Increased cortical expression of the zinc transporter SLC39A12 suggests a breakdown in zinc cellular homeostasis as part of the pathophysiology of schizophrenia.
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The Emerging Role for Zinc in Depression and Psychosis.Changed gene expression in subjects with schizophrenia and low cortical muscarinic M1 receptors predicts disrupted upstream pathways interacting with that receptor.Zinc Signal in Brain Diseases.Recent Advances in the Role of SLC39A/ZIP Zinc Transporters In Vivo.Changed frontal pole gene expression suggest altered interplay between neurotransmitter, developmental, and inflammatory pathways in schizophrenia.A genetic variant in SLC30A2 causes breast dysfunction during lactation by inducing ER stress, oxidative stress and epithelial barrier defects.Brain Cell Type Specific Gene Expression and Co-expression Network Architectures.
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P2860
Increased cortical expression of the zinc transporter SLC39A12 suggests a breakdown in zinc cellular homeostasis as part of the pathophysiology of schizophrenia.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Increased cortical expression ...... hophysiology of schizophrenia.
@en
type
label
Increased cortical expression ...... hophysiology of schizophrenia.
@en
prefLabel
Increased cortical expression ...... hophysiology of schizophrenia.
@en
P2093
P2860
P50
P356
P1433
P1476
Increased cortical expression ...... hophysiology of schizophrenia.
@en
P2093
Aradhana Upadhyay
Elizabeth A Thomas
Elizabeth Scarr
Ian P Everall
Jaclyn Neo
Mark A Greenough
Myoung Suk Seo
P2860
P2888
P356
10.1038/NPJSCHZ.2016.2
P577
2016-03-09T00:00:00Z
P5875
P6179
1028056309