Calcium/calmodulin-dependent protein kinase (CaMK) IV mediates nucleocytoplasmic shuttling and release of HMGB1 during lipopolysaccharide stimulation of macrophages - Wikidata - awgldk - TriplyDB

Calcium/calmodulin-dependent protein kinase (CaMK) IV mediates nucleocytoplasmic shuttling and release of HMGB1 during lipopolysaccharide stimulation of macrophages

Calcium/calmodulin-dependent protein kinase (CaMK) IV mediates nucleocytoplasmic shuttling and release of HMGB1 during lipopolysaccharide stimulation of macrophages

http://www.wikidata.org/entity/Q36984742

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NK Cells, Tumor Cell Transition, and Tumor Progression in Solid Malignancies: New Hints for NK-Based Immunotherapy?Extracellular high-mobility group box 1 protein (HMGB1) as a mediator of persistent pain HMG modifications and nuclear function.Molecular mechanism and therapeutic modulation of high mobility group box 1 release and action: an updated review.Novel HMGB1-inhibiting therapeutic agents for experimental sepsis.High mobility group box 1 protein as a potential drug target for infection- and injury-elicited inflammation.Fas (CD95) induces rapid, TLR4/IRAK4-dependent release of pro-inflammatory HMGB1 from macrophages CK2 phosphorylation of Schistosoma mansoni HMGB1 protein regulates its cellular traffic and secretion but not its DNA transactions Both bone marrow-derived and non-bone marrow-derived cells contribute to AIM2 and NLRP3 inflammasome activation in a MyD88-dependent manner in dietary steatohepatitis.The dengue vector Aedes aegypti contains a functional high mobility group box 1 (HMGB1) protein with a unique regulatory C-terminus.Dengue virus infection mediates HMGB1 release from monocytes involving PCAF acetylase complex and induces vascular leakage in endothelial cells.High mobility group box 1 release from hepatocytes during ischemia and reperfusion injury is mediated by decreased histone deacetylase activity.Transcription control pathways decode patterned synaptic inputs into diverse mRNA expression profiles CaMKIV-dependent preservation of mTOR expression is required for autophagy during lipopolysaccharide-induced inflammation and acute kidney injury.Suppression of autoimmunity and organ pathology in lupus-prone mice upon inhibition of calcium/calmodulin-dependent protein kinase type IV.HMGB1 in health and disease.The heme oxygenase-1 inducer THI-56 negatively regulates iNOS expression and HMGB1 release in LPS-activated RAW 264.7 cells and CLP-induced septic mice.Calcium/calmodulin-dependent protein kinase (CaMK) Ialpha mediates the macrophage inflammatory response to sepsis Release of neuronal HMGB1 by ethanol through decreased HDAC activity activates brain neuroimmune signaling.Apoptotic response through a high mobility box 1 protein-dependent mechanism in LPS/GalN-induced mouse liver failure and glycyrrhizin-mediated inhibition.Ethyl pyruvate inhibits HMGB1 phosphorylation and release by chelating calcium Protective effect of olmesartan against cardiac ischemia/reperfusion injury in spontaneously hypertensive rats Interaction between uric acid and HMGB1 translocation and release from endothelial cells.Ca(2+)/Calmodulin-Dependent Protein Kinase IV Promotes Interplay of Proteins in Chromatoid Body of Male Germ Cells.Inhibition of CaMKIV relieves streptozotocin-induced diabetic neuropathic pain through regulation of HMGB1 HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload Toll-like receptor 4 engagement inhibits adenosine 5'-monophosphate-activated protein kinase activation through a high mobility group box 1 protein-dependent mechanism Calcium/calmodulin-dependent protein kinase IV limits organ damage in hepatic ischemia-reperfusion injury through induction of autophagy.Deacetylation-mediated interaction of SIRT1-HMGB1 improves survival in a mouse model of endotoxemia.Calcium/Calmodulin-Dependent Protein Kinase is Involved in the Release of High Mobility Group Box 1 Via the Interferon-β Signaling Pathway HMGB1 in renal ischemic injury.Inhibition of CaMKK2 reverses age-associated decline in bone mass CaMKIα regulates AMP kinase-dependent, TORC-1-independent autophagy during lipopolysaccharide-induced acute lung neutrophilic inflammation.Biology and Metabolism of Sepsis: Innate Immunity, Bioenergetics, and Autophagy Calcium supplementation during sepsis exacerbates organ failure and mortality via calcium/calmodulin-dependent protein kinase kinase signaling JAK/STAT1 signaling promotes HMGB1 hyperacetylation and nuclear translocation.How macrophages ring the inflammation alarm HMGB1 release by H2O2-induced hepatocytes is regulated through calcium overload and 58-F interference.High-mobility group box 1, oxidative stress, and disease.Role of high-mobility group box 1 protein in inflammatory bowel disease.

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Calcium/calmodulin-dependent protein kinase (CaMK) IV mediates nucleocytoplasmic shuttling and release of HMGB1 during lipopolysaccharide stimulation of macrophages

http://www.wikidata.org/entity/Q36984742

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Calcium/calmodulin-dependent p ...... ide stimulation of macrophages

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Calcium/calmodulin-dependent p ...... ide stimulation of macrophages

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JIMMUNOL.181.7.5015

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Journal of Immunology

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Calcium/calmodulin-dependent p ...... ide stimulation of macrophages

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Anthony R Means

http://www.w3.org/2001/XMLSchema#string

David Wheeler

http://www.w3.org/2001/XMLSchema#string

Lanping Guo

http://www.w3.org/2001/XMLSchema#string

Matthew R Rosengart

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Richard A Shapiro

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Thomas J Ribar

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Timothy R Billiar

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Xianghong Zhang

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Ying Tang

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P2860

Signal-dependent nuclear export of a histone deacetylase regulates muscle differentiation

Differential localization of HDAC4 orchestrates muscle differentiation

Calmodulin-dependent kinase IV links Toll-like receptor 4 signaling with survival pathway of activated dendritic cells

The nuclear protein HMGB1 is secreted by monocytes via a non-classical, vesicle-mediated secretory pathway

Catalytic activity is required for calcium/calmodulin-dependent protein kinase IV to enter the nucleus.

A unique phosphorylation-dependent mechanism for the activation of Ca2+/calmodulin-dependent protein kinase type IV/GR.

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

HMG-1 as a late mediator of endotoxin lethality in mice

Components of a calmodulin-dependent protein kinase cascade. Molecular cloning, functional characterization and cellular localization of Ca2+/calmodulin-dependent protein kinase kinase beta

Calcium/calmodulin-dependent protein kinase activates serum response factor transcription activity by its dissociation from histone deacetylase, HDAC4. Implications in cardiac muscle gene regulation during hypertrophy

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5015-5023

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10.4049/JIMMUNOL.181.7.5015

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7

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23265417

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18802105

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lipopolysaccharide

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2587501

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