A novel transgenic mouse model to study the osteoblast lineage in vivo.
about
Role of IGF-I signaling in muscle bone interactionsProspects for osteoprogenitor stem cells in fracture repair and osteoporosisSnail1 controls bone mass by regulating Runx2 and VDR expression during osteoblast differentiation.Osteoblast precursors, but not mature osteoblasts, move into developing and fractured bones along with invading blood vessels.The hematopoietic stem cell niche.A mouse model of osteochondromagenesis from clonal inactivation of Ext1 in chondrocytes.ATF4 promotes bone angiogenesis by increasing VEGF expression and release in the bone environment.Loss-of-function of ACVR1 in osteoblasts increases bone mass and activates canonical Wnt signaling through suppression of Wnt inhibitors SOST and DKK1.Bone and Muscle Pleiotropy: The Genetics of Associated Traits.Identification of a clonally expanding haematopoietic compartment in bone marrowBMP signaling negatively regulates bone mass through sclerostin by inhibiting the canonical Wnt pathway.Postnatal ablation of osteoblast Smad4 enhances proliferative responses to canonical Wnt signaling through interactions with β-catenin.Ras signaling regulates osteoprogenitor cell proliferation and bone formation.Regulation of osteogenesis-angiogenesis coupling by HIFs and VEGFAging of the hematopoietic stem cells niche.Ephrin B2/EphB4 mediates the actions of IGF-I signaling in regulating endochondral bone formation.Autocrine and Paracrine Actions of IGF-I Signaling in Skeletal Development.Hereditary Multiple Exostoses: New Insights into Pathogenesis, Clinical Complications, and Potential Treatments.BMP signalling in skeletal development, disease and repair.A phenotypic comparison of osteoblast cell lines versus human primary osteoblasts for biomaterials testing.Hypoxia, HIFs and bone development.Role of Osteoblast Gi Signaling in Age-Related Bone Loss in Female Mice.In Vivo Rescue of the Hematopoietic Niche By Pluripotent Stem Cell Complementation of Defective Osteoblast Compartments.Loss of Gi G-Protein-Coupled Receptor Signaling in Osteoblasts Accelerates Bone Fracture Healing.The pathogenic roles of heparan sulfate deficiency in hereditary multiple exostoses.Paracrine osteoprotegerin and β-catenin stabilization support synovial sarcomagenesis in periosteal cells.Bone loss in adult offspring induced by low-dose exposure to teratogens.Evaluation of the long-term skeletal effect induced by teratogen 5-aza-2'deoxycytidine on offspring of high (C3H/HeJ) and low (C57BL/6J) bone mass phenotype mice.
P2860
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P2860
A novel transgenic mouse model to study the osteoblast lineage in vivo.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
A novel transgenic mouse model to study the osteoblast lineage in vivo.
@en
A novel transgenic mouse model to study the osteoblast lineage in vivo.
@nl
type
label
A novel transgenic mouse model to study the osteoblast lineage in vivo.
@en
A novel transgenic mouse model to study the osteoblast lineage in vivo.
@nl
prefLabel
A novel transgenic mouse model to study the osteoblast lineage in vivo.
@en
A novel transgenic mouse model to study the osteoblast lineage in vivo.
@nl
P2093
P356
P1476
A novel transgenic mouse model to study the osteoblast lineage in vivo.
@en
P2093
Christa Maes
Henry M Kronenberg
Tatsuya Kobayashi
P304
P356
10.1196/ANNALS.1402.060
P407
P577
2007-11-01T00:00:00Z