Importance of TLR2 in early innate immune response to acute pulmonary infection with Porphyromonas gingivalis in mice
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Toll-like receptor 2 regulates organic dust-induced airway inflammationThe C5a receptor impairs IL-12-dependent clearance of Porphyromonas gingivalis and is required for induction of periodontal bone lossDNA from Porphyromonas gingivalis and Tannerella forsythia induce cytokine production in human monocytic cell lines.Manipulation of Neutrophils by Porphyromonas gingivalis in the Development of Periodontitis.Effects of the TLR2 agonists MALP-2 and Pam3Cys in isolated mouse lungsPorphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis.Mechanisms of neutrophil accumulation in the lungs against bacteria.Review: Pathogen-induced inflammation at sites distant from oral infection: bacterial persistence and induction of cell-specific innate immune inflammatory pathways.Receptor for advanced glycation endproducts mediates pro-atherogenic responses to periodontal infection in vascular endothelial cellsMobilization of endothelial progenitors by recurrent bacteremias with a periodontal pathogen.In vivo function of airway epithelial TLR2 in host defense against bacterial infectionFree lipid A isolated from Porphyromonas gingivalis lipopolysaccharide is contaminated with phosphorylated dihydroceramide lipids: recovery in diseased dental samples.A metalloproteinase karilysin present in the majority of Tannerella forsythia isolates inhibits all pathways of the complement system.Inhibition of Sprouty2 polarizes macrophages toward an M2 phenotype by stimulation with interferon γ and Porphyromonas gingivalis lipopolysaccharide.Gingipains: Critical Factors in the Development of Aspiration Pneumonia Caused by Porphyromonas gingivalis.Toll-like receptors in atherosclerosisAge-related alterations in innate immune receptor expression and ability of macrophages to respond to pathogen challenge in vitro.Neutrophil recruitment to the lungs during bacterial pneumonia.Toll gates to periodontal host modulation and vaccine therapyComplementary Tolls in the periodontium: how periodontal bacteria modify complement and Toll-like receptor responses to prevail in the host.Breaking bad: manipulation of the host response by Porphyromonas gingivalis.Too old to fight? Aging and its toll on innate immunity.Complement and periodontitis.The expression and functions of toll-like receptors in atherosclerosis.IRF6 Regulates the Expression of IL-36γ by Human Oral Epithelial Cells in Response to Porphyromonas gingivalis.Differential virulence and innate immune interactions of Type I and II fimbrial genotypes of Porphyromonas gingivalis.Immune response of macrophages from young and aged mice to the oral pathogenic bacterium Porphyromonas gingivalis.Variability of the dendritic cell response triggered by different serotypes of Aggregatibacter actinomycetemcomitans or Porphyromonas gingivalis is toll-like receptor 2 (TLR2) or TLR4 dependent.Signaling mechanisms involved in altered function of macrophages from diet-induced obese mice affect immune responses.Lack of soluble tumor necrosis factor alpha receptor 1 and 2 and interleukin-1beta compartmentalization in lungs of mice after a single intratracheal inoculation with live Porphyromonas gingivalis.Scavenger receptor A is expressed by macrophages in response to Porphyromonas gingivalis, and participates in TNF-alpha expression.The role of toll-like receptor 2 in the recognition of Aggregatibacter actinomycetemcomitans.Oral Biofilms from Symbiotic to Pathogenic Interactions and Associated Disease -Connection of Periodontitis and Rheumatic Arthritis by Peptidylarginine Deiminase.Host response to pulmonary fungal infections: A highlight on cell-driven immunity to Cryptococcus species and Aspergillus fumigatus.Macrophage Polarization in Chronic Inflammatory Diseases: Killers or Builders?TLR2-dependent amelioration of allergic airway inflammation by parasitic nematode type II MIF in mice.Neutrophil Elastase Subverts the Immune Response by Cleaving Toll-Like Receptors and Cytokines in Pneumococcal Pneumonia.OmpA-like proteins of Porphyromonas gingivalis contribute to serum resistance and prevent Toll-like receptor 4-mediated host cell activation
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Importance of TLR2 in early innate immune response to acute pulmonary infection with Porphyromonas gingivalis in mice
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 2008
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Importance of TLR2 in early in ...... rphyromonas gingivalis in mice
@en
Importance of TLR2 in early in ...... phyromonas gingivalis in mice.
@nl
type
label
Importance of TLR2 in early in ...... rphyromonas gingivalis in mice
@en
Importance of TLR2 in early in ...... phyromonas gingivalis in mice.
@nl
prefLabel
Importance of TLR2 in early in ...... rphyromonas gingivalis in mice
@en
Importance of TLR2 in early in ...... phyromonas gingivalis in mice.
@nl
P2093
P2860
P1476
Importance of TLR2 in early in ...... rphyromonas gingivalis in mice
@en
P2093
George Hajishengallis
Gregory J Bagby
Steve Nelson
P2860
P304
P356
10.4049/JIMMUNOL.181.6.4141
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P577
2008-09-01T00:00:00Z