Calcium/calmodulin-dependent protein kinase (CaMK) IV mediates nucleocytoplasmic shuttling and release of HMGB1 during lipopolysaccharide stimulation of macrophages
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NK Cells, Tumor Cell Transition, and Tumor Progression in Solid Malignancies: New Hints for NK-Based Immunotherapy?Extracellular high-mobility group box 1 protein (HMGB1) as a mediator of persistent painHMG modifications and nuclear function.Molecular mechanism and therapeutic modulation of high mobility group box 1 release and action: an updated review.Novel HMGB1-inhibiting therapeutic agents for experimental sepsis.High mobility group box 1 protein as a potential drug target for infection- and injury-elicited inflammation.Fas (CD95) induces rapid, TLR4/IRAK4-dependent release of pro-inflammatory HMGB1 from macrophagesCK2 phosphorylation of Schistosoma mansoni HMGB1 protein regulates its cellular traffic and secretion but not its DNA transactionsBoth bone marrow-derived and non-bone marrow-derived cells contribute to AIM2 and NLRP3 inflammasome activation in a MyD88-dependent manner in dietary steatohepatitis.The dengue vector Aedes aegypti contains a functional high mobility group box 1 (HMGB1) protein with a unique regulatory C-terminus.Dengue virus infection mediates HMGB1 release from monocytes involving PCAF acetylase complex and induces vascular leakage in endothelial cells.High mobility group box 1 release from hepatocytes during ischemia and reperfusion injury is mediated by decreased histone deacetylase activity.Transcription control pathways decode patterned synaptic inputs into diverse mRNA expression profilesCaMKIV-dependent preservation of mTOR expression is required for autophagy during lipopolysaccharide-induced inflammation and acute kidney injury.Suppression of autoimmunity and organ pathology in lupus-prone mice upon inhibition of calcium/calmodulin-dependent protein kinase type IV.HMGB1 in health and disease.The heme oxygenase-1 inducer THI-56 negatively regulates iNOS expression and HMGB1 release in LPS-activated RAW 264.7 cells and CLP-induced septic mice.Calcium/calmodulin-dependent protein kinase (CaMK) Ialpha mediates the macrophage inflammatory response to sepsisRelease of neuronal HMGB1 by ethanol through decreased HDAC activity activates brain neuroimmune signaling.Apoptotic response through a high mobility box 1 protein-dependent mechanism in LPS/GalN-induced mouse liver failure and glycyrrhizin-mediated inhibition.Ethyl pyruvate inhibits HMGB1 phosphorylation and release by chelating calciumProtective effect of olmesartan against cardiac ischemia/reperfusion injury in spontaneously hypertensive ratsInteraction between uric acid and HMGB1 translocation and release from endothelial cells.Ca(2+)/Calmodulin-Dependent Protein Kinase IV Promotes Interplay of Proteins in Chromatoid Body of Male Germ Cells.Inhibition of CaMKIV relieves streptozotocin-induced diabetic neuropathic pain through regulation of HMGB1HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-OverloadToll-like receptor 4 engagement inhibits adenosine 5'-monophosphate-activated protein kinase activation through a high mobility group box 1 protein-dependent mechanismCalcium/calmodulin-dependent protein kinase IV limits organ damage in hepatic ischemia-reperfusion injury through induction of autophagy.Deacetylation-mediated interaction of SIRT1-HMGB1 improves survival in a mouse model of endotoxemia.Calcium/Calmodulin-Dependent Protein Kinase is Involved in the Release of High Mobility Group Box 1 Via the Interferon-β Signaling PathwayHMGB1 in renal ischemic injury.Inhibition of CaMKK2 reverses age-associated decline in bone massCaMKIα regulates AMP kinase-dependent, TORC-1-independent autophagy during lipopolysaccharide-induced acute lung neutrophilic inflammation.Biology and Metabolism of Sepsis: Innate Immunity, Bioenergetics, and AutophagyCalcium supplementation during sepsis exacerbates organ failure and mortality via calcium/calmodulin-dependent protein kinase kinase signalingJAK/STAT1 signaling promotes HMGB1 hyperacetylation and nuclear translocation.How macrophages ring the inflammation alarmHMGB1 release by H2O2-induced hepatocytes is regulated through calcium overload and 58-F interference.High-mobility group box 1, oxidative stress, and disease.Role of high-mobility group box 1 protein in inflammatory bowel disease.
P2860
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P2860
Calcium/calmodulin-dependent protein kinase (CaMK) IV mediates nucleocytoplasmic shuttling and release of HMGB1 during lipopolysaccharide stimulation of macrophages
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Calcium/calmodulin-dependent p ...... ide stimulation of macrophages
@en
type
label
Calcium/calmodulin-dependent p ...... ide stimulation of macrophages
@en
prefLabel
Calcium/calmodulin-dependent p ...... ide stimulation of macrophages
@en
P2093
P2860
P1476
Calcium/calmodulin-dependent p ...... ide stimulation of macrophages
@en
P2093
Anthony R Means
David Wheeler
Lanping Guo
Matthew R Rosengart
Richard A Shapiro
Thomas J Ribar
Timothy R Billiar
Xianghong Zhang
P2860
P304
P356
10.4049/JIMMUNOL.181.7.5015
P407
P577
2008-10-01T00:00:00Z