mda-9/Syntenin promotes metastasis in human melanoma cells by activating c-Src.
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Extensions of PSD-95/discs large/ZO-1 (PDZ) domains influence lipid binding and membrane targeting of syntenin-1Syntenin regulates TGF-β1-induced Smad activation and the epithelial-to-mesenchymal transition by inhibiting caveolin-mediated TGF-β type I receptor internalizationIdentifying mRNA, microRNA and protein profiles of melanoma exosomesSyntenin controls migration, growth, proliferation, and cell cycle progression in cancer cells.Binding to syntenin-1 protein defines a new mode of ubiquitin-based interactions regulated by phosphorylation.The PDZ-adaptor protein syntenin-1 regulates HIV-1 entryGene expression analysis after receptor tyrosine kinase activation reveals new potential melanoma proteins.Elevated expression of syntenin in breast cancer is correlated with lymph node metastasis and poor patient survivalSrc kinase activation is mandatory for MDA-9/syntenin-mediated activation of nuclear factor-kappaB.Syntenin promotes VEGF-induced VEGFR2 endocytosis and angiogenesis by increasing ephrin-B2 function in endothelial cells.Mda-9/syntenin is expressed in uveal melanoma and correlates with metastatic progressionSyndecan binding protein (SDCBP) is overexpressed in estrogen receptor negative breast cancers, and is a potential promoter for tumor proliferation.MDA-9/syntenin is essential for factor VIIa-induced signaling, migration, and metastasis in melanoma cellsmda-9/Syntenin protein positively regulates the activation of Akt protein by facilitating integrin-linked kinase adaptor function during adhesion to type I collagen.Targeting tumor invasion: the roles of MDA-9/Syntenin.Dasatinib inhibits c-src phosphorylation and prevents the proliferation of Triple-Negative Breast Cancer (TNBC) cells which overexpress Syndecan-Binding Protein (SDCBP).Syntenin is expressed in human gliomas and may correlate with tumor migration.Src, p130Cas, and Mechanotransduction in Cancer CellsAnalysis of SRC oncogenic signaling in colorectal cancer by stable isotope labeling with heavy amino acids in mouse xenograftsMDA-9/syntenin and IGFBP-2 promote angiogenesis in human melanomaExpression patterns of MDA-9/syntenin during development of the mouse embryo.D-pinitol inhibits prostate cancer metastasis through inhibition of αVβ3 integrin by modulating FAK, c-Src and NF-κB pathways.Berberine Reduces the Metastasis of Chondrosarcoma by Modulating the α v β 3 Integrin and the PKC δ , c-Src, and AP-1 Signaling Pathways.Examination of Epigenetic and other Molecular Factors Associated with mda-9/Syntenin Dysregulation in Cancer Through Integrated Analyses of Public Genomic DatasetsSyntenin is expressed in human follicular dendritic cells and involved in the activation of focal adhesion kinase.MDA-9/syntenin is a key regulator of glioma pathogenesis.Novel role of MDA-9/syntenin in regulating urothelial cell proliferation by modulating EGFR signaling.Raf kinase inhibitor RKIP inhibits MDA-9/syntenin-mediated metastasis in melanomaSyntenin increases the invasiveness of small cell lung cancer cells by activating p38, AKT, focal adhesion kinase and SP1.Novel function of MDA-9/Syntenin (SDCBP) as a regulator of survival and stemness in glioma stem cells.MDA-9/Syntenin (SDCBP) modulates small GTPases RhoA and Cdc42 via transforming growth factor β1 to enhance epithelial-mesenchymal transition in breast cancer.The role of mitogen- and stress-activated protein kinase pathways in melanoma.CSPG4, a potential therapeutic target, facilitates malignant progression of melanoma.Non-BRAF targeted therapies for melanoma: protein kinase inhibitors in Phase II clinical trials.The RNA-binding protein Musashi1 affects medulloblastoma growth via a network of cancer-related genes and is an indicator of poor prognosis.MDA-9/Syntenin Control.Unravelling the Mechanism of TrkA-Induced Cell Death by Macropinocytosis in Medulloblastoma Daoy CellsInhibition of Transforming Growth Factor-β (TGF-β) Signaling by Scutellaria baicalensis and Fritillaria cirrhosa Extracts in Endometrial Cancer.Targeting invasive properties of melanoma cells.Syntenin-mediated regulation of Sox4 proteasomal degradation modulates transcriptional output.
P2860
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P2860
mda-9/Syntenin promotes metastasis in human melanoma cells by activating c-Src.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
mda-9/Syntenin promotes metastasis in human melanoma cells by activating c-Src.
@en
type
label
mda-9/Syntenin promotes metastasis in human melanoma cells by activating c-Src.
@en
prefLabel
mda-9/Syntenin promotes metastasis in human melanoma cells by activating c-Src.
@en
P2093
P2860
P356
P1476
mda-9/Syntenin promotes metastasis in human melanoma cells by activating c-Src.
@en
P2093
Célia Prévot
Devanand Sarkar
Habib Boukerche
Paul B Fisher
Zao-zhong Su
P2860
P304
15914-15919
P356
10.1073/PNAS.0808171105
P407
P577
2008-10-02T00:00:00Z