Hexosamine biosynthesis and protein O-glycosylation: the first line of defense against stress, ischemia, and trauma.
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O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter?Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signalingCross-talk between two essential nutrient-sensitive enzymes: O-GlcNAc transferase (OGT) and AMP-activated protein kinase (AMPK)The hexosamine signaling pathway: O-GlcNAc cycling in feast or famineInfluence of glucosamine on glomerular mesangial cell turnover: implications for hyperglycemia and hexosamine pathway fluxGlycopeptide-specific monoclonal antibodies suggest new roles for O-GlcNAc.Dynamic O-GlcNAc cycling at promoters of Caenorhabditis elegans genes regulating longevity, stress, and immunity.O-GlcNAc signaling in the cardiovascular system.O-GlcNAcylation: a novel post-translational mechanism to alter vascular cellular signaling in health and disease: focus on hypertension.O-GlcNAcylation: a novel pathway contributing to the effects of endothelin in the vasculature.Physiological effects of oral glucosamine on joint health: current status and consensus on future research prioritiesIncreased O-linked beta-N-acetylglucosamine levels on proteins improves survival, reduces inflammation and organ damage 24 hours after trauma-hemorrhage in ratsHigh glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylation.Metabolic effects of glutamine on the heart: anaplerosis versus the hexosamine biosynthetic pathwayThe dynamic stress-induced "O-GlcNAc-ome" highlights functions for O-GlcNAc in regulating DNA damage/repair and other cellular pathways.NOS1AP O-GlcNAc Modification Involved in Neuron Apoptosis Induced by ExcitotoxicityThe roles of O-linked β-N-acetylglucosamine in cardiovascular physiology and disease.Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.Dynamic O-GlcNAcylation and its roles in the cellular stress response and homeostasis.Unique hexosaminidase reduces metabolic survival signal and sensitizes cardiac myocytes to hypoxia/reoxygenation injury.Endothelial inflammation induced by excess glucose is associated with cytosolic glucose 6-phosphate but not increased mitochondrial respiration.O-GlcNAc signaling attenuates ER stress-induced cardiomyocyte death.The role of hyperglycemia in burned patients: evidence-based studies.O-GlcNAc and the cardiovascular system.Modulation of transcription factor function by O-GlcNAc modification.The increase in O-linked N-acetylglucosamine protein modification stimulates chondrogenic differentiation both in vitro and in vivo.O-linked beta-N-acetylglucosamine (O-GlcNAc) regulates stress-induced heat shock protein expression in a GSK-3beta-dependent manner.O-GlcNAcylation Reduces Ischemia-Reperfusion-Induced Brain Injury.Detection of O-Linked-N-Acetylglucosamine Modification and Its Associated Enzymes in Human Degenerated Intervertebral Discs.The Role of Stress-Induced O-GlcNAc Protein Modification in the Regulation of Membrane Transport.'-GlcNAc Code' Mediated Biological Functions of Downstream Proteins
P2860
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P2860
Hexosamine biosynthesis and protein O-glycosylation: the first line of defense against stress, ischemia, and trauma.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Hexosamine biosynthesis and pr ...... stress, ischemia, and trauma.
@en
type
label
Hexosamine biosynthesis and pr ...... stress, ischemia, and trauma.
@en
prefLabel
Hexosamine biosynthesis and pr ...... stress, ischemia, and trauma.
@en
P2093
P1433
P1476
Hexosamine biosynthesis and pr ...... t stress, ischemia, and trauma
@en
P2093
Laszlo G Nöt
Norbert Fülöp
Richard B Marchase
P304
P356
10.1097/SHK.0B013E3181598BAD
P50
P577
2008-04-01T00:00:00Z