Responses of endothelial cell and astrocyte matrix-integrin receptors to ischemia mimic those observed in the neurovascular unit
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Blood-brain barrier breakdown in acute and chronic cerebrovascular diseaseAging and the neurovascular unitThe role of integrin alpha(v)beta (8) in neonatal hypoxic-ischemic brain injuryHemostasis and alterations of the central nervous systemIncreased plasma and tissue MMP levels are associated with BCSFB and BBB disruption evident on post-contrast FLAIR after experimental stroke.Innate inflammatory responses in stroke: mechanisms and potential therapeutic targets.Relationship of neurovascular elements to neuron injury during ischemia.Implantation site and lesion topology determine efficacy of a human neural stem cell line in a rat model of chronic stroke.The dual role of the neuroinflammatory response after ischemic stroke: modulatory effects of hypothermia.Microglial activation state exerts a biphasic influence on brain endothelial cell proliferation by regulating the balance of TNF and TGF-β1.Extensive vascular remodeling in the spinal cord of pre-symptomatic experimental autoimmune encephalomyelitis mice; increased vessel expression of fibronectin and the α5β1 integrin.A dual role for microglia in promoting tissue inhibitor of metalloproteinase (TIMP) expression in glial cells in response to neuroinflammatory stimuliInterendothelial claudin-5 expression depends on cerebral endothelial cell-matrix adhesion by β(1)-integrinsPolymorphisms of integrin, alpha 6 contribute to the development and neurologic symptoms of intracerebral hemorrhage in korean population.Transitory loss of glia and the subsequent modulation in inflammatory cytokines/chemokines regulate paracellular claudin-5 expression in endothelial cells.Microglial cell activation is a source of metalloproteinase generation during hemorrhagic transformation.Nanoagonist-mediated endothelial tight junction opening: A strategy for safely increasing brain drug delivery in mice.Chronic cerebral hypoxia promotes arteriogenic remodeling events that can be identified by reduced endoglin (CD105) expression and a switch in β1 integrinsCathepsin L acutely alters microvessel integrity within the neurovascular unit during focal cerebral ischemia.Dabigatran abrogates brain endothelial cell permeability in response to thrombin.Extracellular matrix and matrix receptors in blood-brain barrier formation and stroke.Perlecan domain V is upregulated in human brain arteriovenous malformation and could mediate the vascular endothelial growth factor effect in lesional tissueNeuron-derived FGF10 ameliorates cerebral ischemia injury via inhibiting NF-κB-dependent neuroinflammation and activating PI3K/Akt survival signaling pathway in miceDisruption of dystroglycan-laminin interactions modulates water uptake by astrocytesCell therapy for stroke: remaining issues to address before embarking on clinical trials.TNF-α promotes cerebral pericyte remodeling in vitro, via a switch from α1 to α2 integrins.The rapid decrease in astrocyte-associated dystroglycan expression by focal cerebral ischemia is protease-dependent.Increased expression of fibronectin and the alpha 5 beta 1 integrin in angiogenic cerebral blood vessels of mice subject to hypobaric hypoxia.An angiogenic role for the α5β1 integrin in promoting endothelial cell proliferation during cerebral hypoxia.Cerebral ischemia-induced angiogenesis is dependent on tumor necrosis factor receptor 1-mediated upregulation of α5β1 and αVβ3 integrins.Lipopolysaccharide-induced blood brain barrier permeability is enhanced by alpha-synuclein expressionInflammation and the neurovascular unit in the setting of focal cerebral ischemia.Perlecan domain V is neuroprotective and affords functional improvement in a photothrombotic stroke model in young and aged mice.Microglia preconditioned by oxygen-glucose deprivation promote functional recovery in ischemic rats.The neurovascular unit in the setting of stroke.Partial recovery of the damaged rat blood-brain barrier is mediated by adherens junction complexes, extracellular matrix remodeling and macrophage infiltration following focal astrocyte loss.Heterogeneity in the penumbra.Neurological diseases in relation to the blood-brain barrier.Perlecan and the blood-brain barrier: beneficial proteolysis?The cell-matrix interface: a possible target for treating retinal vascular related pathologies.
P2860
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P2860
Responses of endothelial cell and astrocyte matrix-integrin receptors to ischemia mimic those observed in the neurovascular unit
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Responses of endothelial cell ...... rved in the neurovascular unit
@en
type
label
Responses of endothelial cell ...... rved in the neurovascular unit
@en
prefLabel
Responses of endothelial cell ...... rved in the neurovascular unit
@en
P2093
P2860
P1433
P1476
Responses of endothelial cell ...... rved in the neurovascular unit
@en
P2093
Gregory J del Zoppo
Greta I Berg
Maria Spatz
Richard Milner
Stephanie Hung
Xiaoyun Wang
P2860
P304
P356
10.1161/STROKEAHA.107.486134
P407
P577
2007-11-21T00:00:00Z