Stabilization of HIF-1alpha is critical to improve wound healing in diabetic mice.
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Hypoxia-based strategies for angiogenic induction: the dawn of a new era for ischemia therapy and tissue regenerationHypoxia-inducible factors and the response to hypoxic stressThe possible mechanisms underlying the impairment of HIF-1α pathway signaling in hyperglycemia and the beneficial effects of certain therapiesChallenges and Opportunities in Drug Delivery for Wound HealingHypoxic signaling during tissue repair and regenerative medicineHypoxia and Integrin-Mediated Epithelial Restitution during Mucosal InflammationHypoxia-inducible factors in physiology and medicineThe mammalian blastema: regeneration at our fingertipsThe Role of Hypoxia-Inducible Factor in Wound HealingAntimycotic ciclopirox olamine in the diabetic environment promotes angiogenesis and enhances wound healingIdentification of the critical therapeutic entity in secreted Hsp90α that promotes wound healing in newly re-standardized healthy and diabetic pig modelsImpaired angiogenesis and mobilization of circulating angiogenic cells in HIF-1alpha heterozygous-null mice after burn wounding.Altered glucose metabolism and hypoxic response in alloxan-induced diabetic atherosclerosis in rabbits.Vascular responses to hypoxia and ischemia.STAT3 controls the neutrophil migratory response to CXCR2 ligands by direct activation of G-CSF-induced CXCR2 expression and via modulation of CXCR2 signal transductionLocal injection of deferoxamine improves neovascularization in ischemic diabetic random flap by increasing HIF-1α and VEGF expressionReduction of ARNT in myeloid cells causes immune suppression and delayed wound healingCombined vascular endothelial growth factor-A and fibroblast growth factor 4 gene transfer improves wound healing in diabetic mice.Systemic preconditioning by a prolyl hydroxylase inhibitor promotes prevention of skin flap necrosis via HIF-1-induced bone marrow-derived cells.Low-level laser therapy (808 nm) contributes to muscle regeneration and prevents fibrosis in rat tibialis anterior muscle after cryolesionThe prolyl hydroxylase inhibitor dimethyloxalylglycine enhances dentin sialophoshoprotein expression through VEGF-induced Runx2 stabilizationDrug-induced regeneration in adult mice.Dimethyloxalylglycine prevents bone loss in ovariectomized C57BL/6J mice through enhanced angiogenesis and osteogenesis.HIF-1α transgenic bone marrow cells can promote tissue repair in cases of corticosteroid-induced osteonecrosis of the femoral head in rabbits.Low level laser therapy increases angiogenesis in a model of ischemic skin flap in rats mediated by VEGF, HIF-1α and MMP-2.Transdermal deferoxamine prevents pressure-induced diabetic ulcers.Metabolic reprogramming by HIF-1 promotes the survival of bone marrow-derived angiogenic cells in ischemic tissue.Partial deficiency of HIF-1α stimulates pathological cardiac changes in streptozotocin-induced diabetic mice.Improved diabetic wound healing through topical silencing of p53 is associated with augmented vasculogenic mediatorsAdipose-derived stem cells accelerate neovascularization in ischaemic diabetic skin flap via expression of hypoxia-inducible factor-1α.Role of HIF-1α in maternal hyperglycemia-induced embryonic vasculopathyA fragment of secreted Hsp90α carries properties that enable it to accelerate effectively both acute and diabetic wound healing in miceHypoxia-inducible factor as an angiogenic master switchVascular regeneration effect of adipose-derived stem cells with light-emitting diode phototherapy in ischemic tissue.Effects of dimethyloxalylglycine on wound healing of palatal mucosa in a rat modelTie2-dependent knockout of HIF-1 impairs burn wound vascularization and homing of bone marrow-derived angiogenic cellsSecreted heat shock protein-90 (Hsp90) in wound healing and cancer.Hsp90α and Hsp90β together operate a hypoxia and nutrient paucity stress-response mechanism during wound healing.Arnebin-1 promotes angiogenesis by inducing eNOS, VEGF and HIF-1α expression through the PI3K-dependent pathway.Short Hairpin RNA Silencing of PHD-2 Improves Neovascularization and Functional Outcomes in Diabetic Wounds and Ischemic Limbs
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P2860
Stabilization of HIF-1alpha is critical to improve wound healing in diabetic mice.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 04 December 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Stabilization of HIF-1alpha is critical to improve wound healing in diabetic mice.
@en
Stabilization of HIF-1alpha is critical to improve wound healing in diabetic mice.
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type
label
Stabilization of HIF-1alpha is critical to improve wound healing in diabetic mice.
@en
Stabilization of HIF-1alpha is critical to improve wound healing in diabetic mice.
@nl
prefLabel
Stabilization of HIF-1alpha is critical to improve wound healing in diabetic mice.
@en
Stabilization of HIF-1alpha is critical to improve wound healing in diabetic mice.
@nl
P2093
P2860
P50
P356
P1476
Stabilization of HIF-1alpha is critical to improve wound healing in diabetic mice.
@en
P2093
Anca Irinel Catrina
Ileana Ruxandra Botusan
Jacob Grünler
Lorenz Poellinger
Octavian Savu
Sergiu-Bogdan Catrina
Stina Lindberg
Vivekananda Gupta Sunkari
P2860
P304
19426-19431
P356
10.1073/PNAS.0805230105
P407
P577
2008-12-04T00:00:00Z