Minimal change nephropathy and focal segmental glomerulosclerosis.
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Occurrence of minimal change nephrotic syndrome in classical Hodgkin lymphoma is closely related to the induction of c-mip in Hodgkin-Reed Sternberg cells and podocytesCell biology and genetics of minimal change diseaseIQGAP1 mediates angiotensin II-induced apoptosis of podocytes via the ERK1/2 MAPK signaling pathwayc-mip impairs podocyte proximal signaling and induces heavy proteinuriaUpregulation of nuclear factor-related kappa B suggests a disorder of transcriptional regulation in minimal change nephrotic syndrome.Induction of podocyte-derived VEGF ameliorates podocyte injury and subsequent abnormal glomerular development caused by puromycin aminonucleosideIQGAP1 regulates actin cytoskeleton organization in podocytes through interaction with nephrinMycophenolate mofetil (MMF) treatment efficacy in children with primary and secondary glomerulonephritis.Minimal change nephrotic syndrome associated with non-Hodgkin lymphoid disorders: a retrospective study of 18 cases.Inducible rodent models of acquired podocyte diseases.Actin-associated Proteins in the Pathogenesis of Podocyte InjuryMinimal mesangial lupus nephritis: a systematic review.Immunopathogenesis of idiopathic nephrotic syndrome with relapse.The struggle for energy in podocytes leads to nephrotic syndrome.Our experience with rituximab therapy for adult-onset primary glomerulonephritis and review of literature.Active proteases in nephrotic plasma lead to a podocin-dependent phosphorylation of VASP in podocytes via protease activated receptor-1.Role of CD2-associated protein in albumin overload-induced apoptosis in podocytes.The level of urinary semaphorin3A is associated with disease activity in patients with minimal change nephrotic syndrome.LPS and PAN-induced podocyte injury in an in vitro model of minimal change disease: changes in TLR profile.mTORC1 activation triggers the unfolded protein response in podocytes and leads to nephrotic syndrome.Taurine Supplementation Alleviates Puromycin Aminonucleoside Damage by Modulating Endoplasmic Reticulum Stress and Mitochondrial-Related Apoptosis in Rat Kidney.
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P2860
Minimal change nephropathy and focal segmental glomerulosclerosis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 23 October 2007
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Minimal change nephropathy and focal segmental glomerulosclerosis.
@en
type
label
Minimal change nephropathy and focal segmental glomerulosclerosis.
@en
prefLabel
Minimal change nephropathy and focal segmental glomerulosclerosis.
@en
P2860
P1476
Minimal change nephropathy and focal segmental glomerulosclerosis.
@en
P2093
Peter W Mathieson
P2860
P2888
P304
P356
10.1007/S00281-007-0094-Z
P577
2007-10-23T00:00:00Z