous germinal center formation and autoimmunity.

http://www.wikidata.org/entity/Q36863144

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

IFN-γ receptor and STAT1 signa ...... er formation and autoimmunity.

@en

type

label

IFN-γ receptor and STAT1 signa ...... er formation and autoimmunity.

@en

prefLabel

IFN-γ receptor and STAT1 signa ...... er formation and autoimmunity.

@en

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P1433

Journal of Experimental Medicine

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IFN-γ receptor and STAT1 signa ...... er formation and autoimmunity.

@en

P2093

Chetna Soni

http://www.w3.org/2001/XMLSchema#string

Daisuke Kitamura

http://www.w3.org/2001/XMLSchema#string

Eric B Wong

http://www.w3.org/2001/XMLSchema#string

Melinda J Elias

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Sathi Babu Chodisetti

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Stephanie L Schell

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Timothy K Cooper

http://www.w3.org/2001/XMLSchema#string

Ziaur S M Rahman

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P2860

Age-Associated B Cells: A T-bet-Dependent Effector with Roles in Protective and Pathogenic Immunity

B cells take the front seat: dysregulated B cell signals orchestrate loss of tolerance and autoantibody production

Normal induction but attenuated progression of germinal center responses in BAFF and BAFF-R signaling-deficient mice

T-bet regulates IgG class switching and pathogenic autoantibody production

Chromatin-IgG complexes activate B cells by dual engagement of IgM and Toll-like receptors

The lupus-prone NZM2410/NZW strain-derived Sle1b sublocus alters the germinal center checkpoint in female mice in a B cell-intrinsic manner.

B cell-intrinsic signaling through IL-21 receptor and STAT3 is required for establishing long-lived antibody responses in humans.

IL-21 regulates germinal center B cell differentiation and proliferation through a B cell-intrinsic mechanism.

IL-21 acts directly on B cells to regulate Bcl-6 expression and germinal center responses.

Rationale for B cell targeting in SLE

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715-732

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scholarly article

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10.1084/JEM.20151722

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P433

5

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213

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Phillip P. Domeier

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2016-04-11T00:00:00Z

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27069112

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4854731

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